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Movability - Wellness & Sport Sciences

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Richmond Hill, ON
Movability - Wellness & Sport Sciences

Chiropractic, pelvic floor physiotherapy, acupuncture, massage, orthotics, naturopathic care, and rehab all under one roof.

We treat complex conditions through full-body assessment, root-cause care, and a collaborative, patient-first approach.

11/26/2025

IT WASN’T HER ANXIETY.

She came in in her mid 40s with a body that felt out of control. Heart racing out of nowhere. Sudden waves of dread. Waking at 3 a.m. wired and sweating.

Her doctor kept calling it “anxiety.” First an SSRI. Then another. Then an as needed pill for panic.

Nothing touched it. She kept saying, “It feels like my body is doing this to me.”

When we ran labs, the pattern was clear. Cycles getting irregular. Estradiol swinging low. Progesterone tanked. FSH in a perimenopausal range. Thyroid and basics looked ok.

Her brain was reacting to a hormone storm.

Estrogen helps regulate serotonin and norepinephrine, the chemicals that keep mood steady and threat signals in check. Progesterone turns into a calming neurosteroid that boosts GABA, the brain’s brake pedal. In perimenopause those hormones spike and crash. The nervous system feels that as surges of fear, racing thoughts, and a body that cannot settle.

We built a plan with her medical team. Her physician started appropriate hormone replacement therapy. We rebuilt sleep, blood sugar stability, and gentle movement.

Within weeks her night sweats eased, sleep returned, and that “out of nowhere” panic quieted.

If you are in your 40s, your periods have changed, and anxiety suddenly showed up like a stranger, it might not be “just anxiety.” It might be perimenopause.

If you want deeper breakdowns on cases like this and more, I teach them inside Movability Masterclass ✅ Link in bio

Dr. Sina

11/24/2025

Do you ever stand up from your desk and your spine feels like one solid rod. Glued together from your low back into your neck.

I see this often in people with flexion heavy jobs. Most are told it is “just posture” or “tight muscles.” Almost no one hears about a tiny recurrent nerve at each spinal level called the sinuvertebral nerve.

This nerve listens to your discs, the ligament on the back of your vertebrae, the front of your dura, and the endplates. When you live in flexion, you keep loading the exact tissue it is wired to protect.

Hours of sitting, laptop hunching, bent over tools or patients, lifting with a rounded back, all load the back edge of your discs. Over time those tissues get irritated and this nerve sensitizes. It stops sending quiet status updates and starts sending threat signals. Your brain does not hear “one sore spot.” It hears “protect everything” and your whole spine turns into armor.

You cannot foam roll this quiet, but you can give it different input.

Two resets you can use daily or as breaks:
1. Wall decompression breath
Back to a wall, feet about a foot forward. Soften your knees. Gently tuck your tail so your low back just touches. Lightly tuck your chin so the back of your head touches. Take 6 slow breaths. Inhale through your nose into the lower ribs, exhale through your mouth and imagine your spine lengthening away from your pelvis. This lets the posterior disc and dura feel long instead of constantly crumpled.
2. Prone spine glide
Lie face down with a folded towel under your chest. Forearms on the floor like a gentle sphinx. Think of your breastbone gliding forward as you lengthen the top of your head away. Slight lift of the upper chest, gaze down. Hold 5 slow breaths, lower, repeat a few times. You are feeding your spine an opposite to all day flexion without jamming into end range.

Use these once or twice a day, especially after long flexion blocks.

When I release the stiff structures that keep tugging on these nerves, people often stand up and say it feels like their spine is brand new. They are always a bit surprised by how light and flexible they feel once that protective rod finally lets go.

— Dr. Sina

11/23/2025

If your body feels like a roller coaster from hour to hour, this is for you.

To everyone living with hEDS, POTS, MCAS, endo, adenomyosis, PCOS, ME or CFS, migraine, IC, pelvic pain, scoliosis, PCS, pudendal neuralgia, autoimmune disease, complex hormone issues, mold illness, Lyme, long Covid, EBV, or a cluster of “mystery symptoms” that still does not have a clear name, I see you. Even if nobody has given you a diagnosis yet, your experience is real.

You look “fine” on the outside, yet you are carefully rationing every bit of energy just to show up. You are tracking symptoms, bathroom access, safe foods and crash points in your head while the world assumes you are healthy.

You have been told “your tests are normal”, “it is all in your head”, “it is probably anxiety”, “let us wait and see”. You have been made to feel dramatic, difficult or broken when in reality your body is fighting incredibly hard every single day.

Your pain is real. Your fatigue is real. Your brain fog, dizziness, heart racing, pelvic pain, bladder urgency, gut flares and hormonal storms are real. You are not making it up. You are not lazy. You are not failing at self care.

If getting dressed, working a few hours, attending a class, doing school drop off or answering messages is all you managed today, that is not “nothing”. That is real work in a body that does not play by the rules. If all you could do was stay in bed and breathe through another flare, that still counts.

I believe you. I trust your experience more than any normal lab result. I sit with patients every day whose lives have been derailed by these invisible conditions. With me, you are not alone, you are not imagining it, and you deserve care that actually takes you seriously.

— Dr. Sina

11/21/2025

If everything gets worse at night, this is going to blow your mind.

All day your cold, your joints, your back feel annoying but manageable. Then it hits 10 p.m. and suddenly you are congested, burning, aching, lying there thinking, “What is wrong with my body?”

Here is the wild part. No one ever explains this part. Nothing is random. Inflammation runs on a clock.

During the day you have more cortisol in your system. That is one of your built in anti inflammatory drugs. It keeps a lid on swelling, fever, and pain signals so you can function.

At night cortisol drops. Melatonin rises. That combo tells your immune system, “Night shift, go.” White blood cells stick to vessel walls more easily and move into your tissues. Great for killing germs and cleaning up damage, but it also makes joints puffy, nerves extra sensitive, and temperature creep up.

Then posture piles on. You lie flat, fluid shifts, discs and joints take on water, anything irritated feels tighter. Blood pools in your nose so congestion and sinus pressure spike. Acid hangs out in your esophagus so reflux and cough wake up. Asthma often tightens at night too.

It is the same with chronic stuff. Rheumatoid arthritis often has brutal morning stiffness because inflammation has been building all night. Gout flares love the late night hours. Old injuries, nerve pain, low back pain, they all ride the same biology.

So if evenings are always the hardest for you, you are not weak, dramatic, or “too sensitive.” Your body is literally built to turn the volume on inflammation up after dark. Once you understand that, you can start timing your food, meds, and sleep around the way your system actually works.

— Dr. Sina

11/19/2025

Your migraines are not random. They are threshold. When load crosses your line, the attack fires. When the line sits higher, the same day does not take you out.

If light and sound feel hostile, if nausea, brain fog, and canceled plans steal days, you are not imagining it.

Migraine is a clinical label, not a single cause. It describes a brain state, not a culprit. For many, roots cluster across systems, which is why one food swap or one pill rarely changes the story.

Threshold moves with sleep and circadian rhythm, stress chemistry, hormone shifts, hydration and glucose, gut immune signals, sensory load from light, weather, medication patterns. And the part most people miss, mechanics: upper neck and jaw, tongue and diaphragm, eyes and vestibular system. If grocery aisles, scrolling, or car rides set you off, that pathway is involved.

Science you can feel. Migraine brains are more excitable and can show reduced habituation to repeated input between attacks. CGRP is a key messenger in the trigeminovascular network. Tiny nitric oxide challenges in controlled studies can provoke the premonitory state before pain. The cortical wave tied to aura can also switch on protective programs, a proposed safety net. Wearables flag pre attack shifts in heart rhythm, skin conductance, temperature, movement, sleep.

This is not about one diet or one drug. Patterns higher in omega-3 and lower in excess omega-6 helped in trials for some, not all. CGRP targeted preventives reduced monthly migraine days for many, not everyone. Neither is a one size answer. Both are examples of raising tolerance while we hunt true drivers. Frequent quick relief can quietly keep the line low. Not your fault; the biology is tricky.

My lane is threshold centered, systems first. I read and sequence change: rhythm and regulation, fuel and inflammation, mechanics and environment, hormone rhythm across the lifespan, medication ecology. I have applied this map in thousands of encounters over more than a decade, across continents.

The shift is simple to say and powerful to live. Move from trigger chasing to threshold rising while we map and treat true drivers.

— Dr. Sina

11/17/2025

Ankylosing spondylitis, simplified and research backed. This is more than pain and stiffness.

We reviewed the best evidence across genetics, microbiome and virome, mechanics and imaging, to explain why every system matters.

Here is what a flare does under the microscope. At the enthesis, the tiny anchor where ligament meets bone, the immune system fires an alarm. TNF, IL 17, and IL 23 rise. White cells flood the area. Osteoclasts dissolve the bone edge and leave pits. Then repair overshoots. A soft scaffold is laid down, mineralizes, and becomes thin vertical ridges called syndesmophytes. Layer by layer they can bridge one vertebra to the next. That is how flexible joints become a bamboo spine.

Why it happens, according to the best evidence. Genes matter, especially HLA B27, ERAP1, and IL23R. The gut microbiome and virome are often altered. Viral hits and other infections can tip the system on. Repeated micro stress at the anchors adds fuel. Smoking accelerates damage. Men tend to form bridges faster. Women often present differently and are diagnosed later.

How it gets missed. Onset is usually before 45. Early X rays can be normal. HLA B27 can be negative. MRI of the sacroiliac joints often shows inflammation first. Look for extra clues like uveitis, psoriasis, or bowel inflammation.

Treatment and timing. Modern medicine can drive inflammation toward remission. TNF and IL 17 blockers, and sometimes JAK inhibitors, reduce inflammatory signals. Starting earlier is linked with less structural progression.

Our approach. We work with your doctor, and with our naturopathic doctor, to use the remission window well. While medicine cools the fire, we help regulate the systems that influence it, gut, immune, hormones, bone, connective tissue, lungs and chest wall, heart and vessels, nervous system, sleep and stress, nutrition and metabolism, movement and load. The goal is simple, raise the odds of staying in remission, reduce flare risk, and protect mobility over time.

— Dr. Sina

11/16/2025

🪝 When patients ask me what’s causing their POTS, this is my quick checklist:

• Idiopathic when no clear driver is found.
• Autoimmune: systemic autoimmune disease, autonomic receptor autoantibodies, small fiber autonomic neuropathy. Mast cell activation overlap in some.
• Post infectious: EBV mono, influenza, GI viruses, COVID-19. Lyme in some. Rare post vaccine onset without proven causality.
• Genetic: familial clustering. Rare NET (SLC6A2) mutation. Possible sodium channel variants.
• Connective tissue: hypermobile Ehlers-Danlos or joint hypermobility causing venous pooling.
• Neuropathic secondary: diabetes, amyloidosis, sarcoidosis, chemo-related, Sjögren small fiber neuropathy.
• Hyperadrenergic: high standing norepinephrine, NET dysfunction or drug induced NET inhibition (atomoxetine, reboxetine, some SNRIs or TCAs).
• Hypovolemia with impaired renin-aldosterone responses.
• Deconditioning: prolonged bed rest or illness, microgravity analogs.
• Hormonal and life stage: puberty or growth spurts, menstrual shifts, pregnancy and postpartum.
• Physical stressors: concussion or head or neck trauma, surgery, major illness.
• Structural contributors in select cases: Chiari I or craniocervical instability.
• Medications and other triggers: diuretics, potent vasodilators, anticholinergics, some antipsychotics, drospirenone OCPs.
• Endocrine mimics to rule out: hyperthyroidism, adrenal disorders.

Save to discuss with your clinician, then personalize testing and treatment.

Why this matters: POTS is heterogeneous. Many people have several drivers at once, for example small fiber neuropathy plus venous pooling plus hyperadrenergic physiology. When I meet someone with POTS I look at the whole picture, not one label: onset trigger, comorbidities, hypermobility signs, immune and infectious clues, hormones, sleep and nutrition, medications, volume status, conditioning, and goals. Then I tailor testing and treatment to the dominant pathways. I’ve talk about POTS a lot on my page and will continue to do so. Hope this helps!

— Dr. Sina

11/15/2025

🧠 A common virus may be fanning the flames of autoimmune disease. Epstein Barr virus infects most of us by adulthood, which means a positive EBV panel is expected, not destiny.

What matters is behavior. A new study mapped what EBV is doing inside lupus. The virus hides in memory B cells and in lupus it appears far more often, about 1 in 400 cells compared to fewer than 1 in 10,000 in healthy people. EBV flips genetic switches with a protein called EBNA2. Those infected B cells start acting like turbo antigen presenting cells, showing pieces of your own tissue to T cells. T cells push more B cells to make autoantibodies. The fire spreads through the body. That is fatigue, brain fog, rashes, joint pain, organ flares.

EBV is a s**t disturber. Not because everyone with it gets sick, but because in vulnerable people it tilts the whole immune orchestra off key. EBV biology has also been linked to multiple sclerosis, rheumatoid arthritis, Sjögren’s, autoimmune thyroid disease, systemic sclerosis, and some inflammatory myopathies. Different organs, different symptoms, but a similar spark. Molecular mimicry, confused immune brakes, and tired regulatory cells that cannot calm the response.

Why some get hit harder: genes that amplify inflammation, female hormonal shifts, chronic stress, smoking, low vitamin D, co infections, and the unlucky spot where the virus settles.

What this means for real people:
• Stop burning money on repeated EBV tests. A positive result confirms exposure only.
• Ask better questions. Are B cells overactive. Are T cells being overhelpful. What is driving flares today.
• Use real levers. Improve sleep, support hormones and metabolic health, manage stress, treat true infections, nourish your body, and use evidence based therapies.
• Watch the horizon. EBV vaccines, precision antivirals, and smarter B cell strategies are being built. Not cures yet, but real targets finally in sight.

You are not broken. Your immune system is responding to signals. Science is getting closer to understanding those signals and how to quiet them before they burn the house down.

— Dr. Sina

11/14/2025

If your spine MRI says “disc osteophyte complex,” do not chalk it up to “just aging.” This is a targeted repair response to microinstability. As discs lose proteoglycans they dehydrate, lose height, and develop annular fissures; load shifts to the vertebral rim, endplates, uncovertebral joints, and facets. The spine tries to stabilize by laying down fibrocartilage that ossifies into marginal bone spurs (endochondral ossification). Paired with a bulging or herniated disc, those osteophytes can narrow the central canal or the neural foramina, producing spinal stenosis or foraminal stenosis and nerve root compression. This pattern is typical in cervical spondylosis and degenerative disc disease.

Mechanisms to know: reduced disc hydration and height; annular tears; endplate microfracture and sclerosis; facet and uncovertebral hypertrophy; ligamentum flavum infolding; osteophyte growth along Sharpey fiber attachments. At the molecular level, loss of proteoglycans, a shift toward type I collagen, upregulated MMPs, and inflammatory cytokines like IL‑1 and TNF sustain the cascade. Adaptation comes first; crowding and symptoms come later.

Who is more prone: people with hypermobile Ehlers‑Danlos syndrome or generalized joint hypermobility (segmental laxity increases shear); prior whiplash or disc injury; heavy repetitive loading at work or in sport; smoking; metabolic and hormonal factors that impair disc nutrition and repair; and conditions like DISH that create large anterior osteophytes.

Complications to watch: radiculopathy with dermatomal pain, numbness, or weakness; degenerative cervical myelopathy, also called cervical spondylotic myelopathy, with hand clumsiness, imbalance, and hyperreflexia; dysphagia from large anterior cervical spurs; rare cerebrospinal fluid leaks from ventral dural tears caused by calcified disc or osteophyte, often presenting with orthostatic headache and MRI signs of intracranial hypotension.

My approach is root cause. When I see this on imaging, I always ask why. I review the entire history and the whole body, inside and out, plus the environment you live and train in, to find what drove it so we can stop it from progressing.
—Dr. Sina

11/12/2025

Deep sleep in a glass. This cold and sparkly bedtime drink uses tart cherry, chamomile, lavender, a teaspoon of raw honey, and a pinch of cinnamon to help you wind down and support deep, restful sleep.

I used what I had at home. Ideally, steep high quality organic tea (I used tea bags).

Why it works
• Tart cherry provides natural melatonin and sleep supporting anthocyanins
• Chamomile contains apigenin that interacts with GABA receptors to promote calm
• Lavender aroma is associated with improved sleep quality
• Raw honey gives a gentle glucose nudge that may aid tryptophan availability and soothes the throat
• Cinnamon provides polyphenols that support a steady glucose response and a cozy flavor

Ingredients
• 1 chamomile tea bag
• 1 lavender tea bag
• 2 to 3 oz organic tart cherry juice
• 4 to 6 oz sparkling mineral water
• 1 tsp raw honey
• A light pinch of cinnamon
• Ice optional

Instructions
1. Steep both tea bags in 1/2 cup hot water for 5 to 7 minutes. Remove the bags.
2. Let the tea cool completely. Refrigerate to speed it up.
3. Stir in the honey, then a light pinch of cinnamon. If the tea is slightly warm it will dissolve honey faster. If fully cool, just stir longer.
4. In a clean glass add the tart cherry juice.
5. Pour the cooled tea over the cherry juice.
6. Top with sparkling mineral water and give a gentle stir so you keep the bubbles.
7. Sip and enjoy.

Care so you do not ruin the good stuff
• Do not make this piping hot. Higher heat can dull chamomile and lavender aroma, and it can degrade cherry anthocyanins and the delicate enzymes in raw honey.
• Keep it cold and sparkly. Cool the tea first and add sparkling water last to preserve fizz.
• Sweetness is flexible. Start with one teaspoon of honey and adjust.

Sleep well after you enjoy the Dr. Sina Sleep Tonic.

— Dr. Sina

11/11/2025

Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) research update and path forward, 2025

You are not invisible. Your symptoms are real. Many were told to push through. That was harmful. Modern guidance centers post exertional malaise, respects energy limits, and treats this as a biomedical condition.

What the best evidence says now (
• ME/CFS is a neuroimmune and energy disorder.
• Post exertional malaise is core. There is no single lab test. Diagnosis is clinical and person centered.
• Autonomic dysfunction is common. Orthostatic intolerance and POTS often respond to fluids, salt, compression, and selected medications.
• The crash after exertion can be measured. On a two day CPET many show a reproducible day two drop that is not seen in simple deconditioning.
• Brain studies at ultra high field show elevated lactate in some regions, consistent with energetic stress.
• Immune profiling shows dysregulation and features of immune exhaustion, with overlap seen in long COVID. A subset has autoantibodies to β2 adrenergic or muscarinic receptors. Early studies in carefully selected post COVID cases suggest antibody removal may help, this remains investigational.
• A large randomized trial found that coenzyme Q10 plus NADH improved fatigue and some sleep measures. Not a cure, a useful add on for some.

Care today
Pacing first. Use heart rate informed activity to prevent crashes. Treat orthostatic intolerance. Optimize sleep and pain. Consider low risk adjuncts with evidence. Build a plan that protects limited energy and supports daily function.

Guidance, up to date
NICE NG206 (UK) remains current as of January 2025 and advises against fixed graded exercise. CBT can support coping, it is not curative. A 2024 D A CH consensus (Germany, Austria, Switzerland) reached similar conclusions.

Hope
Research is accelerating across the UK, Europe, Australia, and worldwide. We are mapping subtypes, testing targeted therapies, and building better biomarkers. You deserve validation, careful management, and partnership. You are not alone.

Educational content only. Talk with a clinician experienced in ME/CFS for personal care.

— Dr. Sina

11/10/2025

Your body is a planet seeking balance. Homeostasis is its climate control, the quiet work of keeping temperature, blood sugar, hormones, and the nervous system steady. When you stack detox plans, IVs, new supplements, an elimination diet, and a fresh workout all in the same week, you create weather, not healing. Too many variables become storms. Signals blur. Progress stalls.

Start with stability. Reduce variables. Pick one lever. Define intent. Set a metric and a timeline. Examples that move the needle without chaos: protect 7 to 9 hours of sleep, front load protein and fiber, morning light for circadian rhythm, ten minute walks after meals, hydrate with minerals, one consistent bedtime, five minutes of breath work. Track simple markers, energy, mood, digestion, skin, cycle, resting pulse, cravings, performance.

Honor sensitive seasons when homeostasis is easier to disturb. Puberty. College or first job stress. Pregnancy. Postpartum and breastfeeding. Perimenopause. Menopause. Andropause. Starting or stopping birth control. Fertility treatments. Thyroid shifts. Coming off stimulants or SSRIs. Post viral recovery. After concussion or surgery. Big travel and jet lag. Shift work. High volume training or weight cuts. Grief and major life transitions.

Before any protocol, ask three questions. What is my physiology telling me. What is the single target. How will I measure change over two to four weeks. If you cannot answer, you are not ready to add more. Pull back to foundations until the signal is clear. Your body is the master. You are the steward.

This is the work I do with patients, we reduce variables and trickle in targeted strategies so the system can regulate and repair. If you have been trying ten things at once, take a reset week, stabilize the routine, then introduce one precise intervention. Comment with the one lever you will test next, save this for the next sensitive season, and share with a friend who needs fewer storms. Slow is smooth. Smooth is fast. Strong foundations steady insulin, calm inflammation, support the microbiome, and build metabolic health. When you change less, you learn more, and what you keep actually works.

— Dr. Sina

Address

2 Hunters Point Drive
Richmond Hill, ON
L4C9Y4

Opening Hours

Monday	10am - 7pm
Tuesday	10am - 7pm
Wednesday	10am - 7pm
Thursday	10am - 7pm
Friday	10am - 7pm
Saturday	10am - 4pm
Sunday	10am - 4pm

Telephone

+19057634000

Website

http://www.movability.ca/

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What Makes Us Unique

Movability was created to fill a void in the healthcare world. We noticed a lack of truly custom and comprehensive care, we knew we had to be different to provide the best possible treatment. We set out to create a unique therapeutic experience built on empathy, trust, and unparalleled attention to detail. We spend the time to get to know the real you, your goals, dreams and expectations because you are much more than just a diagnosis. We work with you to create custom treatments that meet those expectations. At Movability there’s no such thing as one size fits all. Experience the difference for yourself.