Movability - Wellness & Sport Sciences

Movability - Wellness & Sport Sciences Chiropractic, pelvic floor physiotherapy, acupuncture, massage, orthotics, naturopathic care, and rehab all under one roof.

We treat complex conditions through full-body assessment, root-cause care, and a collaborative, patient-first approach.

03/06/2026

If your neck feels like it’s wearing a concrete collar after a day at the desk, stretching is not always the answer.

Here’s the missing piece for a lot of desk workers: nerves have to move.

Think of a nerve like a cable that should slide inside a sleeve. It is not designed to be pinned down. If it gets compressed, irritated, or stuck, the system reduces motion to keep it safe.

The radial nerve runs down the arm and passes through a tight neighborhood near the outside of the elbow called the radial tunnel. Hours of mousing, gripping, and holding the wrist in one position can make that area irritated or “sticky.” When a nerve is sensitive or doesn’t glide well, the nervous system treats normal movement like a threat.

So it protects you.

Protection usually shows up as muscle guarding, not as numbness. The forearm tightens, the shoulder hikes, the upper trap turns on, and the neck stiffens to reduce nerve strain. The neck feels tight, but the system is actually putting a brake on the whole chain.

That’s why some people get temporary relief from stretching or massage, then the stiffness snaps right back when they return to the same exposure.

In this video I’m showing a gentle radial nerve self mobilization around the radial tunnel region. The goal is not to press on a nerve or “stretch harder.” It’s to calm sensitivity and improve glide so the nervous system stops bracing. Done correctly this should feel relieving or neutral, and any symptoms should settle quickly when you stop.

Try this, then retest your neck rotation. If it feels easier, that’s a clue the nervous system just took the brake off.

Stop and get assessed if you notice worsening numbness or tingling, symptoms that linger, new weakness, dropping objects, or symptoms spreading.

I’m Dr. Sina and I’ve treated nerve entrapment patterns for over a decade. This distal driver, proximal stiffness pattern is one of the most common traps I see in desk workers. This mechanism applies to all nerves.

Want the full Root Cause Breakdown with anatomy, mechanisms, assessment logic, and how I sequence treatment? I posted the long form deep dive on my Substack, Movability Masterclass. LINK IN BIO ⬆️

03/04/2026

Childhood Stress, Adult Gut Symptoms

Many adults with “mysterious” gut issues were first children with stomachaches no one knew how to translate.

As kids, we do not say, “My nervous system is overwhelmed.”
We say, “My stomach hurts.”

And that is not just poetic, it is biological.

A child’s brain and gut are in constant conversation through the gut-brain axis: the vagus nerve, stress hormones, immune signaling, gut motility, intestinal barrier function, and the microbiome. So when a child lives with chronic stress, fear, unpredictability, neglect, or trauma, the body does not file that away as memory alone. It can start encoding it into physiology.

That is why childhood distress so often shows up as nausea, constipation, diarrhea, cramping, or recurrent stomach pain with no obvious “cause.” The body is speaking long before the child has the language, safety, or support to explain what is happening.

Then adulthood teaches us how to override those signals.

Keep going.
Be productive.
Be easy to be around.
Ignore the knot in your stomach.
Call your shutdown maturity.

But suppression is not healing.

Evidence on early life stress and childhood adversity shows associations with altered stress responses, autonomic imbalance, visceral hypersensitivity, changes in gut motility, inflammatory signaling, and intestinal permeability. Later in life, that pattern can show up as IBS, reflux, chronic bloating, functional abdominal pain, and a body that stays braced for danger even when the danger is over.

So maybe your gut feeling was never irrational.
Maybe it was data.

Children feel the truth in the belly because they have not yet learned how to betray themselves.
Adults learn to disconnect because life demands performance.
And sometimes symptoms are what happens when the body keeps telling the truth after the mind has been trained to stay silent.

I broke the full systems map behind this pattern down in this week’s Movability Masterclass on Substack, including the physiology, what gets missed, and how I think through it clinically. LINK IN BIO ⬆️

Did your body learn to speak in symptoms before it ever felt safe enough to speak in words?

Dr. Sina

03/01/2026

Today feels heavy.

For many of us, Iran is not a headline. It is our parents, our siblings, our grandparents, our childhood, our home. Watching everything unfold from far away brings fear, grief, anger, and survivor guilt all at once. If you feel it in your body, that makes sense. Stress can amplify headaches, migraines, chest tightness, jaw and shoulder pain, back pain, tingling, stomach issues, poor sleep, and a racing heart.

But hear me clearly. Breaking yourself down with stress does not help the people of Iran. Our families back home need us steady, healthy, and clear so we can keep speaking, keep supporting, and keep showing up.

Respect real red flags. If you experience chest pain or pressure, trouble breathing, fainting, sudden one sided weakness or numbness, trouble speaking, vision changes, or a sudden severe headache, do not assume it is anxiety. Get urgent care.

If you are medically cleared, protect your nervous system deliberately. Take your medications as prescribed. Do not skip doses because you are overwhelmed. If you have migraines, asthma, diabetes, heart disease, autoimmune conditions, anxiety, or depression, this is the time to be consistent. Call your doctor if something feels off. Refill early if you can. Keep your medication list on your phone.

Stay informed, but protect your body from nonstop graphic input. When you feel yourself spiraling, switch to text updates for a while. Feet on the floor. Jaw unclenched. Shoulders down. Exhale longer than you inhale. Take a slow walk. Do light stretching. Avoid intense workouts for now. Eat real food. Hydrate. Sleep when you can.

There are moments in history that shift something overnight. Today carries that kind of weight. There is a quiet sense that the change we have been hoping for may finally be closer. And at the same time, there is fear for our families and uncertainty about what comes next. Hope and worry are sitting side by side. Both are real.

We can grieve innocent lives and still believe in a future for Iran rooted in dignity and safety.

Our people back home need us standing.

Stay healthy. Stay steady. Stay strong.

Dr. Sina

02/27/2026

POV: I’m checking your neck and you casually say, “Oh btw, I have endometriosis.” 😵‍💫

That’s my cue to zoom out, because endo is not just period pain. I see this hidden-driver pattern constantly.

In a lot of people endo behaves like a whole-body inflammatory + neuroimmune pattern, so pain can show up as migraines, jaw tension, ribs, back, hips, shoulders, and yes, neck.

How the dots connect:
🔥 Inflammation and prostaglandins can sensitize nerves and tissues.
🧠 Central sensitization can develop over time, real physiology, not “in your head,” and not a reason to stop investigating.
⚡️ Neuropathic patterns (sometimes small fiber dysfunction): burning, buzzing, electric shocks, temperature or touch sensitivity.
🌀 Autonomic and histamine patterns can amplify flares: dizziness, palpitations, GI chaos, “wired but exhausted.”
🫁 Diaphragm or thoracic involvement can refer pain into shoulder and neck via the phrenic nerve, especially when it tracks your cycle.
🧩 Pelvic floor guarding and fascia can pull the whole chain, ribs stiffen, jaw clenches, neck takes the hit.
🩸 Heavy bleeding can mean low ferritin even with “normal” hemoglobin, which can look like fatigue, headaches, shortness of breath, restless legs, muscle aches.
😴 Sleep disruption + mood strain lower pain thresholds, and migraines love that combo.

Clues it’s worth exploring: flares around ovulation or pre-period, painful s*x, bowel or bladder pain, bloating, leg pain, unexplained fatigue, cycle-linked migraines, or shoulder/neck pain with a pattern. A “normal” ultrasound doesn’t automatically rule it out.

Want the full systems map and my sequencing logic?
I just published a long-form Root Cause Breakdown on Substack (Movability Masterclass). Free preview is up. In the members only section, I share the decision tree, sequencing order, a case vignette pivot point, and a bonus endometriosis research update (late 2024 to early 2026).

Save + share, someone’s “random” chronic pain might be undiagnosed endo.
Education only, not medical advice.

Dr. Sina

Link in bio, or search “Movability Masterclass endometriosis beyond period pain” on Substack.

Ever skipped a meal because you knew it would hurt, then got told “everything looks normal”? That disconnect is real. So...
02/25/2026

Ever skipped a meal because you knew it would hurt, then got told “everything looks normal”? That disconnect is real. Sometimes it’s MALS.

MALS (Median Arcuate Ligament Syndrome) is a neurovascular compression. The median arcuate ligament, a band of diaphragm tissue, can press on:
• the celiac artery (blood flow)
• the celiac plexus (a major autonomic nerve hub that drives gut reflexes and carries visceral pain signals)

That nerve piece is why MALS can feel like a full-body problem, not “just GI”.
Common patterns:
• post-meal epigastric pain (The upper center of your abdomen)
• nausea, vomiting, early fullness
• food fear, weight loss
• sometimes exercise-triggered pain
• sometimes lightheadedness or palpitations

Here’s the nuance that gets missed:
• Celiac artery “compression” can show up on imaging in people with zero symptoms. Anatomy alone isn’t a diagnosis.
• Severity on a scan does not always match severity in real life. In a cohort of 96 patients, 84% had decreased post-meal pain after a celiac plexus block, a temporary nerve block, and that relief didn’t neatly track with CT findings.

Complex cases are common. Many patients also have hypermobility spectrum or hEDS, POTS/orthostatic intolerance, MCAS-type reactivity, or other compression syndromes. These can add autonomic vulnerability and small-fiber nerve issues, so two people can share similar anatomy and live completely different realities.

Post-op reality matters too. If symptoms linger, it’s not automatically “anxiety”.
Years of compression can leave behind irritated, remodeled nerves (fibrosis, nerve sprouting). Surgery can remove the squeeze, but nerves may take time to calm down, and some changes can persist. Recovery often needs layers: nutrition support, motility care, dysautonomia management, and targeted pain rehab.

This is the lens I use: anatomy + nerves + physiology, with room for complexity.

Save this. Share it with the person who’s being dismissed.
Educational only, not medical advice.

Want the full systems map and sequencing logic for complex MALS cases? I wrote the deep dive on my Substack, Movability Masterclass. Link in bio.

Dr. Sina

02/22/2026

vitamin d, inflammation, and the gallbladder

If your vitamin D is low and it will not budge, even when you take it perfectly, do not assume you need “more.” In complex cases, a stuck 25(OH)D is usually a bottleneck problem.

Here’s the systems view I use:
1. Vitamin D is fat soluble
Absorption is not just about the dose. It depends on fat digestion.
2. Bile acids are part of the absorption machinery
Bile acids help form micelles, the transport system that allows fats and fat soluble vitamins (A, D, E, K) to be absorbed in the small intestine.
3. The gallbladder is a timing organ
Your liver makes bile continuously. The gallbladder stores and concentrates it, then releases it when you eat, especially with fat. If bile flow is significantly reduced (cholestasis or obstruction), fat absorption can drop, and fat soluble vitamin deficiencies can follow. Even without a full blockage, sluggish emptying can quietly reduce efficiency in the right context.
4. Hormones and metabolism can shift the terrain
Progesterone can reduce gallbladder contractility and slow emptying. Estrogen can change bile composition, and higher estrogen states are linked with higher gallstone risk. In PCOS, impaired gallbladder motility has been reported.
5. Inflammation can flatten the lab
We measure 25 hydroxyvitamin D. Levels often run lower in inflammatory states, and hormone shifts can change vitamin D binding protein, which can shift total lab values. So the number reflects absorption plus distribution plus inflammatory biology.

This is why our holistic care can look like a switch flips. We clear bottlenecks instead of chasing the pill.

Red flags, jaundice, fever, severe right upper belly pain, persistent vomiting.

Want the full systems map and sequencing decision tree? I broke it down in the Root Cause Breakdown in Movability Masterclass on Substack.

Dr. Sina

02/20/2026

If you’ve ever been told “it’s probably nothing,” “your tests are normal,” or “that’s so rare,” keep reading.

I’m tired of hearing “it’s rare” used as an exit sign.

Sometimes it’s not rare. It’s underdiagnosed.

I see the same story on repeat: people sent home with “your labs are normal,” “it’s stress,” “try to relax,” or the classic age-based dismissal, “you’re too young for that,” “you’re too old for this.” Symptoms keep stacking while you’re made to feel like the problem is you.

Here’s what I want you to know, from what I see every day: complex illness rarely shows up as one neat symptom, one neat test, one neat box. The system is built for single-issue problems. A lot of real patients are not single-issue. Pattern recognition is the job, and you can’t do it if you don’t listen.

If you’re in that limbo, hear this clearly: you’re not dramatic, you’re not imagining it, and you deserve to be taken seriously. Your story is not “extra.” It’s data.

A few questions worth bringing to your next visit:
• What are your top possibilities?
• What are we actively ruling out?
• If today’s tests are normal, what is the next step?
• Can you document my symptoms and your plan in my chart?
• Who else should be involved, and why?

What often changes the game is this: stop chasing a label first, start mapping the systems. Nervous system (including autonomic), immune and inflammation, hormones, gut, connective tissue, circulation, energy and metabolism, airway and breathing, and fascia. When you can see which systems are dysregulated, patterns get louder, the differential gets tighter, and the next best tests and referrals actually make sense.

But none of that works without the basics, listening to the patient, taking a thorough history, and doing a real, hands-on physical exam.

This post is a connector. If you feel safe, share your story. What was the “rare” thing that took forever to diagnose, and what finally helped you get answers? Someone scrolling right now might recognize themselves in your comment, ask better questions, and find a path forward, and their people.

Save this. Share it with the person who keeps saying, “I just know something is off.”

Dr. Sina

02/18/2026

We didn’t “randomly” become a society of bloating, reflux, cravings, fatigue, and chaotic appetites. We trained our physiology into it.

Your gut runs on cycles: on-time and off-time.

Every eating episode flips digestion on: stomach mixing, acid and enzymes, bile release, motility shifts, blood sugar rises, insulin rises. Between meals, your system is supposed to finish the job and run housekeeping.

One key housekeeping pattern is the migrating motor complex, a cleanup wave that helps sweep things forward between meals. Constant grazing and sipping calories means stop-start-stop-start, all day.

And “eating episode” is where people miscount. Count everything and you might be at 10 to 12 per day: food, yes, but also the latte with syrup and creamer, the smoothie, the “healthy” bar, the handfuls, the bites while cooking, the sports drink. Your gut doesn’t care that you called it “just something quick.”

Reflux is not always “too much acid.” Often it’s timing plus volume plus pressure plus sensitivity. Keep your stomach busy all day, eat late, brace your abdomen, live stressed, and the valve has more chances to fail.

Add blood sugar volatility and it gets louder. That mid-afternoon “I need something now” can be a glucose drop that feels like urgency or anxiety, not true hunger.

Here’s the uncomfortable part: “on the go” wasn’t a health movement, it was marketing. More eating occasions means more purchases. Portable “functional” snacks keep you coming back.

For the full systems map and clinician-level sequencing, I publish Root Cause Breakdowns on my Substack, Movability Masterclass.

And before you say “I don’t have time,” check your screen time log. Most people can find 10 minutes to sit down and eat one real meal. You have more time than you think you do.

Try this for 7 days: 2 to 3 real meals, reduce caloric drinks between meals, leave a few hours between eating, slow down, chew, stop sipping your calories all day. Then watch what your gut does.

If this connected a few dots, share it with someone living on “quick bites.”

Dr. Sina

02/15/2026

Hiatal hernia content online is full of “push it down” hacks.

Here’s the issue: a sliding hiatal hernia lives inside the rib cage. You can’t safely grab it from the surface. What you can train is the system around it: diaphragm mechanics, rib cage position, and pressure management.

At the esophagogastric junction there isn’t one valve. It’s a stacked barrier:

the LES (smooth muscle)
the crural diaphragm (skeletal muscle)
connective tissue that helps keep the junction aligned
When posture collapses and pressure spikes, that barrier gets less efficient. That’s why the same person can feel fine walking around, then flare when slumped, braced, breath-holding, bending, or lifting.

The drill in this video is my “Hiatus Anchor” sequence: towel feedback around the lower ribs, long “ssss” exhale to drop the ribs and shut off bracing, tiny pause, then a small nasal inhale expanding 360° into the towel. Four cycles.
The goal isn’t to “pull the stomach down.” The goal is to restore clean diaphragm motion and reduce the upward pressure patterns that keep irritating the area.

Quick self-check: if the only way you can get a satisfying inhale is by shrugging your shoulders, that’s a clue you may be relying on accessory muscles because your lower ribs/diaphragm aren’t moving well. Not a diagnosis, but it often travels with the same mechanics that worsen reflux patterns.

Do this 2–3 hours after meals. Stop if it provokes pain, dizziness, or a clear reflux flare. If you have trouble swallowing, vomiting blood, black stools, unexplained weight loss, or chest pain that feels cardiac, get medical care.

Save this. Try it daily for a week, then track symptoms and breathing quality.

If this helped you connect a few dots, feel free to share it with someone stuck in this pattern.

For the deeper anatomy + progressions, Movability Masterclass on Substack. Link at the top of my page.

Big thanks to for the demonstration.

Dr. Sina

02/13/2026

The detergent aisle is a diagnostic clue.

If a “fresh linen” cloud gives someone migraine, flushing, itching, hives, tachycardia, GI cramping, brain fog, and a full-body crash, that is not fragility. It’s a neuroimmune pattern.

Why odors and VOCs can do this:
1. Trigeminal alarm
Many VOCs activate irritant receptors (TRPA1/TRPV1) on trigeminal nerve endings in the nose. That alarm is wired into migraine circuitry.
2. Migraine cascade
In susceptible people, trigeminal activation can trigger CGRP and other neuropeptides, sensitizing tissues around the brain (neurogenic inflammation), and flipping on an attack.
3. Mast cell amplification
Mast cells live in skin, gut, airway, blood vessels, and even the brain. When dysregulated they can release histamine, prostaglandins, leukotrienes, and cytokines, producing multi-system symptoms that mimic “allergies,” “IBS,” “anxiety,” POTS, or unexplained sensitivities.

Clinical clues that should raise suspicion:
• symptoms across 2+ organ systems
• reactions to fragrances, temperature shifts, alcohol, or medications
• flares with hormonal changes
• negative allergy tests
• tryptase that is “normal” (this does not rule MCAS out)

At Movability, we approach this from the inside out.
We start with a deep case review to map triggers and timelines. Our naturopath can order specialty labs, including mast cell mediators that are ideally captured during or soon after a flare. If MCAS is plausible, we collaborate with your physician because meds like H1/H2 blockers, cromolyn, ketotifen, leukotriene inhibitors, or low-dose naltrexone require a prescription. Then we layer mast cell stabilization, gut work, nervous system support, hands-on therapy, and graded movement to raise threshold and rebuild tolerance.

Full Root Cause Breakdown (systems map + decision tree + case review) is up now in Movability Masterclass on Substack.

If this helped connect dots, share it.

Dr. Sina

That thumb bend is not a party trick. It is connective tissue doing what it was built to do, and in hEDS it can mean eve...
02/11/2026

That thumb bend is not a party trick. It is connective tissue doing what it was built to do, and in hEDS it can mean every small task costs extra stability. Hypermobility is not just “loose joints.” Joints move differently, and the missing piece is often fascia.

Fascia is the body-wide collagen and hyaluronan matrix that wraps, connects, and communicates. It is loaded with sensory nerves, autonomic fibers, immune cells, and contractile myofibroblasts. It transmits force between regions, forms the “guy wires” around joints, organizes muscle insertions, and creates corridors for nerves, vessels, and lymph.

For years it was treated like packing material, something to release and forget. Emerging research says otherwise. Ultrasound and elastography are showing measurable differences in fascial thickness, stiffness, and glide in hypermobility. Mechanobiology shows fibroblasts respond to specific loading by remodeling the extracellular matrix. Translation: fascia can be conditioned, not only loosened.

Movement keeps fascia slick, loading aligns collagen. Done properly, fascia training can improve containment around unstable joints, including the cervical spine, without end-range chasing. That is why neck instability, “TOS-like” symptoms, and POTS-type flares often live in the same neighborhood.

This is where hEDS care is headed: smarter connective tissue training that restores glide where densified, builds supportive tension where lax, and improves the sensory signal so the nervous system stops screaming. When fascia and its nerve supply normalize, the ripple effects can show up as better proprioception, less pain amplification, improved joint control, and shifts in symptoms that look neurologic, vascular, and lymphatic.

I have been applying fascia-first strategies in complex hypermobility cases for over a decade, before it became popular. Mark my words, fascia will be treated as the overlooked organ system it is, and the next wave of advancements will help far more than joints alone.

Full Root Cause Breakdown: “hEDS and Fascia” is now on Movability Masterclass (link in bio).

Dr. Sina Yeganeh, chiropractor and global complex case consultant

02/08/2026

Stop blaming arthritis because someone said your X-ray is “fine.” A joint can be breaking down while the X-ray still looks normal. This is one way joints fail early.

AVN (avascular necrosis) means part of a bone is dying because its blood supply is reduced. Bone is living tissue. When blood flow drops, the weak spot is often the subchondral bone (the bone right under the cartilage). Think of cartilage like flooring. If the subfloor cracks, the floor fails next.

The root cause is usually a combo of 3 things:
1. Blockage: tiny vessels get clogged (fat droplets, microclots, sickled cells).
2. Pressure: the marrow space swells or fills with fat, pressure rises, and it squeezes those tiny vessels shut (like stepping on a sponge).
3. Repair mismatch: dead bone can’t remodel fast enough to handle load.

Result: micro cracks, then a bigger crack under the surface (the “crescent sign”), then collapse, then fast arthritis.

Where it hides: hip is #1 (femoral head), then shoulder, knee, ankle. Non-traumatic AVN is often bilateral, so one painful hip can mean a quieter lesion on the other side.

Rare but high-yield: the scaphoid in the wrist. Most of its blood enters from the far end, so the proximal pole is last in line. A fracture can isolate that piece and AVN can follow.

Who’s at risk: trauma (fracture/dislocation), long-term or high-dose steroids (prednisone), heavy alcohol use (it can increase marrow fat, raise pressure, and weaken bone repair), sickle cell disease, lupus, clotting disorders, smoking/high lipids, decompression sickness, some cancer treatments.

Symptoms that fool people: early can be silent. When it speaks, it’s deep joint pain with load (stairs, standing, pivoting). Hip AVN is often groin pain and can refer to the thigh or even feel like knee pain. Later: night pain, stiffness, limp.

In complex cases, I don’t let a normal X-ray end the conversation.

Key truth: early X-rays can be normal. MRI is the stage-defining test. Rule: stage first, then load.

Want the full systems map, staging decision tree, and the body-wide AVN map? I broke it down in Movability Masterclass on Substack (link in bio).

Dr. Sina

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What Makes Us Unique

Movability was created to fill a void in the healthcare world. We noticed a lack of truly custom and comprehensive care, we knew we had to be different to provide the best possible treatment. We set out to create a unique therapeutic experience built on empathy, trust, and unparalleled attention to detail. We spend the time to get to know the real you, your goals, dreams and expectations because you are much more than just a diagnosis. We work with you to create custom treatments that meet those expectations. At Movability there’s no such thing as one size fits all. Experience the difference for yourself.