11/06/2025
Like the Community Therapists Network we are sharing this message containing a number of unanswered questions about the safety of these much talked about new weight loss injections.
Why we want a thorough and independent investigation into the safety of the new weight lost injections (GLP-1 drugs)
There are increasing reasons for questioning the safety of these new injections, including a significant risk of cells in the body becoming cancerous.
And that risk can be exacerbated when a person's immune system is unbalanced; in this case becoming underactive or suppressed.
To explain: when a cell becomes cancerous, and starts to behave abnormally, our immune (self-defence) system should ‘identify it’ as ‘foreign’ to then remove it, and ‘digest it’.
What we believe is particularly concerned about these drugs is the potential risk of cancer could be when the drugs are injected, as per the prescription.
How we have come to understand this begins with a scientific paper published in 2013 by renowned cancer expert Prof Thomas Seyfried. That paper is entitled "Cancer as a metabolic disease: implications for novel therapeutics".
In it, Prof Seyfried writes:
"Emerging evidence indicates that cancer is primarily a metabolic disease involving disturbances in energy production through respiration and fermentation."
This is a landmark paper that began a movement in science to challenge conventional thinking and beliefs that a person’s genes are responsible for cells to turning cancerous.
Ten years on and TV doctor, Dr Ranjan Chatterjee held a conversation with Professor Seyfried; a conversation that was "packed with actions we can all take to reduce our risk not just of cancer, but all the chronic conditions driven by metabolic disruption."
Today, we are sharing this post on GLP-1 drugs because we believe there is reason to think the use of them supports Professor Seyfried’s research and science that cancer is a metabolic disease.
And what led us to believe this?
The very reason for needing to prescribe these chemicals could well be why a cell ’turns cancerous’.
Therefore, it is vitally important for people to more fully understand why these drugs are prescribed.
In an attempt to keep things simple and logical, it is because the cells in a person’s body, that make natural GLP-1 (hormone) then fail to release the GLP-1 from the cell; but they could continue to make it (inside the cell).
It is very important that we emphasise that ‘failing to release it’ does not mean the cells immediately stop the process of making GLP-1.
Why we need to emphasise this distinction?
Because that event which triggers a cell to begin making GLP-1 happens outside the cell. In fact, it is an event that can be triggered from outside a person's body.
And "that event"?
Eating foods, which then get digested by the body; a process triggered at the very moment food enters the mouth, which can potentially begin as a person smells food. That is why we say "it is an event that can be triggered from outside a person's body".
Another major reason for emphasising the difference is:
Because it can help explain why cells may turn cancerous; an event that may happen more frequently with cells in the pancreas and bowel.
That is because cells in both organs should under healthy and balanced conditions make and then release GLP-1.
So why are some people being prescribed this ‘artificial’ GLP-1, when science knows the body should be making and releasing it?
In other words, what is stopping or blocking the cells in the pancreas and bowel, in some people, from releasing this GLP-1? Remember, the cells can still be triggered to start production process.
It turns out that science has uncovered different reasons; one can be when a person experiences chronic stress. Another, the result of eating of foods poor in ‘nutritional density or value for a prolonged period ie several months;
Those are foods people often like to eat to experience a feeling of comfort, and to cope with stressful times; foods typically high in carbohydrates (including sugar).
Now, what if injecting artificial GLP-1 (the drug) can contribute to inhibiting the release of natural GLP-1?
It’s not a ‘what if’!
And that brings us to "How would and could a block in the release of GLP-1 increase a person's risk of cancer, especially when they are taking the artificial GLP-1, AS PRESCRIBED?"
While we don't believe we have the authority to directly answer that question, we hope we have given you sufficient common sense and logical reasoning for you to wish to ask the likes of Google the same questions we asked, that lead us to uncover the answer to that question, and why you are reading this post.
And those questions:
Are pancreatic alpha cells involved in pancreatic cancer?
Are enteroendocrine L cells of the bowel associated with bowel cancer?
How is GLP-1 made?
Is proglucagon involved in cell division?
Is there evidence the proglucagon is associated with cancer?
Can proglucagon increase in the cells, if GLP-1 is not released?
When during the day is GLP-1 normally released?
When should a person inject GLP-1 agonist?
Now, we cannot and will not end this post, and not offer reasons to be hopeful. That is why we also asked this question:
If a chronically high, unbalanced level of proglucagon is associated with cancer, is it possible for a person to change their style of living, for the levels to be rebalanced?
You will of course, intuitively, know the answer.
Therefore, perhaps more importantly is the how?
And that can be answered by engaging in prolonged fasts (3 to 4 days), over a period of a few months.
But if that initially feels too challenging, then we recommend eating keto style foods.
Both approaches will help people to rebalance proglucagon levels and of course their weight.
If, after reading this you have other questions, please do contact us via support@lifestylehealth.org.uk
