Kidney Coach

21/10/2020

How to boost your klotho levels 😲🍄
Research into increasing klotho levels in humans is really just beginning but there are some factors that have been shown to decrease klotho levels which gives us some good information about what we may be able to do to increase our own endogenous klotho.
Tips to boost klotho levels:
• Manage stress levels
• Reduce inflammation and oxidative stress
• Decrease indoxyl sulfate- indoxyl sulfate is a uraemic toxin produced by bacteria in the gut after the breakdown of animal protein. Decreasing animal protein in the diet, increasing dietary fibres and the use of prebiotics and probiotics have all been shown to lower indoxyl sulfate levels.
• Avoid alcohol
• Exercise regularly
Can supplements increase klotho levels?
There are some studies showing that the Chinese Medicine herb Polygonum multiflorum or Fo-ti and the mushroom Cordyceps sinensis increase klotho levels in animals.
Polyonum multiflorum (Fo-ti) is one of the most popular herbs in Chinese medicine. One of the main constituents in Fo-ti is Tetrahydroxystilbene glucoside (TSG for short!) which is a strong antioxidant and free radical scavenger.
A study in aged mice found that administration of TSG from Fo-ti for 8 weeks reversed age related decreases in klotho, increased the levels of klotho in their bloodstream and increased the expression of klotho in the brain, heart, kidney, te**is and epididymis in a dose-dependent manner. Examination of the organs also showed that TSG improved disease related changes in the organs.
The mushroom Cordyceps sinensis was given to rats with high blood pressure, after treatment with Cordyceps, klotho expression was increased, and kidney damage was reduced.

06/10/2020

What is the healthiest cookware? 😕
Pots, pans and storage containers may increase your exposure to perfluorinated compounds (non-stick cookware), toxic metals (aluminium or copper pots) and plastics. Ideally cookware used to heat food should be made of stainless steel, porcelain, glass, enamel coated iron or cast iron.
Heating plastics of any kinds is not recommended because of the risk of chemicals leaching into them.
Pots and Pans:
Aluminium pots and pans aren’t recommended to be used in cooking because of the possible link between aluminium and Alzheimer’s disease. Copper pots and pans should be avoided because copper is naturally antibacterial and may therefore affect the natural microbes in your food. In addition, several cases of copper poisoning have been reported in children who have their food prepared in copper or brass pots.
Non-stick cookware:
Non-stick cookware has received a lot of adverse publicity as a result of the use of PFOA (perfluorooctanoic acid) in its manufacture which is suspected to be carcinogenic, a hormone disruptor and a reproductive toxin. As a result of these concerns, many manufactures phased it out in 2015 however it is difficult to determine if the manufacturers of non-stick cookware continue to use perfluorinated chemicals in their products as there is little to no information about what the alternative process or chemicals are.
Tips to reduce exposure to toxicants in cookware
- Never heat food in plastic
- Cook food in oven/freezer proof glass, porcelain, stainless steel, cast iron or ceramic cookware
- Non-stick cookware should be avoided (ceramic lined non-stick cookware is preferred)
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06/10/2020

How To Lower Creatinine Levels, Improve Kidney Function, and Safeguard Your Kidneys From Further Damage - Introducing An All Natural Step-by-Step Program, Proven To Start Healing Your Kidneys Today!
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06/10/2020

Increased risk of folate deficiency with low potassium diets ❗
Folic acid is a water-soluble vitamin that is present in foods such as spinach, chard, kale, sprouts, zucchini, beans, asparagus, tomatoes, mushrooms, sweet potatoes, bananas, avocados and citrus fruits.
Because folic acid is water soluble and not stored in the fat tissues of the body, serum levels may decrease after only a few weeks of eating a diet low in fruit and vegetables. The main cause of folic acid deficiency is a diet low in fruit and vegetables.
Most folic acid sources are also rich in potassium which presents a problem in those people with kidney disease who need to follow a low potassium diet.
This study looked at patients with stages 1 and 2 CKD not on a low potassium diet and patients with stages 3 and 4 CKD who were on a low potassium diet and found that a low potassium diet increased the likelihood of folic acid deficiency three fold and concluded that folate levels should be measured in people on low potassium diets to assess whether folic acid supplementation is required.

Association of low potassium diet and folic acid deficiency in patients with CKD Kamal Hassan1,2 1Faculty of Medicine in the Galilee, Bar-Ilan University, Safed, 2Department of Nephrology and Hypertension, Peritoneal Dialysis Unit, Galilee Medical Center, Nahariya, Israel Background: Most of the fol...

06/10/2020

Is Klotho The New Superstar In Kidney Disease?
In its early stages, kidney disease has no symptoms and isn’t easily detected which makes it difficult to make an early and accurate diagnosis. Frequently, people aren’t aware they have kidney disease until they have already reached the later stages and a lot of kidney damage has already occurred.
Today I want to introduce you to something that has the potential to change the face of kidney disease. There’s a new protein in town (and by new I really mean twenty years old!) that researchers are hoping will provide a new (and more effective) treatment for both acute and chronic kidney disease.
Introducing……….. Klotho!
What is klotho?
Klotho is a protein found in mammals that is produced by the klotho gene. The discovery of klotho was made in 1997 and it was initially identified as an anti-ageing gene.
Like many scientific breakthroughs the discovery of the Klotho gene was a happy accident.
Klotho was originally identified as a gene in mice when a defect in klotho gene expression resulted in effects that resembled human aging, such as cognitive impairment, osteoporosis, atherosclerosis (hardening of the arteries), hearing loss and a shortened life span.
So, while a reduction of the klotho protein in mice were shown to shorten their lifespan, higher levels of klotho actually extended their lifespan. Pretty cool, huh!
Klotho takes its name from the Greek goddess of destiny. Klotho (or Clotho) is one of the Three Fates of Greek mythology who spun the thread of the lives of all mortals, deciding when they were born and when they died. Once you learn a bit more about the klotho gene you will see that this aptly named gene really is intertwined with lifespan (pun intended!).
Klotho levels are closely related to age. Its serum level is low in the first decade; it then increases and reaches its peak in the 20-40 age groups and gradually decreases thereafter.
In humans, klotho deficiency has been associated with the development of atherosclerosis, diabetes, high blood pressure, chronic kidney disease, osteoporosis, anaemia, and various cancers.
The role of Klotho in Kidney Disease
There is unequivocal evidence in experimental animals that both acute kidney injury (AKI) and CKD are states of systemic klotho deficiency.
Most of the klotho in the body is produced in the kidneys which means that the more kidney damage you have, the less klotho they can make which then results in further damage which becomes a bit of a vicious cycle.
Soluble Klotho is the main functional form in the circulation and is detected in the blood, urine and CSF (cerebrospinal fluid).
Evidence shows that klotho deficiency develops in very early stages of CKD as a response to albuminuria (albumin in urine), kidney inflammation or even inflammation elsewhere in the body.
Having a reliable method of measuring klotho levels could potentially be a game changing development that allows for earlier diagnosis of kidney disease which is desperately needed because the earlier we know about a problem, the sooner we can do something about it.
The actions of klotho in the kidneys include:
Protection of the cells in the kidney
Reduction in oxidative stress
Anti-fibrosis- renal fibrosis is like scarring in the kidneys which results in progressive loss of kidney function and can ultimately lead to end-stage renal failure
Reduces inflammation
Protection against vascular calcification and mineral bone disease (a consequence of later stages of kidney disease)
Inhibits phosphate absorption and promotes excretion of phosphate in urine

Klotho was originally identified as a gene in mice when a defect in klotho gene expression resulted in effects that resembled human aging, such as cognitive impairment, osteoporosis, atherosclerosis (hardening of the arteries), hearing loss and a shortened life span. %