Samvedna homoeo hall satna

15/11/2024

https://www.facebook.com/share/Y12TVwZjtERdjHp8/

21/02/2019

Opening of new branch of my clinic at nai basti jail road beside sbi kiosk satna m.p

14/02/2019

You are cordially invited to Inauguration of my another clinic On dated 17th Feb 2019 & We feel honoured to solicit your presence at the Inauguration .💐💐

10/09/2018

04/08/2018

The ALOPECIA AREATA


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Description

Introduction
Alopecia Areata is an autoimmune disease that causes hair to fall out in small, random patches. The hair loss usually affects the scalp, but it can also occur in other areas of the body.Alopecia areata is a disease that causes hair to fall out in small patches. It develops when the immune system attacks the hair follicles, resulting in hair loss. Sudden hair loss may occur on the scalp and other parts of the body. The condition rarely results in total hair loss, or alopecia universalis, but it can prevent hair from growing back. When hair does grow back, it’s possible for the hair to fall out again. The extent of hair loss and regrowth varies from person-to-person.

Alopecia Areata is "a common condition of undetermined etiology characterized by circumscribed, no scarring, usually asymmetric areas of baldness on the scalp, eyebrows, and bearded portion of the face."

Causes
The condition occurs when white blood cells attack the cells in hair follicles, causing them to shrink and dramatically slow down hair production. It is unknown precisely what causes the body's immune system to target hair follicles in this way.

Alopecia Areata is an autoimmune disease. An autoimmune disease develops when the immune system mistakes healthy cells for foreign substances. Normally, the immune system defends your body against foreign invaders, such as viruses and bacteria. If you have alopecia Areata, however, your immune system mistakenly attacks your hair follicles. Hair follicles are the structures from which hairs grow. The follicles become smaller and stop producing hair, leading to hair loss.

While scientists are unsure why these changes occur, it seems that genetics are involved as Alopecia Areata is more likely to occur in a person who has a close family member with the disease. One in five people with the disease has a family member who has also developed Alopecia Areata.

Symptoms:
The main symptom of Alopecia Areata is hair loss. Hair usually falls out in small round patches on the scalp. These patches are usually several centimetres or less. Hair loss might also occur on other parts of the body. You may first notice clumps of hair on your pillow or in the shower. However, other types of diseases can also cause hair to fall out in a similar pattern. Hair loss alone shouldn’t be used to diagnose Alopecia Areata.

In rare cases, some people may experience more extensive hair loss. This is usually an indication of another type of alopecia, such as:

Alopecia totalis, which is the loss of all hair on the scalp

alopecia universalis, which is the loss of all hair on the entire body

hair loss associated with Alopecia Areata is unpredictable and random. The hair may grow back at any time and then may fall out again. The extent of hair loss and regrowth varies greatly from person-to-person.

26/07/2018

ALLERGIC RHINITIS


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Description

Introduction
Allergic rhinitis (AR) may be defined as an inflammation of the nasal mucous membranes caused by immunoglobulin E (IgE)-mediated (allergic) reaction to aeroallergens.

Allergic rhinitis,also called hay fever or pollinosis, literally means "allergic nose inflammation," where rhino means "to do with the nose" and the ending -itis simply refers to inflammation.

Causes
Allergic rhinitis is an allergic condition like asthma, meaning that the body tends to over react to certain types of outside substances. One way it overreacts is by producing antibodies that signal your immune system to release histamine and other chemicals. These chemicals cause the symptoms of allergic rhinitis including sneezing, runny nose, itchy or watery eyes, and even coughing.

Allergic rhinitis can be inherited, but you probably don't inherit particular allergies, such as to cat dander or ragweed. Instead, you just inherit the tendency to be allergic. Children have a 30% to 60% chance of developing allergic rhinitis if one of their parents is affected and a 50% to 70% chance if both parents have allergic rhinitis.

Symptoms:
Most people with allergic rhinitis know they have it, although it can sometimes be confused with the common cold.

Symptoms include runny nose; sneezing; itchy nose, mouth, throat, or eyes; and congestion. Other symptoms can also occur, such as tearing of the eyes, coughing, sore throat, wheezing, and headache.

24/07/2018

ACID PEPTIC DISEASE


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Description

Introduction
Acid peptic disease – commonly called APD – includes a number of conditions. All these conditions are the result of damage from acid and peptic activity in gastric secretions. APD occurs when the acid starts irritating the inner cells (mucosal layer) of the stomach. Acid peptic diseases mostly affect the oesophagus, stomach, and duodenum.

Causes
Acid peptic diseases are caused by the excessive presence of acid and pepsin. The two main types of acid peptic diseases are gastric and duodenal ulcer. However, they can also occur at the GJ Stoma, lower end of oesophagus, and Meckel’s diverticulum. According to duration and complexity of symptoms they can be acute or chronic.

The main causes are:

Smoking

Alcohol

Foods

Citrus or acidic fruits

Chocolate, caffeine, and alcohol

Greasy, fatty, spicy, and fried foods

Garlic, onions, and mint flavourings

Drugs, such as aspirin and types of steroids

Helicobacter pylori

Symptoms:
A digestive function disorder, acid peptic disease is characterised by dyspepsia (discomfort, heartburn, or nausea). However, the classic symptom of peptic ulcer disease, is defined as pain centered in the upper abdomen or discomfort characterised by fullness, bloating, distention, or nausea. Factors contributing to acid peptic disorder Acid peptic disorder results when the balance between the aggressive and defensive factors in the gastro duodenal mucosa is disrupted. This balance could be disrupted by factors such as H pylori infection, NSAID (Non-steroidal anti-inflammatory drug), and acid secretory abnormalities. A defect in bicarbonate production and acid neutralisation in the duodenal bulb is also a potent cause.

20/07/2018

HAIR DYE POISIONING

Hair dye poisoning: Diagnosis, management, and treatment protocol
M3 India Newsdesk Jul 13, 2018
After insecticides and pesticides, hair dyes are now the most frequently used poisoning agent among people due to their low cost and easy availability. This article is an effort to bring more awareness about the diagnosis and treatment protocol to be followed when a patient presents with suspected hair dye poisoning.

Recently in the news, there was a case in which a 27-year old Indian male consumed hair dye and died due to cardiorespiratory arrest. This is not an isolated incident. Many such cases of accidental or intentional hair dye poisoning due to Paraphenylenediamine (PPD) have been reported all over India.

In easy terms, PPD is a coal tar derivative which on oxidation produces Bandrowski's base. This base is allergic, mutagenic, and highly toxic, leading to dose-dependent systemic toxicity on ingestion.

A large dose will cause death within 6-24 hours of ingestion. The exact lethal dose of PPD in unknown, but it is estimated to be in the range of 7 to 10 g.

In India, PPD is the most commonly used toxic ingredient in hair dyes since decades. Moreover, PPD containing dyes are far cheaper than other hair dyes. Moreover, apart from PPD, the cheap hair dyes contain other toxic agents like naphthalene, toluene diamine, arsenic, bismuth, lead, pyrogallol, mercury, and silver.

Recognising a PPD poisoning
The initial symptoms of PPD poisoning usually develop within 6 hours of ingestion. It includes nausea, vomiting, abdominal pain, lacrimation, redness of eyes, swollen tongue, respiratory distress, and angioedema.

If left untreated, the condition may quickly worsen within 12 hours or sometimes over the next few days or week. If not managed on time, the patient might develop methemoglobinemia, rhabdomyolysis, acute tubular necrosis, arrhythmias, and intravascular haemolysis.

Diagnosing a case of PPD poisoning
There is no specific diagnostic test available for diagnosing PPD poisoning. An individual is suspected of PPD poisoning if the patient has angioedema with stridor, acute renal failure and rhabdomyolysis. These three symptoms are considered as the characteristic triad for suspecting PPD poisoning. The physician should do a complete blood count, renal, and liver functions test for confirming the diagnosis.

Treatment and management protocol for PPD poisoning
There is no specific antidote available for PPD poisoning. Therefore, early recognition and interventions are very important in managing PPD poisoning cases. Respiratory and gastric distress, arrhythmias and kidney failure are the major challenges that present during treatment.

Gastric lavage with 2% sodium bicarbonate has been found to be effective in some PPD cases. Rinsing the patient’s mouth with water and administration of milk have also been found to be effective in managing gastric symptoms. Supportive treatments such as tracheotomy, haemodialysis, oxygen therapy, mechanical ventilation and calcium correction were found to effective in managing the complications of PPD poisoning cases.

However, activated charcoal should not be used as PPD has low absorbability owing to its low molecular weight and hydrophilic nature.

Off late, the number of PPD poisoning cases are increasing in India. Apart from intentional or accidental poisoning, long-term regular use of PPD containing hair dyes can also cause toxicity. While increasing awareness about PPD poisoning can decrease the frequency of accidental poisoning, the lack of a specific diagnostic test and antidote are major concerns that also need to be addressed because early identification and interventions can prevent a fatal outcome

10/07/2018

ACNE TREATMENT


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Description

Introduction
Acne is an inflammation of the skin that causes blackheads, whiteheads, and red spots usually called "pimples" or "zits." The most common type of acne is acne vulgaris (vulgaris means common). Acne appears most often on the face, but can also be a nuisance on your chest, back, and upper arms. Acne affects about 90% of adolescents and can also occur in adulthood, usually affecting people aged 20 to 40 years.

Causes
1) Acne develops because your sebaceous glands are overactive- These glands normally produce small amounts of sebum, which is a thick mixture of oil and skin cells. When you have acne, these glands go into overproduction and the extra sebum can block your follicles and bacteria can move in, particularly the species called Propionibacterium acnes.

2) Hormones can cause an outbreak of pimples, or increase the number you get-The hormones that are active during puberty also trigger your sebaceous glands to produce more sebum. The hormones with the greatest effect on the oil glands are the androgens, the male hormones. Both men and women have androgens, but men have more.

In women, these hormones can also cause acne during the menstrual cycle, and that's why women often find that acne continues into adulthood. Hormones found in some types of birth control pills can also cause flare-ups of acne.

3) Eating junk food and chocolate normally has nothing to do with acne-Greasy hair and skin also doesn't cause acne, but they're often a sign of overactive sebaceous glands, which can cause acne. Research suggests that stress may worsen existing acne, but it doesn't cause it.

4) Other Reasons:

Some medications that contain androgen and lithium.
Greasy cosmetics.
Emotional stress.
Menstruation.