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07/10/2021

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25/10/2015

'2009 Nobel Prize For Medicine' - was awarded for the discovery of 'how chromsomes are protected by Telomeres and the enzyme Telomerase.'

24/07/2015

18/12/2014

06/05/2014

BAD THINGS HAPPEN WHEN TELOMERS GET SHORTER!

How likely are you to catch the next cold bug?
(This article was published in 'The Globe and Mail' Feb. 19 2013)
‘The answer could lie in the length of your telomeres, according to a team of U.S. researchers.
Telomeres are protective caps on the ends of the 23 pairs of chromosomes that contain all our genetic material.
They are often compared to the plastic tips on the ends of shoelaces, preventing the chromosomes from fraying. Telomeres tend to get shorter as a cell undergoes normal division. Eventually, the telomeres become so short they can no longer hold the chromosomes intact and the cell dies. Aside from normal aging, telomeres can be cut short by other factors such as chronic stress or severe early childhood experiences. It’s a bit like speeding up the body’s biological clock.
Previous studies have found that seniors who have extremely short telomeres are at increased risk of cardiovascular disease, cancer and early death. But little is known about the role that telomeres play in the disease susceptibility of younger people. To find out, researchers at Carnegie Mellon University in Pittsburgh recruited 152 healthy volunteers, aged 18 to 55. At the start of the study, blood was drawn from each participant. From this sample, the researchers measured the length of telomeres in a particular type of white blood cell that helps the body fight infection.
Then each of the subjects was exposed to a common cold virus and kept in quarantine for five days, as the researchers observed which ones ended up getting sick. The finding, published this week in the Journal of the American Medical Association, showed that the participants with shorter telomeres were more likely to catch the cold.
This trend was clearly visible in those aged 22 to 55. But there was no relationship between telomere length and risk of infection among the youngest participates of the study, those aged 18 to 21. At this relatively young age, it seems there were just not enough immune cells with truncated telomeres to affect their susceptibility.
Still, the study found that telomere size certainly matters with increasing age.
“As the telomeres get shorter, the cell isn’t functioning as well as it should,” explained the lead researcher, Sheldon Cohen, who is a professor of psychology at Carnegie.
“And if a greater percentage of your [immune] cells are like this, then it means you are going to have fewer of them that are effectively fighting off the [cold] virus.”
Although a lot more research needs to be done, Cohen said telomere length could be used to help predict a person’s general susceptibility to disease.
It might, for instance, pinpoint those who are in need of special medical attention. And that raises another question: Can this susceptibility be reversed so that biology does not become destiny?
Scientists already know that an enzyme called telomerase protects telomeres from degradation. The key is to finding treatments, or activities, that boost the body’s production of telomerase.’

21/04/2014

Heart Attacks and Telomere Length – University of Copenhagen Study.

Every cell in the body has chromosomes with so-called telomeres, which are shortened over time and also through lifestyle choices such as smoking and obesity. Researchers have long speculated that the shortening of telomeres increases the risk of heart attack and early death. Now a large-scale population study in Denmark involving nearly 20,000 people shows that there is in fact a direct link.

The conclusion was clear: If the telomere length was short, the risk of heart attack and early death was increased by 50 and 25 per cent, respectively.

Click here to see a full report on the study. http://news.ku.dk/all_news/2012/2012.2/Cellular-aging-increases-risk-of-heart-attack/

It seems to me that to minimize the risk of Heart Attacks we need to be doing everything we can to maximize the length of our telomeres

A large-scale population study in Denmark involving nearly 20,000 people shows a direct link between cellular aging and risk of heart attack and early death.

19/04/2014

Alzheimer's disease (AD) is the most common form of dementia.

Dementia is the loss of cognitive functioning—thinking, remembering, and reasoning—and behavioral abilities, to such an extent that it interferes with a person’s daily life and activities. Dementia ranges in severity from the mildest stage, when it is just beginning to affect a person’s functioning, to the most severe stage, when the person must depend completely on others for basic activities of daily living.

There is no cure for the disease, which worsens as it progresses, and eventually leads to death

Most often, AD is diagnosed in people over 65 years of age, although the less-prevalent early-onset Alzheimer's can occur much earlier. In 2006, there were 26.6 million sufferers worldwide. Alzheimer's is predicted to affect 1 in 85 people globally by 2050.

As the disease advances, symptoms can include confusion, irritability and aggression, mood swings, trouble with language, and long-term memory loss. As the sufferer declines they often withdraw from family and society. Gradually, bodily functions are lost, ultimately leading to death.

Alzheimer's disease is characterised by loss of neurons and synapses in the cerebral cortex and certain subcortical regions. This loss results in gross atrophy of the affected regions, including degeneration in the temporal lobe and parietal lobe, and parts of the frontal cortex and cingulate gyrus. Studies using MRI and PET have documented reductions in the size of specific brain regions in people with AD as they progressed from mild cognitive impairment to Alzheimer's disease, and in comparison with similar images from healthy older adults

Currently there are about 280,000 Australians living with dementia. Experts suggest that as many as 5.1 million Americans may have Alzheimer’s disease.

Scientific papers have been published which state that :

Telomere shortening correlates with Alzheimer's disease status. http://www.ncbi.nlm.nih.gov/pubmed/12493553

( Click Here for original article )

Alzheimer’s is one of the conditions that are associated with short Telomeres. Remember what happened in Dr DePinho’s mice when the Telomeres were lengthened, the brains of the old mice increased from 75% of normal size back to normal size.

Knowing the above, imagine how excited you would be if you had a loved one who had been diagnosed with this condition, start consuming a Product B, and show some improvements within a few weeks. Greg has told me that his mum is continuing to improve since we shot the video.

Click Here http://noc.talkfusion.com/fusion2/view.asp?MjQ4MjM1OA==_11437780 to see video of Greg discussing his mum’s condition and changes.

The fact that Dr De Pinho has shown that ‘Aged Tissues Have a Point of Return’ and the development of Telomerase Activators will prove to be one of the GREATEST MEDICAL BREAKTHROUGHS of all time !

PubMed comprises more than 23 million citations for biomedical literature from MEDLINE, life science journals, and online books. Citations may include links to full-text content from PubMed Central and publisher web sites.

16/04/2014

The video below is probably the clearest and most comprehensive explanation I have ever seen regarding the nearly unbelievable developments that are happening in the anti-aging field of telomere biology.

It is of Dr Bill Andrews explaining to a group of pharmacists the history and latest developments in Telomere Biology. I think this presentation gives a lot of detail and should be watched by anybody who is serious about understanding the latest in Telomere research and the potential this new field of science has to help people. You can view this presentation at http://www.teamfsi.net/dr-bill-andrews.html ( Note : It is in 2 parts )

The above presentation was on February 23, 2012. Bill has been working continuously since then with John Anderson and they have released a new 3rd Generation Product B in August 2012 which is much stronger than the original version.

Having watched the above video I suggest you then take some time and study all of the video testimonies on http://www.stopthebodyclock.com/home.html Password stopthebodyclock By doing this you will realize that a lot of the conditions associated with short Telomeres and mentioned by Bill Andrews are already being helped…..it seems that Product B is obviously lengthening critically short Telomeres!!!

http://www.stopthebodyclock.com/home.html

12/04/2014

Telomeres, lifestyle, cancer, and aging.

In an article published in 2011 Masood A. Shammas Harvard (Dana Farber) Cancer Institute, Boston,Massachusetts, USA states :

Certain lifestyle factors such as smoking, obesity, lack of exercise, and consumption of unhealthy diet can increase the pace of telomere shortening, leading to illness and/or premature death. Accelerated telomere shortening is associated with early onset of many age-associated health problems, including coronary heart disease, heart failure, diabetes, increased cancer risk,and osteoporosis. The individuals whose leukocyte telomeres are shorter than the corresponding average telomere length have three-fold higher risk to develop myocardial infarction. Evaluation of telomere length in elders shows that the individuals with shorter telomeres have a much higher rate of mortality than those with longer telomeres. Excessive or accelerated telomere shortening can affect health and lifespan at multiple levels.
To watch a 1 Minute Video discussing Telomere Length and Cancer go to : http://youtu.be/PylY3hVzrb4
It has been suggested that keeping Telomeres long will help reduce the likelihood of people getting cancer in the first place and also also increasing the likelihood of surviving cancer if a person does get cancer.
Check out www.restoremybody.net and www.mikegoggan.isagenix.com website so you can become a member and/or purchase Product if you choose to do so.

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09/03/2014

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