07/05/2026
This week, I want to talk about another pathology that is perhaps sometimes overlooked. This one is a little more hock-focused.
We hear a lot about hock pathologies — arthritis being one of the most common, bone spavin, bog spavin, thoroughpins, capped hock… all very real, very common.
But there are other structures in and around the hock that don’t get talked about nearly as much, and yet can have a really significant impact on how a horse moves and feels.
One of those is the retinaculum.
I’ve also put together a short video with some images and clips showing retinaculum-related pathology and what this can sometimes look like in practice… you’ll find it in the comments!
The retinaculum is a band of dense fibrous connective tissue. Its role is to stabilise and guide tendinous structures as they pass around a joint, helping them track correctly and move efficiently during locomotion. Around the hock, these supporting structures play an important role in stabilising the tendinous structures around the calcaneal region, particularly the superficial digital flexor tendon (SDFT), as it passes over the point of hock (tuber calcanei).
When everything is functioning as it should, you don’t notice it. The tendons track smoothly, movement is efficient, and the hock appears stable.
But when that system starts to break down, things can change quite quickly.
I’ve now seen a couple of cases over the past year where disruption to these structures has been associated with hock pain, swelling, and varying degrees of lameness — sometimes quite obvious, sometimes more subtle.
There are a few different ways this can happen.
In some cases, the retinaculum can become lax. That might be influenced by conformation, repetitive strain over time, or changes in how the horse is loading the limb — often as a result of compensation elsewhere. Inflammatory processes within the joint, such as early osteoarthritic change, can also influence the behaviour and integrity of the surrounding soft tissues, including the retinacular structures.
At the more severe end of the spectrum, there can be traumatic damage or rupture, meaning the structure is no longer able to do its job effectively.
When that stabilising support is compromised, one of the key things we can see is the SDFT starting to slip out of position as the limb takes weight. Most commonly, this is to the lateral side of the point of hock, although I have also seen medial displacement occur.
This can be quite striking visually.
The hock may appear enlarged when the limb is weight-bearing, and then look more normal when unloaded. In some cases, there is genuine swelling alongside this, but in others what you’re actually seeing is the tendon sitting where it shouldn’t.
I’ve seen this present intermittently and quite subtly, and I’ve also seen it occur consistently with every step — particularly where there has been more significant disruption to the retinaculum.
From a clinical perspective, this isn’t just a localised “hock issue.”
If the tendon is repeatedly slipping out of position, it’s being exposed to abnormal mechanical stress. Over time, that can lead to irritation, wear and tear, and degenerative change within the SDFT itself.
And that’s where things can start to have a wider impact.
The SDFT plays a really important role in supporting the limb during weight-bearing and locomotion. It helps control extension of the distal limb and contributes to energy storage and release during movement. If it becomes compromised, you don’t just lose efficiency — you start to alter how the entire limb loads.
The gastrocnemius tendon can also become involved. Although it is a separate structure, it is very closely associated with the SDFT around the point of hock, and both contribute to the function of the reciprocal apparatus of the hind limb. If there is instability, inflammation, or abnormal tendon tracking in this region, it can create a surprisingly chaotic mechanical environment around the calcaneus, potentially placing strain on multiple surrounding structures rather than just one isolated tendon.
That can then place additional strain on other structures, change movement patterns, and in some cases contribute to further compensatory issues higher up the limb or elsewhere in the body.
So whilst the primary issue may sit with the retinaculum, the knock-on effects can extend further if these associated structures aren’t considered together.
In terms of management, veterinary involvement is essential, particularly where there is lameness. Anti-inflammatories, including corticosteroids, may be used to reduce inflammation and discomfort, and in some cases this can improve stability if inflammation is a contributing factor. In more severe cases, surgical stabilisation may be considered.
From a physiotherapy perspective, we’re looking at the bigger picture.
That means supporting not just the hock, but also the tendons and surrounding soft tissues being affected, whilst also addressing how the horse is loading and moving as a whole.
Modalities can play a really useful role here alongside a structured rehabilitation plan.
Radiofrequency (Indiba) works at a cellular level to promote tissue healing. It helps improve circulation, supports metabolic activity within the tissues, and can encourage processes involved in repair — including collagen synthesis and cellular activity. In structures like the retinaculum and the SDFT, that can be really valuable in improving tissue quality and supporting healing where there has been strain, inflammation, or degeneration.
Class IV laser also supports healing, particularly through its effects on inflammation, pain modulation, and cellular activity. It can help create a more favourable environment for tissue repair and is a really useful tool when managing both acute and more chronic presentations.
Therapeutic ultrasound works slightly differently, using mechanical energy (sound waves) to influence the tissues. It can be particularly helpful in the later stages of healing, where we’re aiming to improve fibre alignment and tissue organisation — something that is especially relevant when we’re thinking about tendon health, such as the SDFT.
In practice, I will often choose modalities based on the individual case — what stage of healing we’re in, which structures are most involved, and what the overall goal is. It’s rarely a one-size-fits-all approach.
In more severe cases, particularly where there has been a complete rupture of the retinaculum and surgical intervention isn’t pursued, we do have to think carefully about what is fair and appropriate for the horse long term. Without that stabilising structure, the mechanics of the limb are significantly altered, and asking that horse to continue in work may not be in their best interests.
In my opinion, in those situations, retirement is often the kindest option.
That doesn’t mean we stop supporting them altogether. Maintaining comfort and managing secondary strain — particularly within the SDFT — is still really important, and modalities such as therapeutic ultrasound can be a useful and more accessible way of doing that in the long term.
As always, it’s about looking at the whole horse and making decisions that prioritise both function and welfare.