Homoeopathy A Panacea

Homoeopathy A Panacea ILLUSIONS AND REALITIES ABOUT HOMOEOPATHY CASE STUDIES DISCUSSIONS. HEALTH AND DISEASES PHOTOS

Shout out to my newest followers! Excited to have you onboard! Roshankumar Patil, Raghunath LD, Manthiraju Siva Rama Kri...
31/12/2025

Shout out to my newest followers! Excited to have you onboard! Roshankumar Patil, Raghunath LD, Manthiraju Siva Rama Krishna, Sanjay Goel

Welcome to Homoeopathy — A Panacea 🌿
I’m glad you’re here.
This space is dedicated to Homoeopathy as a true medical science — not a hobby, trend, or casual experiment. Here, every case begins with a proper diagnosis and a clear understanding of the disease — not just vague symptoms or “indispositions.”
My goal is to educate, share real clinical insights, and help you see Homoeopathy as a structured, scientific, and compassionate system of healing.
Thank you for joining this journey of learning and awareness. 🙏

30/12/2025

How I Treat Frozen Shoulder with Individualised Homeopathy | Real Practice Approach

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Description:
No two Frozen Shoulder patients are the same.
That’s why individualised homeopathic treatment — based on the complete picture of the person — plays a key role in recovery.
In this video, I share how I assess, plan, and manage Frozen Shoulder cases in my clinic, along with gentle guidance and structured follow-up.
Educational purpose only.

29/12/2025

CLINICAL EYE: - Observe. Analyze.Diagnose Learn.

💡 What is Frozen Shoulder?
Frozen shoulder is a painful stiffness of the shoulder joint caused by thickening and tightening of the joint capsule. This restricts movement — often both active and passive movement.
It usually affects one shoulder (sometimes both sequentially) and is most common in people 40–70 years old — slightly more in women.
🔍 Why Does It Happen?
In many people, the exact cause is unknown (idiopathic).
It is more common if you have:
Diabetes (very common)
Thyroid disorders
Parkinson’s disease
Heart disease
Prolonged immobilization (fracture, sling)
Previous shoulder injury or surgery
Sometimes it follows inflammation, trauma, or repetitive strain.

🚩 If the pain is truly very sudden + severe
…especially after lifting, pushing, or a fall — doctors also consider:
Rotator cuff tear
Calcific tendinitis
Bursitis
Shoulder dislocation
These usually have pain first, stiffness later, and passive movement may still be possible — unlike frozen shoulder where both active & passive movement become limited.

“Very high success rate — most patients recover within 1–3 months with individualized treatment :

28/12/2025

CLINICAL EYE: - Observe. Analyze.Diagnose Learn. ”

I’ll share a careful, clinical way to think about it — not a firm diagnosis, because that needs a proper exam. But with:
5 months of intense itching
multiple family members also itching
worse at night? (often reported in such cases)
little response to random treatments
…the most probable diagnosis is SCABIES — very often “steroid-modified / masked scabies (scabies incognito)” if creams or steroids were used at any point.
Why scabies fits best
Scabies typically causes: ✔ Severe itching — especially at night
✔ Household spread — everyone scratches
✔ Few visible lesions — sometimes only small red dots / scratch marks
✔ May persist for months if not treated correctly
✔ Gets worse or partially masked after steroid creams
In many cases, the rash is subtle — the main symptom is intense itching.
Other conditions that can look similar (but less likely since others are affected):
Papular urticaria / bed bug bites – clusters, seasonal, sleep-related
Allergic / contact dermatitis – usually not contagious
Atopic eczema – chronic but not infectious
Chronic xerosis (dry skin) – usually mild itching only
But the fact that other members are suffering too makes a contagious cause highest on the list → scabies until proven otherwise.
Why treatment may have failed
I see this VERY often:
Only one person is treated
Or dose / protocol is incomplete
Or steroid creams were used
Or linens and clothes not treated
Or repeat treatment not done
Result → itching never stops.




One of the hardest sentences in medicine is: “We do not yet know.”In this case, multiple advanced centers offered possib...
25/12/2025

One of the hardest sentences in medicine is: “We do not yet know.”
In this case, multiple advanced centers offered possibilities, assumptions, and supplements — but not that sentence.
This is not a criticism of individuals. It is a reminder that medicine is still evolving, and that uncertainty is not failure. Pretending certainty, however, can be.

Reversal of Congenital Nail Growth Arrest in a Child: A Case ReportCLINICAL EYE:  - Observe. Analyze.Diagnose  Learn.   ...
25/12/2025

Reversal of Congenital Nail Growth Arrest in a Child: A
Case Report

CLINICAL EYE: - Observe. Analyze.Diagnose Learn. ”

Abstract
Congenital disorders of nail growth are rare and often lack definitive therapeutic options in conventional medical practice, particularly in the absence of syndromic associations. We report the case of an 8-year-old female child presenting with lifelong absence of normal nail growth affecting multiple fingernails. Extensive prior evaluations failed to identify a definitive diagnosis or offer effective treatment. Following individualized homoeopathic intervention, objective nail growth was observed within three months. This case highlights a rare presentation of congenital nail matrix dysfunction and raises the possibility of functional reversibility when addressed through individualized systemic therapy.

Keywords
Congenital anonychia, Nail matrix hypoplasia, Nail growth arrest, Pediatric nail disorder, Idiopathic nail dystrophy

Introduction
Normal nail development depends on the functional integrity of the nail matrix, an ectodermal structure responsible for keratin production. Congenital nail abnormalities range from complete absence (anonychia) to hypoplastic or dystrophic nails. Isolated congenital nail growth arrest without associated skeletal, ectodermal, or genetic syndromes is extremely uncommon. Management options in conventional medicine are limited and largely supportive. This report documents a rare case of congenital nail matrix inactivity with subsequent restoration of nail growth following individualized homoeopathic treatment.

Case Presentation
An 8-year-old girl was brought for consultation with the chief complaint of absence of normal nail growth since early childhood. According to parental history, the fingernails had remained persistently short and underdeveloped, with no appreciable forward growth over several years.
The child had previously been evaluated at multiple tertiary-care hospitals in major cities. Despite clinical examinations and routine investigations, no definitive diagnosis or treatment plan was offered. There was no history of trauma, habitual nail biting, chemical exposure, or chronic medication use. Family history was non-contributory. Growth and developmental milestones were appropriate for age.
On examination, the fingernails were present but markedly short, thin, and hypoplastic, consistent with arrested nail growth. The periungual skin appeared normal. There were no associated skeletal abnormalities or features suggestive of ectodermal dysplasia.
Based on clinical findings, the condition was assessed as partial congenital anonychia with functional nail matrix hypoplasia, idiopathic in nature.

Intervention and Outcome

The patient was managed with individualized homoeopathic treatment, prescribed after detailed case analysis. No local or external applications were used during the treatment period.
Within three months of initiating therapy, clear and progressive nail growth was observed in multiple fingernails, representing the first documented nail growth in the patient’s life. The improvement was sustained on subsequent follow-up visits.

Discussion
Congenital nail growth disorders are primarily considered structural or genetic, with limited scope for reversal once established. The absence of syndromic features in this case suggests a functional rather than irreversible structural defect of the nail matrix. The observed response indicates possible reactivation of nail matrix function, emphasizing the importance of individualized systemic approaches in select idiopathic cases.
This case also underscores the limitations of symptom-based management in rare developmental nail disorders and highlights the need for broader therapeutic perspectives.
Conclusion
This case documents a rare presentation of congenital nail growth arrest with subsequent restoration of nail growth following individualized homoeopathic treatment. While further studies are required, the findings suggest that certain congenital nail matrix dysfunctions may not be entirely irreversible.




24/12/2025

for more Clinical cases.

“Can a nail that never grew… start growing?
This child had a rare congenital nail matrix disorder.
No known treatment in conventional medicine.
Yet the nail matrix showed functional recovery.
Medicine still has unanswered questions.”





23/12/2025

For more cases

Skin diseases

A 40-year-old patient with facial warts for over 1 year shares an important observation:
“The more creams I apply, the more warts appear.”
This is a common clinical mistake.
Warts are not merely a surface problem of the skin.
Repeated external applications — even when prescribed — often suppress the condition instead of resolving it.
When the internal imbalance is left untouched,
the body finds another outlet… and new warts appear.
🔍 Treatment must address the root cause, not just the visible lesions.
• What suppression means in chronic skin diseases
• The correct approach to treating warts
Stay informed. Choose wisely




Why mustard oil can worsen hyperkeratosis1. Strong rubefacient & irritant actionMustard oil contains allyl isothiocyanat...
18/12/2025

Why mustard oil can worsen hyperkeratosis

1. Strong rubefacient & irritant action

Mustard oil contains allyl isothiocyanate, a potent irritant.

It increases local blood flow

Produces heat, burning, and redness

Stimulates already overactive epidermal cells

👉 In hyperkeratosis, keratinocyte proliferation is already excessive. Mustard oil pushes this process further, leading to thickening, cracking, and inflammation.

---

2. Disruption of the skin barrier

Hyperkeratotic skin has:

Poor barrier function

Reduced hydration

Abnormal lipid composition

Mustard oil:

Penetrates deeply

Further disturbs the stratum corneum

Increases transepidermal water loss

👉 Result: more dryness → more compensatory keratin formation

---

3. Inflammation-driven hyperproliferation

Chronic low-grade inflammation is central to hyperkeratosis.

Mustard oil:

Activates inflammatory mediators

Stimulates nerve endings

Causes repeated micro-injury

👉 The skin responds defensively by laying down more keratin, worsening plaques and fissures.

---

4. Aggravation in fissured or cracked skin

In cases with:

Heel cracks

Palmar/plantar hyperkeratosis

Keratoderma

Mustard oil causes:

Burning pain

Secondary inflammation

Delayed healing

Patients often report temporary softness followed by rebound thickening, which is a classic sign of irritation-driven pathology.

18/12/2025

Multiple warts appearing after burning a single wart—
a classic example of suppression.

This case was treated only with constitutional homoeopathic medicines,
without any external applications.
Gradual, stable recovery followed.

Skin diseases need understanding, not shortcuts.

👉 Share this to spread awareness
📩 Consult for proper guidance

CLINICAL EYE: - Observe. Analyze.Diagnose Learn. ”

For more cases








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18/12/2025

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Telephone

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