12/02/2023
We 👩⚕️are excited 😆 to celebrate 🎉 5 years of Sakariya Physiotherapy Clinic 🏥where we help the GOD 🙏 gifted children 👶 👧who are challenged 🧎🏻♀️by GOD 🙏 to live their full functionality🦾 life.
Contact information, map and directions, contact form, opening hours, services, ratings, photos, videos and announcements from Sakariya Physiotherapy Clinic, 4th floor, sargam doctor house, hirabaug, varachha Road, Surat.
(6)
27/01/2023
Republic Day Celebration # Pedialove # Teamwork # Special kids..😍
14/01/2023
Uttrayan Celebration # Special People # Happy Faces..😍
27/12/2022
Christmas Celebration wid Special Kids… Happiness is showing these cute smile on Special faces..😍😘
24/10/2022
Happy Diwali….😍
Festival vibes # Special kids # Smily Faces.. 😍😘
06/10/2022
Happiness # Smily faces # Team Work # Proud being part of these Special Heroes # Visit for more Information..😍😘
23/09/2022
Glimpse of group Therapy # Special kids # Happiness # Love # Team Work..😍😘
19/08/2022
Janmasthmi Celebration wid differently abled children # Proud to make Smile on their Faces # Unique bonds # Special Relations 😍 Happy Janmasthmi..😘
18/08/2022
Late Post…
15 August Celebration # Special kids # Happiness # Special Bonds..😍😘
21/03/2022
World Down Syndrome Day..😍😘
31/08/2021
Janmasthmi Celebration # Special Children # Proud to make Smile on their Faces # Sakariya Physiotherapy Clinic..😍😘
11/02/2021
On The Occasions Of Completing 3 Years Of Paediatric Department
We Feel Proud And Honoured To Serve Differently Abled Child.....
We Are Thankful To Everyone Who Have Supported And Trusted Us One or the Other Way.
Hope For Your Continues Support And Trust🥰
Thank you once again to all supportive doctors and all sweet patients
13/11/2020
06/10/2020
World Cerebral Palsy Day...
07/09/2020
Only 1 day left...
30/08/2020
Recently i have came across 2 patient of BRACHIAL PLEXUS NEURITIS, both patient have a symptoms which include severe pain over shoulder girdle with whole upper limb radicular pain and severe weakness of Deltoid n Rotator cuff muscles leads to inability of shoulder movements.
One of the patient 40 yr male had a dengue fever, during ongoing treatment of dengue patient have devoloped symptoms of brachial neuritis. Patient have done MRI of cervical spine which is normal, MRI of shoulder shows mild supraspinatous tear which is insignificant as per symptoms.
Another patient 25 yr male had an industrial accident, had a fracture of talus and calcaneum for which he had undergone surgery. Afterwards patient had devoloped symptoms of brachial neuritis without any symptoms of viral infection. Patients shoulder n cervical MRI is normal.
Unfortunately both patient have history of close contact with Covid-19 positive patients.
Can patient devolop neuritis after dengue?
Can patient have a neural effect of silent covid-19 infection?
28/05/2020
Today SAKARIYA PHYSIOTHERAPY CLINIC completed 14 years of successful journey, we have served more than 17000 patients in last 14 year.
There is tremendous role n support of many people in our success, so i heartily thankful to all of them.
Thank you all DOCTORS who trusted us and played immense role in our success
Thank you all my dear PATIENTS who is the pioneer of our success
Thank you all my PHYSIO FAMILY and my TEAM MEMBERS for such a wonderful journey
Thank you my FAMILY for all compromises for me and my success
Thank you VARACHHA
Thank you SURAT.
19/05/2020
Full fledged work started at sakariya physiotherapy clinic with
Social distancing- appointment wise patients
Sanitisation of instruments couch n floor
Frequent use of hand sanitizer for patients n relatives
Use of mask is compulsory for each patient n relatives
Moving ahead with proper safety of physios n patients
10/03/2020
Rainbow has 7 color together which simbolized as the special child ,it's proved that everyone has their own way to celebrate Holi.Wish you a very joyfull holi."
07/02/2020
07/02/2020
સાકરીયા ફિઝિયોથેરાપી ક્લિનિક છેલ્લા 14 વર્ષથી દર્દી ની સેવામાં કાર્યરત છે, છેલ્લા 2 વર્ષથી અમારી સેવાના વ્યાપ સ્વરૂપે બાળકો ની ફિઝિયોથેરાપી સારવાર કાર્યરત છે, 11/02/2020 ના રોજ 2 વર્ષ પૂર્ણ થયા નિમિતે કેમ્પ નું આયોજન કરેલ છે, જેમાં બાળકો નું નિદાન, રાહત દરે રિપોર્ટ, નિષ્ણાંત ડોક્ટરો દ્વારા કરવામાં આવશે.
14/01/2020
To become a Mother of special Child is extremely tough & dedicated Role. They are God gifted with enormous power to handle Special Child. We salute all such mothers specially associated with Us who have demonstrated their Creativity in Kite making Festival organised by Sakariya Physiotherapy Clinic, 4th floor Saragam Doctor house, Hirabag
11/10/2019
Clinical reasoning for lateral elbow pain
24/08/2019
Janmasthmi Celebrations with our Special Kanah nd Radhas with Modern & Traditional Style...Sakariya Team work in Janmasthmi Festival many more to Come...😍
11/07/2019
IMPORTANTS OF EARLY PHYSIOTHERAPY INTERVENTION IN CEREBRAL PALSY
Early intervention for children with Cerebral Palsy
• Limit the impact of Cerebral Palsy symptoms.
• Targets the challenges associated with a specific Cerebral Palsy diagnosis and puts the family on the offensive against this very difficult condition.
• Can come in many Forms, its overall effectiveness is often contingent on its timing and focus.
• Early intervention is Crucial when considering the future development of children with neurodevelopment conditions.
• The Best chance of preventing Mental disorders is by providing Early intervention
• It will minimize the impact of mental illness
So what types of early intervention would children with Cerebral Palsy benefit from?
The most common form of early intervention comes in the way of Physical therapy.
• Whether stretching/massaging muscles,
• working with gait
• relieving spasticity, or
• enabling movement in a wide form of manners or
• targeting stiffness in a number of different ways,
• Reducing tension or spasticity, while also retraining the body in how it functions.
• In order to maximize learning, the movements performed by the child should be self-initiating, and allow problem-solving in order to achieve a goal.
• Thus, it is imperative that therapists avoid being too hands-on by excessively holding or supporting the child while performing tasks.
• Rather, the therapist should use finger-tip control, or use equipment such as a strap, harness, wheeled apparatus or shoe holders.
• In addition, the therapist needs to enable the child to practice the desired movement/skill in a way that is challenging but feasible given his/her current physical and cognitive level and abilities.
• The child must be able to experience errors and successes, and be motivated to continue to try to acquire the desired skill.
09/06/2019
# Late Post.. #
These adolescents 👼🏻are our pride, we live with them , we laugh 😆 with them but never let them to cry 😢. Come and join with us to make them tomorrow's rising stats💫💫💫
Contact us to make an impossible to possible
25/12/2018
Christmas Celebration wid our differently able children nd Parents...
26/05/2018
આપના સહયોગ થી ૧૨ વર્ષ પુર્ણ કરવા નિમીતે સાકરીયા ફિઝીયોથેરાપી કિલનીકે આપની સેવા માટે કેમ્પ નુ આયોજન કરેલ છે.
11/05/2018
28/05/2013
our clinic have completed seven successful years.
09/05/2013
Hip muscles that can affect the knee-
The primary hip lateral rotators include the deep lateral rotators and gluteus maximus. The intrinsic or deep lateral rotators of the hip include the piriformis, gemellus superior and inferior, obturator internus and externus, and quadrates femoris muscles. As primary lateral rotators of the hip, the deep lateral rotators serve to provide precise control of rotation of the femoral head in the acetabulum, thus maintaining the integrity and stability of the hip similar to the way that the rotator cuff muscles provide control of the humeral head in the glenoid. In patients with knee pain syndromes, these muscles often become lengthened and weak, thus losing the precise control of the hip and allowing hip medial rotation to occur.
A large portion of the gluteus maximus muscle attaches into the ITB. Therefore shortness or stiffness of the gluteus maximus can contribute to relative stiffness/flexibility issues involving the ITB. Relative stiffness/flexibility as a result of shortness or stiffness of the gluteus maximus through the ITB seems to be more common in males than females and should be suspected if the patient sits in excessive hip abduction.
The secondary lateral rotators of the hip include the sartorius, the biceps femoris, and the posterior fibers of the gluteus minimus and medius. The posterior gluteus medius acts to abduct, extend, and laterally rotate the hip. The length and strength of the posterior gluteus medius is often affected by postural changes at the pelvis and hip. If an individual stands with the right iliac crest higher than the left or with the right hip in medial rotation, the posterior gluteus medius on the right may be lengthened and possibly weak, particularly if tested in the shortened position. Poor performance of this muscle is often a key factor in knee pain problems.
The TFL-ITB is a two-joint muscle complex that flexes, abducts, and medially rotates the hip and laterally rotates the tibia through its insertion onto the lateral tibial tubercle. The TFL-ITB also assists in stabilizing the knee when the knee is extended, although it does not actively extend the knee. When the TFL-ITB becomes short or too stiff, it can cause a compensatory motion such as lateral tibial rotation or valgus at the knee. The TFL-ITB can also contribute to patellar lateral glide as a result of its insertion on to the lateral patella.
The anterior fibers of the gluteus medius abduct, flex, and medially rotate the hip. Because the anterior fibers of the gluteus medius tend to be stronger than the posterior fibers, the imbalance often results in excessive medial rotation.
22/04/2013
FRSL - Type 2, non neutral lumbar vertebral somatic dysfunction
Address
4th Floor, Sargam Doctor House, Hirabaug, Varachha Road
Surat
395008
Opening Hours
| Monday | 8am - 9pm |
| Tuesday | 8am - 9pm |
| Wednesday | 8am - 9pm |
| Thursday | 8am - 9pm |
| Friday | 8am - 9pm |
| Saturday | 8am - 9pm |
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World Down Syndrome Day..😍😘
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Vel com
Hello
happy birthday for owner of this clinic.......!!!!!!!!
"U Sonic" ECG & Ultrasound Gel by 4Sure Group
U Sonic Ultrasound Gel is water based. It is typically clear and thick.
Non-toxic and Non-irritating to skin
It works by passing sound waves into a person’s body. It is placed on the patient’s skin at the beginning of the ultrasound examination or therapy.
Main materials:
Water-soluble & Hypoallergenic - Colorless or Blue
Non Toxic and non-irritable on skin
Characteristics:
Better sound transmission, distinctive image display.
No toxic and irritant effect on the skin.
No damage and corrosion to the ultrasound probe.
Used By:
U Sonic gel is widely applicable in hospitals and medical laboratories during ultrasound of internal organs of human bodies. Professionals like Cardiologist, Diagnostic Centers, Gynecologist, Hospitals, Orthopedics and Physiotherapist use these gels on routinely basis.
Packing:
Retailers Pack of 6 & 12 250ml bottles
Wholesale Pack - 24 & 48 250ml bottles
We do customise packing
For Orders:
Bulk/Import Order 15,000 250ml bottles
We do take Contract manufacturing Brand/Buyer's Name subject minimum Order of 30,000 250ml bottles per export shipment.
Delivery for Bulk/Import Orders:
Within 4 weeks from the date of advance received across the globe.
Contact:
We invite Sales Inquiries and Orders at [email protected]
U Sonic Ultrasound Gel is water based. It is typically clear and thick.
Non-toxic and Non-irritating to skin
It works by passing sound waves into a person’s body. It is placed on the patient’s skin at the beginning of the ultrasound examination or therapy.
Main materials:
Water-soluble & Hypoallergenic - Colorless or Blue
Non Toxic and non-irritable on skin
Characteristics:
Better sound transmission, distinctive image display.
No toxic and irritant effect on the skin.
No damage and corrosion to the ultrasound probe.
Used By:
U Sonic gel is widely applicable in hospitals and medical laboratories during ultrasound of internal organs of human bodies. Professionals like Cardiologist, Diagnostic Centers, Gynecologist, Hospitals, Orthopedics and Physiotherapist use these gels on routinely basis.
Packing:
Retailers Pack of 6 & 12 250ml bottles
Wholesale Pack - 24 & 48 250ml bottles
We do customise packing
For Orders:
Bulk/Import Order 15,000 250ml bottles
We do take Contract manufacturing Brand/Buyer's Name subject minimum Order of 30,000 250ml bottles per export shipment.
Delivery for Bulk/Import Orders:
Within 4 weeks from the date of advance received across the globe.
Contact:
We invite Sales Inquiries and Orders at [email protected]
Explanation of COMPENSATORY RELATIVE FLEXIBILITY with reference of PRONE KNEE BENDING.
CLINICAL OBSERVATIONS. Hypertrophy increases the stiffness of muscles through the range of motion. Because of the intersegmental variations in the springlike behavior of muscles, a reasonable hypothesis is that increased stiffness of one muscle group can cause compensatory movement at an adjoining joint that is controlled by muscles or joints with less stiffness. A common clinical observation is that when passively testing the length of a muscle, movement of a contiguous joint occurs long before the muscle is fully elongated. The movement of the contiguous joint is a compensatory motion. For example, if the lumbar spine is particularly flexible in the extension direction and the latissimus dorsi muscle is relatively stiffer, the lumbar spine will extend when the patient performs shoulder flexion, even before reaching the end of the length of the latissimus dorsi muscle.
Under optimal conditions when the therapist passively flexes the knee with the patient lying prone, which stretches the re**us femoris muscle, there should not be movement of the pelvis and spine except possibly near the end of the knee flexion range of 115 to 125 degrees. If movement of the pelvis and spine occurs between 45 and 115 degrees of knee flexion, it may be that segments of the spine are more flexible than the re**us femoris
muscle is extensible. As discussed later, this phenomenon does not necessarily mean that the re**us femoris muscle is short; but it implies that it is stiffer than the support provided to the pelvis and spine and therefore the stiffness produces lumbar extension. When a patient performs active knee flexion, there are automatic stabilizing responses that can affect the movement of the pelvis and spine. For example, during active knee flexion in the prone position, the contraction of the hamstring muscles will tilt the pelvis posteriorly. However, to stabilize and limit the movement of the pelvis, the hip flexors and back extensor muscles should contract. This stabilizing action of the muscles can either be excessive or insufficient. (Alterations of this stabilization pattern are discussed under the section on motor control impairments.) The examples given in Figure demonstrate different combinations of muscle stiffness and length impairments and their role in compensatory movements of the pelvis and spine.
The pelvis and lumbar spine are in the same correct alignment in the starting position. During either active and passive knee flexions, the following observations can be made:
1. Normal length of the re**us femoris muscle. The knee is flexed without lumbopelvic movement.
2. Short re**us femoris. Without lumbopelvic compensation, the knee is flexed without movement of the pelvis or lumbar spine, but knee flexion stops at 90 degrees, indicating short quadriceps muscles.
3. Stiff and short re**us femoris muscle with lumbopelvic compensation. The knee is flexed and the pelvis tilts anteriorly. The lumbar extension increases at 60 degrees of knee flexion, but the knee flexes to 135 degrees. When the therapist stabilizes the pelvis, the knee flexion stops at 90 degrees.
4. Stiffness, not shortness, of re**us femoris muscle with lumbopelvic compensation. The knee is flexed and the pelvis is tilted anteriorly. The lumbar extension increases at 60 degrees of knee flexion, but the knee is flexed to 135 degrees. When the therapist stabilizes the pelvis, the knee still flexes to 135 degrees.
5. Stiffness of re**us femoris muscle with automatic lumbopelvic stabilization. During passive motion, but not active knee flexion, the compensatory lumbar extension motion is observed.
6. Deficient lumbopelvic counter stabilization. At the initiation of knee flexion, the pelvis is tilted posteriorly and the lumbar spine slightly reduces its curve.
Explanation of Figure
1. Optimal balance of muscle stiffness and joint stability. The re**us femoris muscle is stretched without compensatory lumbopelvic motion. Therefore the stiffness of the anterior supporting structures of the spine and the passive stiffness of the abdominal muscles are greater than or equal to the stiffness of the re**us femoris muscle.
2. Shortness of re**us femoris muscle with counterbalancing stiffness of spinal structures and abdominal muscles. Because the knee flexes to only 90 degrees, the re**us femoris muscle is short and the muscle excursion does not reach the expected standard. However, lumbopelvic compensatory motion is not evident even though the re**us femoris muscle is short. It is not stiffer than the anterior supporting structures of the lumbar spine and the passive extensibility of the abdominal muscles.
3. Shortness of re**us femoris muscle with compensatory lumbopelvic motion (Position 3A). With knee flexion, compensatory anterior pelvic tilt and lumbar extension occurs, even before the muscle reaches the limit of its excursion. The pelvic tilt increases as the knee flexion range increases (Position 3B). When the pelvis is stabilized, which prevents anterior pelvic tilt, the knee flexion is limited to 90 degrees (Position 3C). In contrast to the situation in Position 2, the shortness of the re**us femoris muscle is associated with compensatory anterior pelvic tilt. Thus not only is the re**us femoris shortened, but its stiffness is also greater than the stiffness of the anterior supporting structures of the lumbar spine and the abdominal muscles. An important implication is that when the re**us femoris muscle is stretched to improve its overall length, the through-the-range stiffness remains. Therefore knee flexion elicits anterior pelvic tilt as long as the re**us femoris muscle is relatively stiffer than the structures preventing the anterior pelvic tilt or the lumbar extension. This phenomenon occurs even though the re**us femoris muscle is able to fully elongate. Correcting the faulty, compensatory pattern requires increasing the stiffness of the abdominal muscles and anterior supporting structures of the spine, in addition to stretching the re**us femoris muscle. It is possible that the compensatory motion occurs only when the re**us femoris muscle reaches the end of its excursion. At this point the resistance is particularly high and thus causes the compensatory motion of the pelvis. In this condition, increasing the length of the re**us femoris muscle eliminates the motion of the pelvis. This condition is not common.
4. Compensatory motion without muscle shortness. The knee flexes to 135 degrees (Position 4), but early in the range there is an associated anterior pelvic tilt and lumbar extension. When the pelvis is stabilized, the knee still flexes to 135 degrees. Clearly the compensatory motion is not associated with a short muscle. The most reasonable explanation is that the anterior supporting structures of the spine and the abdominal muscles are not as stiff as the re**us femoris muscle that has normal length. The relative degree of through-the-range stiffness of the re**us femoris versus the anterior trunk muscles and the anterior supporting structures of the spine is the key factor in determining the movement pattern and in creating the compensatory motion. The compensatory motion occurred long before the muscle reached the end of its range. Correction requires increasing the stiffness of the anterior trunk muscles.
5. Compensatory motion with passive flexion controlled by active muscle contraction. When the knee passively flexed, the stiffness of the re**us femoris muscle is greater than the stiffness of the anterior supporting structures of the spine and the abdominal muscles, which causes compensatory anterior pelvic tilt and lumbar extension (Position5A). When the hamstring muscles actively contract to flex the knee, the compensatory motion is eliminated (Position 5B). Possible explanations are that the posterior pelvic tilt elicited by hamstring contraction is sufficient to counteract the stiffness of the re**us femoris. Another explanation is that the abdominal muscles contract enough to counterbalance the anterior pelvic tilt and lumbar extension.
6. Exaggerated posterior pelvic tilt. In the normal joint stabilization pattern, the muscles that counteract the effect on joints (which are to remain stable) contract before the prime mover (Position 6). If they fail to do so or do not generate enough tension, the pelvis will posteriorly tilt. (Impairments in this control are discussed in the section on modulator elements.)
CLINICAL OBSERVATIONS. Hypertrophy increases the stiffness of muscles through the range of motion. Because of the intersegmental variations in the springlike behavior of muscles, a reasonable hypothesis is that increased stiffness of one muscle group can cause compensatory movement at an adjoining joint that is controlled by muscles or joints with less stiffness. A common clinical observation is that when passively testing the length of a muscle, movement of a contiguous joint occurs long before the muscle is fully elongated. The movement of the contiguous joint is a compensatory motion. For example, if the lumbar spine is particularly flexible in the extension direction and the latissimus dorsi muscle is relatively stiffer, the lumbar spine will extend when the patient performs shoulder flexion, even before reaching the end of the length of the latissimus dorsi muscle.
Under optimal conditions when the therapist passively flexes the knee with the patient lying prone, which stretches the re**us femoris muscle, there should not be movement of the pelvis and spine except possibly near the end of the knee flexion range of 115 to 125 degrees. If movement of the pelvis and spine occurs between 45 and 115 degrees of knee flexion, it may be that segments of the spine are more flexible than the re**us femoris
muscle is extensible. As discussed later, this phenomenon does not necessarily mean that the re**us femoris muscle is short; but it implies that it is stiffer than the support provided to the pelvis and spine and therefore the stiffness produces lumbar extension. When a patient performs active knee flexion, there are automatic stabilizing responses that can affect the movement of the pelvis and spine. For example, during active knee flexion in the prone position, the contraction of the hamstring muscles will tilt the pelvis posteriorly. However, to stabilize and limit the movement of the pelvis, the hip flexors and back extensor muscles should contract. This stabilizing action of the muscles can either be excessive or insufficient. (Alterations of this stabilization pattern are discussed under the section on motor control impairments.) The examples given in Figure demonstrate different combinations of muscle stiffness and length impairments and their role in compensatory movements of the pelvis and spine.
The pelvis and lumbar spine are in the same correct alignment in the starting position. During either active and passive knee flexions, the following observations can be made:
1. Normal length of the re**us femoris muscle. The knee is flexed without lumbopelvic movement.
2. Short re**us femoris. Without lumbopelvic compensation, the knee is flexed without movement of the pelvis or lumbar spine, but knee flexion stops at 90 degrees, indicating short quadriceps muscles.
3. Stiff and short re**us femoris muscle with lumbopelvic compensation. The knee is flexed and the pelvis tilts anteriorly. The lumbar extension increases at 60 degrees of knee flexion, but the knee flexes to 135 degrees. When the therapist stabilizes the pelvis, the knee flexion stops at 90 degrees.
4. Stiffness, not shortness, of re**us femoris muscle with lumbopelvic compensation. The knee is flexed and the pelvis is tilted anteriorly. The lumbar extension increases at 60 degrees of knee flexion, but the knee is flexed to 135 degrees. When the therapist stabilizes the pelvis, the knee still flexes to 135 degrees.
5. Stiffness of re**us femoris muscle with automatic lumbopelvic stabilization. During passive motion, but not active knee flexion, the compensatory lumbar extension motion is observed.
6. Deficient lumbopelvic counter stabilization. At the initiation of knee flexion, the pelvis is tilted posteriorly and the lumbar spine slightly reduces its curve.
Explanation of Figure
1. Optimal balance of muscle stiffness and joint stability. The re**us femoris muscle is stretched without compensatory lumbopelvic motion. Therefore the stiffness of the anterior supporting structures of the spine and the passive stiffness of the abdominal muscles are greater than or equal to the stiffness of the re**us femoris muscle.
2. Shortness of re**us femoris muscle with counterbalancing stiffness of spinal structures and abdominal muscles. Because the knee flexes to only 90 degrees, the re**us femoris muscle is short and the muscle excursion does not reach the expected standard. However, lumbopelvic compensatory motion is not evident even though the re**us femoris muscle is short. It is not stiffer than the anterior supporting structures of the lumbar spine and the passive extensibility of the abdominal muscles.
3. Shortness of re**us femoris muscle with compensatory lumbopelvic motion (Position 3A). With knee flexion, compensatory anterior pelvic tilt and lumbar extension occurs, even before the muscle reaches the limit of its excursion. The pelvic tilt increases as the knee flexion range increases (Position 3B). When the pelvis is stabilized, which prevents anterior pelvic tilt, the knee flexion is limited to 90 degrees (Position 3C). In contrast to the situation in Position 2, the shortness of the re**us femoris muscle is associated with compensatory anterior pelvic tilt. Thus not only is the re**us femoris shortened, but its stiffness is also greater than the stiffness of the anterior supporting structures of the lumbar spine and the abdominal muscles. An important implication is that when the re**us femoris muscle is stretched to improve its overall length, the through-the-range stiffness remains. Therefore knee flexion elicits anterior pelvic tilt as long as the re**us femoris muscle is relatively stiffer than the structures preventing the anterior pelvic tilt or the lumbar extension. This phenomenon occurs even though the re**us femoris muscle is able to fully elongate. Correcting the faulty, compensatory pattern requires increasing the stiffness of the abdominal muscles and anterior supporting structures of the spine, in addition to stretching the re**us femoris muscle. It is possible that the compensatory motion occurs only when the re**us femoris muscle reaches the end of its excursion. At this point the resistance is particularly high and thus causes the compensatory motion of the pelvis. In this condition, increasing the length of the re**us femoris muscle eliminates the motion of the pelvis. This condition is not common.
4. Compensatory motion without muscle shortness. The knee flexes to 135 degrees (Position 4), but early in the range there is an associated anterior pelvic tilt and lumbar extension. When the pelvis is stabilized, the knee still flexes to 135 degrees. Clearly the compensatory motion is not associated with a short muscle. The most reasonable explanation is that the anterior supporting structures of the spine and the abdominal muscles are not as stiff as the re**us femoris muscle that has normal length. The relative degree of through-the-range stiffness of the re**us femoris versus the anterior trunk muscles and the anterior supporting structures of the spine is the key factor in determining the movement pattern and in creating the compensatory motion. The compensatory motion occurred long before the muscle reached the end of its range. Correction requires increasing the stiffness of the anterior trunk muscles.
5. Compensatory motion with passive flexion controlled by active muscle contraction. When the knee passively flexed, the stiffness of the re**us femoris muscle is greater than the stiffness of the anterior supporting structures of the spine and the abdominal muscles, which causes compensatory anterior pelvic tilt and lumbar extension (Position5A). When the hamstring muscles actively contract to flex the knee, the compensatory motion is eliminated (Position 5B). Possible explanations are that the posterior pelvic tilt elicited by hamstring contraction is sufficient to counteract the stiffness of the re**us femoris. Another explanation is that the abdominal muscles contract enough to counterbalance the anterior pelvic tilt and lumbar extension.
6. Exaggerated posterior pelvic tilt. In the normal joint stabilization pattern, the muscles that counteract the effect on joints (which are to remain stable) contract before the prime mover (Position 6). If they fail to do so or do not generate enough tension, the pelvis will posteriorly tilt. (Impairments in this control are discussed in the section on modulator elements.)
Require physiotherapist BPT or MPT, part time or full time at Sakariya Physiotherapy Clinic Sargam Doctor House,Hirabaug, varachha.
Contact on: 9228469866
Contact on: 9228469866
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