06/12/2025
Hi everyone - after researching why some cancers might respond to fenbendazole while others may take longer periods , I wanted to share what I've learned since this question comes up. I've also explored how ivermectin might work alongside other components which makes up the complete protocol
The Science (Simplified)These medications seem to work in different but complementary ways:
Fenbendazole:
• Disrupts cell division by targeting microtubules (cancer's internal highway system)
• Interferes with cancer cell metabolism, cutting off its fuel supply
• May trigger cell death in cancer cells
Ivermectin:
• Blocks cancer's ability to repair damaged DNA
• Disrupts communication pathways cancer cells need to survive
• May enhance immune system recognition of cancer cells
Why the Differences?Cancers that might respond better tend to:
• Grow quickly (more vulnerable to microtubule disruption)
• Have high energy demands
• Lack multiple resistance mechanisms
Cancers that might respond less tend to:
• Grow slowly
• Have built-in resistance mechanisms
• Be protected by barriers (like brain tumors)
Ivermectin:
•Inhibit cancer cell growth: Some studies suggest ivermectin can inhibit the growth of certain cancer cells by affecting their ability to divide and survive.
• Enhance chemotherapy effectiveness: There is ongoing research on how ivermectin may improve the effectiveness of other cancer treatments, such as chemotherapy or targeted therapies.safe to use ivermectin while doing chemo
• Impact on cell signaling pathways: Ivermectin may influence pathways involved in cancer cell survival and metastasis, such as those affecting autophagy (the process through which cells break down and recycle components) and cell death.
My research suggests cancers like lymphomas, certain lung cancers, and some breast cancers may be more vulnerable based on their biology. Others like pancreatic cancer and glioblastoma may be more resistant.
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