Art Therapy for Autism, Dyslexia, ADHD and Other Neurodiversities

Art Therapy for Autism, Dyslexia, ADHD and Other Neurodiversities Lynsey Baughen, an Art Therapist embracing working NOR while supporting a neurodivergent community.

21/08/2025
21/08/2025

New Opinion Piece on the EAC website: Autistic children don’t break the budget. Broken systems do.
This week, the government announced the new “Thriving Kids” program, alongside plans to change how children with disability access the NDIS.
In this opinion piece, Heidi La Paglia challenges the myths and highlights what’s really putting pressure on the system.
📖 Read the full opinion piece: https://everyaustraliancounts.com.au/opinion/autistic-children-dont-break-the-budget/

[Image description: A graphic with a red textured background features a white paper-like overlay. At the top is the "Every Australian Counts" logo. The text reads: “Opinion: Autistic Children Don’t Break the Budget. Broken Systems Do.” by Heidi La Paglia. At the bottom right is a circular photo of Heidi La Paglia smiling.]

21/08/2025

Like many of you, we have heard the recent NDIS reform announcement that includes removing children with “mild and moderate autism” and sending them to the new Thriving Kids program.

We are still processing this news and trying to gather more information. At this stage, there are many unanswered questions about this new program, like:
• How will Autistic children and families be supported?
• Will the approach be neuro-affirming?
• Will there be genuine involvement of Autistic people in shaping the program’s design?

As an Autistic-led organisation, we believe that any program for Autistic people must be co-designed with Autistic people. Without this, there is a real risk that supports will not meet the needs of our community.

We want to acknowledge that uncertainty naturally brings concern, fear and worry, and many families are feeling this right now. We remain dedicated to advocating for Autistic voices to be heard and Autistic children to be best supported. We will continue to connect with and serve the Autistic community in whatever form the reforms take.

We’d like to hear from our neurokin and our allies: How are you feeling about these changes and what would you like to see?

20/08/2025

Children with mild autism and developmental delays will be removed from the ballooning NDIS and placed in a new capped program.

WHAT WE KNOW ABOUT THE NEW "THRIVING KIDS" PROGRAM SO FARThe Albanese Government has announced a major change to how sup...
20/08/2025

WHAT WE KNOW ABOUT THE NEW "THRIVING KIDS" PROGRAM SO FAR

The Albanese Government has announced a major change to how support will be delivered for children with mild to moderate autism and developmental delay. Instead of entering the NDIS, these children will move into a new $2 billion program called “Thriving Kids.”

Here’s what’s been outlined across Minister Butler’s press club speech (Health.gov.au, 20 Aug 2025), ABC News (20 Aug 2025), The Guardian, News.com.au, and 9News:

🔹 When will this happen?
* The rollout of Thriving Kids will begin in 2026.
* Families will still have a choice during a one-year transition.
* The full change—where children with mild/moderate needs are no longer placed on the NDIS—won’t take
effect until mid-2027.

🔹 What is Thriving Kids?
* A foundational support program designed to provide early intervention, therapies, and community-based supports outside of the NDIS.
* Focused on helping children with developmental delay or mild/moderate autism thrive through schools, health services, and local providers.
* Backed by joint state and federal funding.

🔹 Why is this happening?
* According to Minister Butler, the NDIS is growing faster than sustainable—autism and developmental delay accounted for 70% of new child participants in 2024.
* Some children were receiving more therapy sessions than clinical guidelines recommend.
* Thriving Kids is intended to better target services while keeping the NDIS focused on people with significant and permanent disabilities.

🔹 What about adults?
* Work is underway on a separate foundational supports program for adults with severe and complex mental illness, acknowledging the need for system-wide reform.

This is a major shift. Many families, providers, and advocacy groups are now asking how the Thriving Kids model will be funded, delivered, and whether it will truly meet the needs of children who might otherwise have accessed the NDIS.

📰 Sources: Health.gov.au – Minister Mark Butler National Press Club Speech (20 Aug 2025), ABC News, The Guardian, News.com.au, 9News.

https://youtu.be/8__AWHcff54

Hundreds of thousands of children with autism will be diverted away from the NDIS under a new scheme announced by the federal government. Federal Health Mini...

⚠️NDIS SET TO MOVE “CHILDREN WITH MILD TO MODERATE AUTISM” OFF THE SCHEME⚠️The federal government has announced plans to...
20/08/2025

⚠️NDIS SET TO MOVE “CHILDREN WITH MILD TO MODERATE AUTISM” OFF THE SCHEME⚠️

The federal government has announced plans to move children with mild to moderate autism, or developmental delays, from the NDIS into a new support program called “Thriving Kids.” This program, backed by $2 billion in funding, will be rolled out in partnership with state and territory governments to deliver early intervention and community-based supports.

NDIS providers are set to respond to the changes at 6pm tonight.

📺 Watch here: https://youtu.be/3HuBuo8MvOc

NDIS National Disability Insurance Scheme 7NEWS Perth

The Australian government is implementing a major crackdown on the National Disability Insurance Scheme (NDIS), removing access for children under nine with ...

…it’s not that simple
19/08/2025

…it’s not that simple

“If you swear one more time…”
05/08/2025

“If you swear one more time…”

03/08/2025

Concerning: Simon Baron-Cohen

I’ve had a lot of people quote Simon Baron-Cohen to me lately, often using his credentials to dismiss autistic lived experience. In that light, I felt compelled to share some thoughts (and these are not merely my thoughts) on why his continued influence is so concerning.

When I was a psychology undergraduate, Simon Baron-Cohen was required reading. His theories were central to my studies, and he was recognised as the foremost authority on autism. He shaped much of the mainstream thinking, and still has a profound impact on how we’re seen by the world.

He’s best known for promoting the Theory of Mind deficit, the idea that autistic people can’t understand others’ thoughts or emotions, and the Extreme Male Brain theory, which tries to explain autism as an exaggeration of stereotypical male traits.
both theories frame autism as a deficit and have been widely criticised for reinforcing gendered stereotypes that marginalise autistic women and ignore lived experience.

He never listened to autistic people to develop these theories - he focused his attention on a (homogenous, gender-exclusive) group of autistic people, spit balled some thoughts about why they were so “broken”, and has continued to regurgitate that for more than 3 decades.

Even as an undergrad, his theories didn’t sit right with me. I just didn’t yet have the understanding to explain why they were so fundamentally flawed. I know now that the reason is his biased perspective; his theories are all deficit-based. His studies are built around observations from the outside looking in, with neuronormative behaviour as the standard and any deviation framed as a deficit, as degrees away from the right kind of brain.

Simon Baron-Cohen’s work builds on, and seeks to explain, the Triad of Impairments (Wing & Gould, 1979), which defines autism as involving deficits in three key areas: social interaction, social communication, and imagination.

This narrow, biased perspective fails to acknowledge that many of these so-called “impairments” emerge from being autistic brains being forced to exist in environments that are inaccessible, unaccommodating, and harmful. It’s like saying a sunflower is deficient in growing capability because it wilts in the Sahara.

Now to be clear, I’m not criticising him for not knowing what we know now. When he published much of this work, concepts like monotropism, sensory integration, and the double empathy problem hadn’t yet gained mainstream recognition. The issue isn’t not knowing then; it’s that he knows NOW and yet still clings to these outdated theories. He has disregarded better, more inclusive understanding as it has emerged because it doesn’t validate his own flawed research.

The double empathy problem, for example, shows that communication breakdowns between autistic and non-autistic people go both ways. Yet Baron-Cohen’s work continues to place the full burden on autistic people, that autistic people are deficient in communication because we lack Theory of Mind. His work continues to ignore that neurotypical people struggle to understand autistic people as frequently as autistic people struggle to understand neurotypical people; it continues to ignore that autistic-to-autistic communication tends to be far more effective than neurotypical-to-neurotypical communication. Regardless of the data, he continues to treat autistic communication norms as being deficits rather than differences.

Why does this matter? Because his work has helped define how autistic people have been perceived for decades. It was highly influential in defining the diagnostic criteria in the DSM-IV, influenced education and therapy approaches. It has shaped mainstream media portrayals. He is viewed as the expert (despite his own lack of scientific rigour) because he talked about autism when no one else did, and when absolutely no one thought to speak to the actually autistic people. More recently, he led the Spectrum 10K genetic study, which sparked huge backlash due to ethical concerns and a complete lack of autistic involvement - again, excluding us from our own narrative. The study has since been cancelled, but the damage to trust remains. (It’s simple, Simon: Nothing about us, without us.)

You might ask, “Why judge SBC so harshly?” After all, Freud was wrong about a lot, and we still credit him for shaping the field. But Freud evolved, and importantly, he acknowledged when he was wrong. That’s the difference. With what we now understand about monotropism, the double empathy problem, sensory processing, and the massive impact of environmental factors, the flaws in his work are clear. But he doesn’t acknowledge that. He is complicit in allowing harmful and outdated information to define how the world sees us, not simply for the research he did, but for refusing to revise it in light of new knowledge.

His work treats autistic people like puzzles to be solved, rejecting the complexity and fullness of the internal autistic experience. You might ask, “How much harm could that be doing?” A LOT. Because his theories are still used to underpin autism interventions in education and mental health. And they still influence how we’re portrayed in mainstream media - stigmatising, isolating, dehumanising portrayals. Baron-Cohen gave the world the “Extreme Male Brain” theory, and in return, we get characters like The Good Doctor — analytical, emotionally distant, robotic, devoid of humanness until filled with “normality” by the neurotypical people who take pity on the poor broken soul. His comments and musings are taken as fact and used against autistic people all the time. His depiction of autistic women as unfeeling has been highly influential in autistic women being denied custody of their children in the UK. Following seeing new data (by other-less-biased researchers) he posted on Twitter to say it showed that “Sometimes, autistic women can even be good mothers” (yes, he actually said that) - an opinion then reshared widely across social media to validate the view that autistic women shouldn’t be allowed to reproduce.

This barely scratches the surface of the harm he’s caused. The most damaging thing isn’t that he got it wrong before, it’s that, in the face of overwhelming rejection from the people he claims to represent, he steadfastly refuses to let go now of the theories that built his career.

And, really, this is the heart of the problem with SBC - he is in the exceptionally privileged position of being a world-renowned academic with the power to shape public perception, a power he could use to make sure autistic voices are heard. Instead, continues to climb on the backs of autistic people to push himself higher; he uses our community as the pedestal upon which he places himself to be admired. Despite widespread rejection of his theories by the neurodivergent community, and even significant criticism from his peers, SBC clings to the same deficit-based narrative, perpetuating the idea that autistic people are less, that our communication style is deficient, and that we need to be fixed. In science terms, he actively disregards relevant first-hand data (autistic peoples’ accounts of internal experience) in favor of relying on second-hand observations. Why would he do this? Because the quality first-hand data disproves his hypotheses and to acknowledge it would be to dislodge himself from being central to the discussion.

It’s just objectively bad science.

And it tells you everything you need to know about how he views us.

This might all sound like it’s personal for me. Yes, this is personal. His refusal to acknowledge the flaws in his theories harms autistic people every day. His refusal to centre autistic voices harms autistic people every day. He declared himself the leading expert on being autistic without ever seeing autistic people as humans; it would be like Data on ST:TNG declaring itself an expert on human emotional regulation based on observations and then telling all the humans that they’re broken if their experiences don’t match his expectations.

To be very, very clear: autistic people do not lack empathy. We’re not broken versions of “normal.” We’re not failing to manage our deficits effectively.

We are neurodivergent - different, not less than - and we deserve research and representation that reflects that.

Thankfully, there are autistic researchers, writers, and advocates doing that work — shaping a future grounded in lived experience, not just clinical theory.

If you’re looking to grow your understanding of the autistic experience — especially if you’re studying psychology or working with autistic people — please don’t look to Simon Baron-Cohen for anything beyond historical context. His work may have shaped where we’ve come from, but it does not represent where we’re going.

I’ll be compiling a list of books and sources on autism, masking, neurodivergence, and monotropism, and pinning it in the comments.
It’s gotten later than I planned, so that’ll likely go up tomorrow.

If you’re still here, thanks for reading.
— John


Emergent Divergence: The neurodivergent ramblings of David Gray-Hammond
The Autistic Advocate

30/07/2025

Juggling all of life’s things is not easy. Keep an eye on your capacity and don’t feel bad for making adjustments or taking breaks when you need to. Human-ing is hard 🤹

NEW CLINICAL PRACTICE GUIDELINES- AGA Practice Update on GI Manifestations and Autonomic or Immune Dysfunction in Hyperm...
30/07/2025

NEW CLINICAL PRACTICE GUIDELINES- AGA

Practice Update on GI Manifestations and Autonomic or Immune Dysfunction in Hypermobile Ehlers-Danlos Syndrome

A new Clinical Practice Update published in Clinical Gastroenterology and Hepatology from the American Gastroenterological Association and a group of experts provides long-overdue guidance for clinicians treating patients with hypermobile Ehlers-Danlos syndrome (hEDS) or hypermobility spectrum disorders (HSDs) who also experience gut-brain interaction disorders (DGBIs). There are 16 best practice advice statements in the publication.

16 Best Practice Advice Statements

CLINICAL ASSOICIATIONS: Clinicians should be aware of the observed associations between hEDS or HSDs and POTS and/or MCAS and their overlapping gastrointestinal (GI) manifestations.

WHO TO TEST? Testing for POTS/MCAS should be targeted to patients presenting with clinical manifestations of POTS/MCAS, but universal testing for POTS/MCAS in all patients with hEDS/HSDs is not supported by the current evidence.

SCREENING FOR HYPERMOBILITY/HEDS: Gastroenterologists seeing patients with DGBI should inquire about joint hypermobility and strongly consider incorporating the Beighton score for assessing joint hypermobility into their practice as a screening tool; if the screen is positive, gastroenterologists may consider applying 2017 diagnostic criteria to diagnose or refer to specialist.

TESTING FOR POTS: Testing for POTS through postural vital signs and referral to specialty practices for autonomic testing should be considered in patients with hEDS/HSDs and refractory GI symptoms who also report orthostatic intolerance after exclusion of medication side effects and appropriate lifestyle or behavioral modifications have been attempted but is not required for all patients with hEDS/HSDs who report GI symptoms alone.

CELIAC TESTING: Testing for celiac disease may be considered earlier in the diagnostic evaluation of patients with hEDS/HSDs who report a variety of GI symptoms and not only limited to those with diarrhea. There is insufficient research to support routine testing for disaccharidase deficiencies or other diet-mediated mechanisms as causes of GI symptoms in hEDS/HSDs.

FUNCTIONAL DEFECATION DISORDERS: Diagnostic testing for functional defecation disorders with anore**al manometry, balloon expulsion test, or defecography should be considered in patients with hEDS/HSDs and lower GI symptoms such as incomplete evacuation given the high prevalence of pelvic floor dysfunction, especially re**al hyposensitivity, in this population.

GASTRIC EMPTYING: In patients with hEDS/HSDs and comorbid POTS who report chronic upper GI symptoms, timely diagnostic testing of gastric motor functions should be considered after appropriate exclusion of anatomical and structural diseases, as abnormal gastric emptying may be more common than in the general population.

MEDICAL MANAGEMENT: Medical management of GI symptoms in hEDS/HSDs and POTS/MCAS should focus on treating the most prominent GI symptoms and abnormal GI function test results. In addition to general DGBIs and GI motility disorder treatment, management should also include treating any symptoms attributable to POTS and/or MCAS.

TESTING FOR MCAS: In patients presenting to gastroenterology providers, testing for mast cell disorders including MCAS should be considered in patients with hEDS/HSDs and DGBI who also present with episodic symptoms that suggest a more generalized mast cell disorder involving 2 or more physiological systems, but current data do not support the use of these tests for routine evaluation of GI symptoms in all patients with hEDS/HSDs without clinical or laboratory evidence of a primary or secondary mast cell disorder.

TRYPTASE TESTING: If MCAS is suspected, diagnostic testing with serum tryptase levels collected at baseline and 1-4 hours following symptom flares may be considered by the gastroenterologist; increases of 20% above baseline plus 2 ng/mL are necessary to demonstrate evidence of mast cell activation.

ADDITIONAL ALLERGY TESTING: If a diagnosis of MCAS is supported through clinical and/or laboratory features, patients should be referred to an allergy specialist or mast cell disease research center where additional testing may be performed.

DGBI EVALUATION: Diagnostic evaluation of GI symptoms consistent with DGBI in patients with hEDS/HSDs and comorbid POTS and/or MCAS should follow a similar approach to the evaluation of DGBI as in the general population including the use of a positive symptom-based diagnostic strategy and limited noninvasive testing.

TREATMENT OF POTS: Treatment of POTS may include increasing fluid and salt intake, exercise training, and use of compression garments. Special pharmacological treatments for volume expansion, heart rate control, and vasoconstriction with integrated care from multiple specialties should be considered in patients who do not respond to conservative lifestyle measures.

TREATMENT OF MCAS: When MCAS is suspected, patients can benefit from treatment with histamine receptor antagonists and/or mast cell stabilizers, in addition to avoiding triggers such as certain foods, alcohol, strong smells, temperature changes, mechanical stimuli, emotional distress, or specific medications.

NUTRITION: Besides general nutritional support, special diets including a gastroparesis diet and various elimination diets can be considered for improving GI symptoms. Dietary interventions should be delivered with appropriate nutritional counseling or guidance to avoid the pitfalls of restrictive eating.

MANAGEMENT OF GI SYMPTOMS: Management of chronic GI symptoms in patients with hEDS/HSDs who do not exhibit symptoms consistent with POTS or MCAS should align with existing approaches to management of DGBI and GI motility disorders in the general population, including integrated multidisciplinary care involving multiple specialties, where appropriate.

https://pubmed.ncbi.nlm.nih.gov/40387691/

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