The Soft Tissue OT, Mount Hawthorn (2025)

20/10/2023

How to improve hip extension 👏🏼

🗣️ We often get asked what is the best way to improve hip extension - particularly for those who seem to default into using their low back to extend instead of true hip extension.

Answer 👉 the prone hip extension exercise.

👀 This exercise is a brilliant way to work hip extension movement, while limiting the ability the the lumbar spine to create movement.

👉 Watch the whole video to find out tips on how to perform this exercise and improve your hip extension 🤗

13/12/2021

🤔 Can you spot why they failed ⁉️
⠀
Take a look 👀 at these two lifters, they have something in common which causes them to fail their lift.
⠀
💪🏼 Muscles receive a neural activation signal from the pool of innervating motor neurons. This neural signal is the sum of the spiking activities of motor neurons and is referred to as neural drive ⚡️
⠀
🏋🏼‍♂️ During the lift, you can see small movements occurring which causes the body to reduce 📉the neural drive (the power supply) to the muscles. Listen 👂 to Professor Stuart McGill, renowned spinal expert, explain this in more detail.
⠀

13/06/2020

🍁WINTER WARMER GIVEAWAY🍁

To have some fun and celebrate the colder months, we're giving away a winter warmer hamper.

✅2 SIMPLE STEPS TO ENTER:
👍LIKE Enhanced Occupational Therapy page
🙌SHARE & LIKE this post

🎁BONUS entry if you TAG your friends in this post!

The lucky winner will be announced on Saturday 11th July 🤞

28/08/2019

Welcome to Worth-a-Word Wednesdays, where I review and summarise a study or article 📖 that I think provides some useful and interesting information. This week – The association of saturated fat with cardiovascular disease! ❤️

Article Title: Meta-analysis of prospective cohort studies evaluating the association of saturated fat with cardiovascular disease

This study was published by Siri-Tarino, P. W., Sun, Q., Hu, F. B., & Krauss, R. M. (2010). In the American Journal of Clinical Nutrition, 91(3), 535-546.

This meta-analysis evaluated the evidence related to dietary saturated fat and its association with risk of coronary heart disease (CHD), stroke, and cardiovascular disease (CVD).

21 studies qualified for this analysis and the results are as follows;
•Markedly, studies that reported positive associations between saturated fat and CVD were specific to subgroups of the study population i.e- older vs younger participants, male vs female participants. 🧐
•10 of the studies evaluated did not report any statistically significant association between dietary saturated fat and CHD
•The authors stated that their results suggested publication bias 🚫, as that studies that showed an increased association between saturated fat and CVD were inclined to receive more favourable publication, 😮 “Our results suggested publication bias, such that studies with significant associations tended to be received more favourably for publication. If unpublished studies with null associations were included in the current analysis, the pooled relative risk estimate for CVD could be even closer to null.” 👍
•The analysis concluded that there is insufficient evidence from prospective studies to conclude that dietary saturated fat is associated with an increased risk of CHD, Stroke or CVD.👀

This is not a substitute for independent professional advice. Nothing contained in this post is intended to be used as medical advice and it is not intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professional's advice. The editor of this post and the account owner do not accept any liability for any injury, loss or damage incurred by use of or reliance on the information provided on this post.

22/08/2019

Dry Needling is a useful tool to aid and assist in recovery or management of conditions and injuries, including;
✔️Elbow pain
✔️Shoulder pain/dysfunction
✔️Hip pain
✔️Neck pain
✔️Acute injury
✔️Knee pain
✔️Calf issues
✔️Chronic conditions
✔️Pain management
✔️And more...

Book in with Jordan today to see how needling could benefit you!

21/08/2019

Welcome to Worth-a-Word Wednesdays, where I review and summarise a study or article 📖 that I think provides some useful and interesting information. This week – Low carb vs moderate carb, low fat diet and type 2 diabetes!

Article Title: Twelve-month outcomes of a randomized trial of a moderate-carbohydrate versus very low-carbohydrate diet in overweight adults with type 2 diabetes mellitus or prediabetes.

This study was published by Saslow, L. R., et al. (2017) In Nutrition and Diabetes 7(12), 304.

Today we are talking about a 2017 study of Low-Carbohydrate Ketogenic diet (LCk) versus a Moderate Carbohydrate, Calorie Restricted, low-fat diet (MCCR) and type 2 diabetes. Participants with type 2 diabetes, glycated haemoglobin 🍬 (a form of haemoglobin that is chemically linked to sugar) >6.0% and increased body weight (BMI > 25) we randomly put into two groups, with different diets 🍽 ; LCK and MCCR. After 12 months, the study found the following;

•LCK group had a greater reduction in glycated haemoglobin
•Participants in the LCK group lost more weight ⬇️ (an average of 8kgs in LCK vs 1.7kgs in MCCR)
•LCK group experienced a larger reduction in the amount of diabetes-related medication they required 🧪
•Specifically, of the participants who took sulfonylureas or dipeptidyl peptidase-4 inhibitors (diabetes related medication 💉) at the beginning of the study, 6/10 in the LCK group discontinued use of these medications compared to 0/6 in the MCCR group.

This is not a substitute for independent professional advice. Nothing contained in this post is intended to be used as medical advice and it is not intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professional's advice. The editor of this post and the account owner do not accept any liability for any injury, loss or damage incurred by use of or reliance on the information provided on this post.

07/08/2019

Welcome to Worth-a-Word Wednesdays, where I review and summarise a study or article 📖 that I think provides some useful and interesting information. This week – Part II of Parkinsons disease and the Gut!

Article Title: Transneuronal Propagation of Pathologic α-Synuclein from the Gut to the Brain Models Parkinson’s Disease.

This study was published by Kim, S. et al. (2019). In Neuron.

This study followed on from the postulate that α-synuclein 🧬(misfolded protein) pathology could spread from the gut to brain via the vagus nerve 🧠
As more of these misfolded proteins clump together, they cause nerve cells to die resulting in large bundles of dead brain matter known as Lewy bodies.

The researchers injected 💉 the misfolded proteins into the gut of healthy mice 🐁 and evaluated the mice brain tissue at different time intervals. Some mice were the control group, some mice had their vagus nerve cut and some had their vagus nerve left intact.

The mice that were injected with the misfolded proteins and with an intact vagus nerve:
❌The misfolded proteins began to build up where the vagus nerve connected to the gut
❌From there it began to spread its way up the nerve to parts of the brain

The mice that were injected with the misfolded proteins and with a cut 🔪 vagus nerve:
✅The mice showed none of the signs of cell death that the mice with intact vagus nerves displayed
✅The severed nerve appeared to halt the advance of the misfolded protein.

Furthermore, the researchers evaluated whether these physical changes in Parkinson’s Disease (PD) progression affected the cognitive behaviour of the mice and found that the mice exhibiting PD progression showed:
Reduced fine motor ability
Exhibited higher levels of anxiety related behaviours

More clinically 👩‍🔬, the study reported the following:
•Gut-to-brain propagation of pathologic α-synuclein via the vagus nerve causes PD
•Dopamine neurons degenerate in the pathologic α-synuclein gut-to-brain model of PD
•Gut injection of pathologic α-synuclein causes PD-like motor and non-motor symptoms
•PD-like pathology and symptoms require endogenous α-synuclein

Overall, the results of this study show that misfolded alpha-synuclein 🧬 can be transmitted from the gut to the brain in mice along the vagus nerve, and blocking the transmission route could be key to preventing the physical and cognitive manifestations of Parkinson's disease .

This is not a substitute for independent professional advice. Nothing contained in this post is intended to be used as medical advice and it is not intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professional's advice. The editor of this post and the account owner do not accept any liability for any injury, loss or damage incurred by use of or reliance on the information provided on this post.

31/07/2019

Welcome to Worth-a-Word Wednesdays, where I review and summarise a study or article 📖 that I think provides some useful and interesting information. This week – Parkinsons disease and the Gut!

Article Title: Vagotomy and subsequent risk of Parkinson's disease.
This article was published by Svensson, E., et al. (2015). In Annals of Neurology, 78(4), 522-529.

Today we are talking about a 2015 article that examined the relationship between the gut and Parkinson’s Disease (PD). 👩‍🔬 The study looked at close to 15,000 people whom have had their vagus nerve in their stomach cut ✂️

•The study examined participants whom have previously had their vagus nerve severed (this used to be a common treatment for ulcers 💉)
•The vagus nerve provides a link between organs (such as the stomach) and the brain. It provides communication between the brain and the gut 🧠
•The article explained that many patients often suffer from gastrointestinal symptoms before receiving their diagnosis of PD
•The study provides strong evidence to suggest that PD originates in the gastrointestinal tract and spreads to the brain via the vagus nerve
•The study shows that patients who have had the entire vagus nerve severed were protected against PD. Their risk was halved after 20 years 〽️
•Patients whom had only a small part of the vagus nerve cut were not protected from PD 🚫
•Full truncal vagotomy is associated with a decreased risk for subsequent PD, suggesting that the vagus nerve may be critically involved in the pathogenesis of PD.

*This study has sparked more interest into investigating the pathogenesis of PD and its relation to the gut. More recent studies have evaluated this with promising results – stay tuned for next weeks ‘Worth-a-Word’ 👀 as we discuss the very latest study of the Gut and PD.

This is not a substitute for independent professional advice. Nothing contained in this post is intended to be used as medical advice and it is not intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professional's advice. The editor of this post and the account owner do not accept any liability for any injury, loss or damage incurred by use of or reliance on the information provided on this post.

30/07/2019

The rotator cuff muscles 💪
These relatively small muscles play a massive role in the body – they stabilise the shoulder. As your shoulder is the most moveable joint in the body, these guys have a rather hefty job.

Due to their intensive job, they can often become injured – this can cause or contribute to;
•Rotator cuff tendinitis and tendinopathy ❌
•Rotator cuff tears ❌
•Sub-acromial pain syndrome/ shoulder impingement ❌
•Adhesive capsulitis (frozen shoulder) ❌
•Shoulder Bursitis and more… ❌

These conditions or injuries can often be prevented. So, if you have any shoulder issues or pain be sure to book your appointment today and don’t let it develop into something nasty! Keep these muscles happy to keep your shoulders happy 😊

24/07/2019

Welcome to Worth-a-Word Wednesdays, where I review and summarise a study or article 📖 that I think provides some useful and interesting information. This week – Nothing Boring about Boron!

Article Title: Nothing Boring About Boron. 😁

This article was published by Pizzorno, L. (2015). In Integrative Medicine: A Clinician's Journal, 14(4), 35.

Today we are talking about a 2015 article that discusses the many mechanisms and benefits that boron has on the body.

The key findings of the article are as follows;
•Is essential for the growth and maintenance of bone 🦴
•Greatly improves wound healing
•Beneficially impacts the body’s use of estrogen, testosterone, and vitamin D
•Boosts magnesium absorption
•Reduces levels of inflammatory biomarkers
•Raises levels of antioxidant enzymes,
•Protects against pesticide-induced oxidative stress and heavy-metal toxicity ☣️
•Improves the brains electrical activity 🧠
•Cognitive performance, and short-term memory for elders
•Influences the formation and activity of key biomolecules 🧬
•Has demonstrated preventive and therapeutic effects in a number of cancers, such as prostate, cervical, and lung cancers, and multiple and non-Hodgkin’s lymphoma
•May help ameliorate the adverse effects of traditional chemotherapeutic agents
•Recommend a daily intake of 3mg of Boron a day.

This is not a substitute for independent professional advice. Nothing contained in this post is intended to be used as medical advice and it is not intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professional's advice. The editor of this post and the account owner do not accept any liability for any injury, loss or damage incurred by use of or reliance on the information provided on this post.

17/07/2019

Welcome to Worth-a-Word Wednesdays 🤓, where I review and summarise a study or article 📖 that I think provides some useful and interesting information. This week – Dietary ketosis and memory! 🧠

This one goes out to all those Ketogenic and Banting people out there 😉

Article Title: Dietary Ketosis enhances memory in mild cognitive impairment.

This study was published by Krikorian, R., et al. (2012) in Neurobiology of aging, 33(2), 425-e19.

Today we are talking about a 2012 study of dietary ketosis and its effects on memory for people with mild cognitive impairment. The participants of this study were put on either a high carbohydrate or very low carbohydrate diet. What this study found;
•Improved verbal memory performance for the low carbohydrate participants
•Reductions ⬇️ in weight for the low carbohydrate participants
•Reduction ⬇️ in waist circumference, fasting glucose and fasting insulin for the low carbohydrate participants
•Ketone levels were positively correlated with memory performance
• Final comment from study, "These findings suggest that very low carbohydrate consumption, even in the short-term, can improve memory function in older adults with increased risk for Alzheimer’s disease."

*Further investigation is needed into this area however it seems to have a great potential!

This is not a substitute for independent professional advice. Nothing contained in this post is intended to be used as medical advice and it is not intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professional's advice. The editor of this post and the account owner do not accept any liability for any injury, loss or damage incurred by use of or reliance on the information provided on this post.

10/07/2019

Welcome to Worth-a-Word Wednesdays, where I review and summarise a study or article 📖 that I think provides some useful and interesting information. This week – Fructose consumption and weight gain! 🌽🍏

Article Title: Effects of Fructose Vs Glucose on Regional Cerebral Blood Flow in Brain Regions Involved with Appetite and Reward Pathways

This article was authored by Page, K. A., et al. (2013) in the Journal of the American Medical Association, 309(1), 63-70.

Today we are talking about an article that looked at neurophysiological 🧠 factors that might underlie the association between fructose consumption and weight gain. The consumption of fructose was compared against glucose and the following was identified;

•Fructose ingestion did not reduce the activation of the hypothalamus, insula, and striatum—regions of the brain that regulate appetite, motivation, and reward processing
•Glucose ingestion increased brain activity which signaled increased satiety (feeling satisfied)
•The responses to fructose were associated with reduced systemic levels of the satiety-signalling hormone insulin
•Not only did fructose fail to diminish hypothalamic activity (which leads to reduced hunger/increased satiety) but it instead induced a small, transient increase in hypothalamic activity
•Fructose ingestion did not result in deactivation of the striatum. Hypothalamic and striatal deactivation occur when initially hungry individuals reach satiety

In other words, when you eat fructose it does not reduce hunger or increase satiety – which can result in you eating more 🍽 and feeling less satisfied…

This is not a substitute for independent professional advice. Nothing contained in this post is intended to be used as medical advice and it is not intended to be used to diagnose, treat, cure or prevent any disease, nor should it be used for therapeutic purposes or as a substitute for your own health professional's advice. The editor of this post and the account owner do not accept any liability for any injury, loss or damage incurred by use of or reliance on the information provided on this post.

The Soft Tissue OT

20/10/2023

13/12/2021

13/06/2020

28/08/2019

22/08/2019

21/08/2019

07/08/2019

31/07/2019

30/07/2019

24/07/2019

17/07/2019

10/07/2019

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