19/10/2023
Some interesting facts around the Facet joint :💡
👉 The highly innervated, diarthrodial zygapophysial joint, or the facet joint, is located at either side of the posterior vertebral body. The facet joint’s opposing bony surfaces are covered by a layer of hyaline articular cartilage (thickness: ∼0.5 mm), and the joint is encapsulated by the synovium and fibrous capsule. Whereas the articular cartilage of the facet joint is aneural, the subchondral bone, synovium, synovial folds, and joint capsule are innervated extensively (https://pubmed.ncbi.nlm.nih.gov/22470845/).
👉 This joint can have meniscus-like structures that improve joint congruency. Facet joints work in pairs, along with the intervertebral disc (IVD), to constrain the motion of the vertebrae while aiding in the transmission of spinal loads (https://pubmed.ncbi.nlm.nih.gov/29494214/).
👉 The largely sagittal orientation of the lumbar facet joint, in combination with the high degree of mutual convexity and concavity of the opposing joint surfaces in this region, enables a greater range of motion in terms of flexion, as well as higher resistance to axial rotation (https://pubmed.ncbi.nlm.nih.gov/10529057/).
👉 In the lumbar spine, in vitro studies have shown that the facet articular surface alone bears 6–30% of axial compressive loads, depending on the mode of spinal motion (https://pubmed.ncbi.nlm.nih.gov/20881657/). The cervical and upper thoracic facet joints transmit 23% of axial compressive loads (https://pubmed.ncbi.nlm.nih.gov/3693090/).
👉 Note that loads in the cervical spine are smaller than those in the lumbar spine (cervical spine: flexion/extension: 17–27 N, lateral bending: 17–40 N, axial rotation: 26–30 N versus lumbar spine: flexion/extension: 46–109 N, lateral bending: 10–75 N, axial rotation: 56–120 N. (https://pubmed.ncbi.nlm.nih.gov/24661835/, https://pubmed.ncbi.nlm.nih.gov/24094992/, https://pubmed.ncbi.nlm.nih.gov/16198356/)
👉 Osteoarthritis (OA) of the facet joint is strongly associated with degeneration of the intervertebral disc (IVD) due to their intrinsically linked biomechanics. This relationship was originally described as a cascade of degenerative events initiated either by the facet joints or by the disc (https://pubmed.ncbi.nlm.nih.gov/6210480/). It is believed that facet degeneration usually follows disc degeneration, with facet overloading resulting from disc incompetence (https://pubmed.ncbi.nlm.nih.gov/2326704/, https://pubmed.ncbi.nlm.nih.gov/10552323/).
☝️ However, this sequence has been challenged by studies that have found facet joint OA in the absence of disc degeneration (https://pubmed.ncbi.nlm.nih.gov/21914197/, https://pubmed.ncbi.nlm.nih.gov/17767079/).
☝️ Ironically, surgical treatments of degenerated discs like spinal fusion or disc arthroplasty can encourage facet OA progression and induce adjacent segment disease (ASD, https://pubmed.ncbi.nlm.nih.gov/15534420/, https://pubmed.ncbi.nlm.nih.gov/25102498/, https://pubmed.ncbi.nlm.nih.gov/22706091/, https://pubmed.ncbi.nlm.nih.gov/21587111/).
👉 Degeneration of the facet joint might play a significant role in other back-related morbidities, such as degenerative spinal stenosis (progression of facet joint hypertrophy with subsequent development of osteophytes can compress spinal neural elements, leading to neurogenic intermittent claudication) degenerative spondylolisthesis (https://pubmed.ncbi.nlm.nih.gov/29494214/) and trauma (https://pubmed.ncbi.nlm.nih.gov/22020601/, https://pubmed.ncbi.nlm.nih.gov/28025352/).
📷Illustration: Pathophysiologic aspects of facet joint pain, https://link.springer.com/chapter/10.1007/978-1-4419-0352-5_10
Back pain and neck pain are the most common cause of chronic pain and disability. It is a complex, often multifactorial condition affecting millions of persons worldwide. Disability from spinal pain is associated with a nonspecific diagnosis and suboptimal outcomes....