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Ageless Fitness Institute, 3046 SOUTH DELAWARE Avenue, MILWAUKEE, Bay View, WI (2025)

MUCH INVOLVES MIND-BODY FOCUSES, -- INVOLVING SOME DEGREE OF BODY MOVEMENT "HORMESIS".

11/01/2025

REALLY-! ?? IT'S MOSTLY MODIFIED "TAI-CHI". CALLED "REI-HO". BUT SEEMS FAR LESS NUANCED. WHY USE WARRIOR TRAINING FOR AGING POPULATIONS - THAT TYPICALLY NEED SPINAL DECOMPRESSION & HEIGHTENED NUTRIENT UPTAKE & BRAIN HEALTH FOCUS FIRST. - MORESO THAN "UPRIGHT QUAD LOADING".

GYMS & GYM EQUIPMENT ARE FAR MORE VERSITAL AND SAFER.

"Traditional Samurai Movement Improves Knee Strength and Mobility for Seniors"

Story at-a-glance.

A traditional Japanese practice called Rei-ho improved knee strength by more than 25% in just three months of short daily sessions.

The slow, upright movements in Rei-ho target your quadriceps, the key muscles that help you stand, walk, and climb stairs with ease.

Practicing Rei-ho takes only five minutes a day, making it realistic for busy schedules and easy to stick with long term.

Slow, controlled movement reduces stress on your joints, lowering the risk of injury while still delivering powerful strength gains.

Stronger knees from Rei-ho lower your risk of falls and help you maintain independence as you age.

Aging changes more than appearance — it directly impacts how strong and mobile your body stays over time.

One of the earliest and most noticeable shifts happens in your legs, where muscle loss erodes balance, stability, and confidence.

Without steady strength, even routine movements like standing from a chair or walking across the room become more difficult.

The problem is not just weakness — it's the cascade of risks that follow. Loss of leg strength raises the likelihood of falls, fractures, and hospital stays, which often trigger a downward spiral in independence. Once daily tasks feel unsafe, quality of life drops and recovery from setbacks becomes harder.

Conventional strength training has long been the answer, yet it's not practical for everyone. Heavy weights, gyms, and high-intensity exercise often discourage older adults who fear injury or lack time. What many people need is a simple, safe, and sustainable way to keep their legs strong enough to support the life they want to live.

That search for practical solutions has led researchers to revisit movement patterns hidden in tradition. One example is Rei-ho, a centuries-old practice from Japan's samurai culture, now being studied as a modern tool for protecting mobility.

Samurai-Inspired Movements Deliver Significant Strength Gains.

A study published in the Tohoku Journal of Experimental Medicine explored whether a traditional Japanese movement practice known as Rei-ho could strengthen knee muscles in adults.1 Rei-ho's effectiveness has cultural origins. The movements come from traditional Japanese practices like floor-sitting, using futons, and squat toilets, which naturally involved frequent squatting and standing.

As Western-style chairs and beds replaced these habits, opportunities for natural strength training disappeared. Rei-ho revives these beneficial motions in a structured way that fits modern life. The researchers were interested in testing if these short, structured movements rooted in samurai etiquette could produce measurable gains in leg strength without the need for heavy weights or long workout sessions.

•Rei-ho originated as a form of daily movement practiced by samurai — It's defined by slow, deliberate sit-to-stand motions. The key features include keeping your feet together, avoiding leaning your upper body forward, moving at a steady rhythm, and holding each squat or stand for four to five seconds. Unlike fast squats, your buttocks remain above your knees, which forces your thigh muscles to stay engaged longer and builds strength without added weights.

•Participants were healthy adults with no prior training — The trial included 34 healthy adults who had never practiced Rei-ho before. They were randomly assigned to either a training group or a control group. Those in the training group practiced Rei-ho for just five minutes per day, four times a week, over three months. The control group continued their normal routines with no new exercise.

•Knee strength increased by more than 25% — The results were striking: participants who practiced Rei-ho increased their knee extension strength by 25.9% compared to only 2.5% in the control group. This means that the training was not only effective but also highly efficient, especially when compared with more time-consuming or equipment-heavy exercise programs.

•Rei-ho improved quadriceps strength — Your quadriceps, the large muscle group at the front of your thighs, plays a central role in standing, walking, climbing stairs, and preventing falls. The study showed that Rei-ho movements, which keep your trunk upright and push your knees forward, placed more emphasis on activating the quadriceps than conventional squats. That activation translated directly into stronger, more resilient legs.

•Improvements occurred within three months of practice — Over the 12-week period, participants consistently practiced a set of controlled movements, such as slow squats and sit-to-stands, with each motion taking four to five seconds to complete. This slow pace forced the muscles to stay engaged longer, and the accumulated effort over time produced clear, measurable strength gains without the strain of heavy lifting.

Daily Rei-ho Requires a Minimal Time Commitment.

The total training session lasted only five minutes per day, and most participants averaged fewer than five days of training per week. Despite the small time investment, the gains were comparable to or even greater than those seen in previous studies requiring much higher training volumes. This highlights how Rei-ho is both realistic and sustainable for people with busy schedules.

•Benefits were seen in those who practiced consistently — Participants who followed the training protocol averaged 4.6 days per week and experienced strong improvements in knee extension strength. This finding shows that even small amounts of effort, when done consistently, deliver noticeable results, reinforcing the importance of routine for long-term strength and mobility.

•Rei-ho produced better results than conventional squat programs — When compared with past squat-based training studies, Rei-ho stacked up impressively. Some squat programs with up to 99 repetitions per day over several months only increased strength by 8% to 16%, while Rei-ho achieved more than 25% improvement with fewer total repetitions and less time commitment. This makes Rei-ho a highly efficient choice for strengthening your legs.

•Muscle activation patterns explained the strength gains — Rei-ho movements were deliberately slow, upright, and controlled. Unlike conventional squats that rely on leaning forward to balance, Rei-ho shifted more torque onto the knees and quadriceps. This biomechanical loading pattern required sustained contraction of the thigh muscles, creating the conditions for strength improvements without heavy weights.

•Slow movement reduced risk of injury — One of the most important findings was safety. The study emphasized that the slow, deliberate pace of Rei-ho avoided sudden or jerky motions that often injure ligaments. Because movements lasted four to five seconds in each direction, participants built strength while protecting the knees and back. Only one participant reported temporary knee discomfort, which resolved without stopping training.

•Rei-ho builds strength while keeping blood pressure stable — High-intensity strength training often spikes blood pressure, which is risky for older adults. In contrast, slow bodyweight movements like Rei-ho kept intensity low and stress on the cardiovascular system minimal. This makes the practice especially suitable for seniors or anyone concerned about heart health.

The study proves that even if you avoid gyms or heavy weights, you still have options to protect your mobility and independence. Adding just five minutes of Rei-ho to your day provides a realistic, effective way to build leg strength, lower your risk of falls, and keep everyday movements easier as you age. By practicing slowly and consistently, you gain confidence and control over your health without needing expensive equipment or long workouts.

Simple Steps to Strengthen Your Legs and Protect Your Independence.

Losing strength in your legs isn't inevitable. The root cause of weakness in your knees and thighs is lack of consistent, targeted use. Sitting too much and avoiding strength-based movement sets the stage for muscle loss, making everyday activities harder. If you want to stay independent, prevent falls, and keep doing the things you love, you need simple strategies that train your legs in ways that fit your daily life. Here's how you can start:

1.Practice Rei-ho daily for five minutes — Begin with Rei-ho, which involves slow, deliberate sit-to-stands and squats, where each movement lasts four to five seconds. Just five minutes a day, at least four days a week, improves knee strength by over 25% in three months.2 You don't need weights, equipment, or even a gym — just your body and a chair.

2.Use natural movements throughout your day — If you avoid exercise routines, you can still build strength by making small changes. Try sitting on the floor instead of the couch, or use stairs instead of elevators. These movements mimic the natural squats and stands that used to be part of everyday Japanese life. Each time you move this way, you train your legs to support you better.

3.Go slow to protect your joints — Fast, jerky movements stress your knees and back. By slowing down, you increase muscle activation while lowering your risk of injury. Think of Rei-ho as moving in slow motion: five seconds down, five seconds up. This steady rhythm keeps your quadriceps working hard while keeping your joints safe.

4.Stay consistent rather than intense — Your body responds to steady effort, not one-time bursts. If you're busy or easily discouraged by long workouts, remind yourself that shorter, regular practice works better. Even four sessions a week is enough to create change. Consistency builds both strength and confidence, giving you proof that your actions pay off.

5.Track your progress and challenge yourself — You're more likely to stick with a habit if you see results. Keep a simple log of your sessions, or mark each day on a calendar. Over time, increase the number of repetitions slightly, or add an extra practice day. This gives you a sense of accomplishment and turns training into a personal challenge you can win.

By putting these steps into action, you address the root cause of weakness — muscle loss from inactivity — and replace it with habits that restore strength and stability. This isn't about punishing workouts. It's about using simple, natural movements to keep your body strong enough to live life on your terms.

FAQs About Rei-ho for Leg Strength.

Q: What is Rei-ho and why does it matter for leg strength?

A: Rei-ho is a traditional Japanese samurai practice involving slow, controlled squats and sit-to-stand movements. A study in the Tohoku Journal of Experimental Medicine found that practicing Rei-ho for just five minutes a day increased knee strength by more than 25%.3 Stronger knees help you stay mobile, reduce your risk of falls, and protect your independence as you age.

Q: How much time do I need to practice Rei-ho for results?

A: The study showed that five minutes a day, at least four times per week, was enough to significantly increase knee extension strength. Consistency matters more than duration, so sticking with this routine gives you better results than longer but less frequent workouts.

Q: How is Rei-ho different from regular squats?

A: Unlike conventional squats that involve leaning your trunk forward, Rei-ho keeps your torso upright and pushes your knees forward. This posture shifts more load to your quadriceps — the large muscles at the front of your thighs — making them stronger with less strain on your back.

Q: Is Rei-ho safe for older adults or people with knee concerns?

A: Yes. Because the movements are slow and bodyweight-based, Rei-ho lowers the risk of injury compared to heavy weightlifting. The deliberate pace also avoids sudden stress on your joints, making it safer for older adults and those worried about joint pain.

Q: What practical steps can I take to start strengthening my legs today?

A: You can begin with five minutes of Rei-ho, practice standing from a chair slowly, and add more natural movements like using stairs or sitting on the floor. Track your progress by logging sessions or marking a calendar. Over time, this builds confidence, consistency, and strength that helps you stay active and independent.

A centuries-old samurai practice is proving to be one of the simplest ways to restore strength and protect independence as you age.

11/01/2025

ALZHEIMERS, DMSO, & THE BETA AMALOID PLAQUE "SCAM". & ALSO DEMENTIA ; OTHER ISSUES.

Story at-a-glance.

Alzheimer’s disease is commonly thought to result from abnormal plaque buildup in the brain that gradually destroys brain tissue.

Almost all Alzheimer’s research for decades has been directed toward eliminating amyloid, even after the basis for much of this work was shown to stem from fraudulent research.

The billions spent on amyloid Alzheimer’s research have only produced three drugs, all of which offer minuscule benefits and severe side effects.

In contrast, affordable and straightforward treatments that reduce dementia or the preceding cognitive impairment have been maligned and buried by the medical industry.

DMSO for example, has incredible neuroprotective qualities that have spared many stroke and spinal cord injury victims from a life of “incurable” disability. Decades of forgotten research also show it treats cognitive impairment and dementia.

This article will review the great amyloid scam and the simple therapies for cognitive decline we’re never told about.

Medicine is strongly biased towards adopting biochemical models of disease as this facilitates costly therapeutics being developed for each disease and hence sustains the medical industry.
Unfortunately, in many cases, the biochemical approach to disease, at best, can manage symptoms, and as a result, many conditions remain “incurable” while non-patentable natural therapies that can cure them languish in obscurity.

That’s why, despite spending an ever increasing amount of money on Alzheimer’s research (e.g., the NIH spent 2.9 billion in 2020 and 3.9 billion in 20241), we’ve still failed to make any real progress on the disease. This is particularly remarkable given the vast costs to the country (e.g., last year Alzheimer’s was estimated to cost the United States 360 billion dollars2) and the even greater social costs that accompany it.

The Amyloid Juggernaut.

In 1906, plaques (of amyloid) in the brain were identified as the cause of Alzheimer’s disease. As the years have gone by, the majority of research for treating Alzheimer’s disease has been targeted at eliminating these plaques. Unfortunately, to quote a 2022 article:3

“Hundreds of clinical trials of amyloid-targeted therapies have yielded few glimmers of promise, however; only the underwhelming Aduhelm has gained FDA approval. Yet Aβ still dominates research and drug development. NIH spent about $1.6 billion on projects that mention amyloids in this fiscal year, about half its overall Alzheimer’s funding.

Scientists who advance other potential Alzheimer’s causes, such as immune dysfunction or inflammation, complain they have been sidelined by the ‘amyloid mafia.’ Forsayeth says the amyloid hypothesis became ‘the scientific equivalent of the Ptolemaic model of the Solar System,’ in which the Sun and planets rotate around Earth.”

Note: Frequently, when a faulty paradigm fails to explain the disease it claims to address, rather than admit the paradigm is flawed, its adherents will label each conflicting piece of evidence as a paradox (e.g., the French “paradox” disproves the notion cholesterol causes heart disease4) and dig deeper and deeper until they can find something to continue propping up their ideology (e.g., cholesterol reducing statins provide almost no benefit for heart disease while having significant side effects yet continue being pushed on patients).

The consistent failure of the amyloid model to cure Alzheimer’s gradually invited increasing skepticism towards it, which resulted in more and more scientists studying alternative models of the disease. Before long, they found other factors played a far more significant role in causing the disease (e.g., chronic inflammation), and by 2006, this perspective appeared poised to change the direction of Alzheimer’s research.

In response, the amyloid proponents pivoted to defending their failed hypothesis was due not to amyloid clumps, but rather toxic parts of it (oligomers) and a Nature 2006 paper appeared which identified a previously unknown toxic oligomer, Aβ*56, and provided proof that it caused dementia in rats.5

This paper cemented both the amyloid beta and toxic oligomer hypotheses (as it provided the proof many adherents to the theory had been waiting for) and rapidly became one of the most cited works in the field of Alzheimer’s research. Its authors rose to academic stardom, produced further papers validating their initial hypothesis, and billions more were invested by both the NIH and the pharmaceutical industry in research of the amyloid and toxic oligomer hypothesis.

It should be noted that some were skeptical of their findings and likewise were unable to replicate this data, but rarely had a voice in the debate:

“The spotty evidence that Aβ*56 plays a role in Alzheimer’s had [long] raised eyebrows.6 Wilcock has long doubted studies that claim to use ‘purified’ Aβ*56. Such oligomers are notoriously unstable, converting to other oligomer types spontaneously. Multiple types can be present in a sample even after purification efforts, making it hard to say any cognitive effects are due to Aβ*56 alone, she notes — assuming it exists.

In fact, Wilcock and others say, several labs have tried and failed to find Aβ*56, although few have published those findings. Journals are often uninterested in negative results, and researchers can be reluctant to contradict a famous investigator.”

The Amyloid Scandal.

At the end of 2021, a neuroscientist physician was hired by investors to evaluate an experimental Alzheimer’s drug and discovered signs that its data consisted of doctored Western Blots (and therefore erroneous assessments of what oligomers were present within research subjects’ brains).7 As he explored the topic further, he discovered other papers within the Alzheimer’s literature had been flagged for containing doctored Western Blots.

Note: Western blots, used to test for proteins, are one of the few easily detectable forms of research fraud (e.g., we discovered Pfizer submitted fake Western blots to regulators to “prove” their vaccine worked). Regrettably, far more undetectable fraud exists throughout the scientific literature (e.g., independent researchers comparing regulatory submissions discovered Pfizer also submitted doctored data on where the COVID vaccine is distributed in the body8).

Before long, the neuroscientist noticed three of those suspect papers had been published by the same author and decided to investigate the author’s other publications. This led him to the seminal 2006 Alzheimer’s publication, which contained clear signs of fraud.9

As investigation then uncovered 20 doctored papers written by the author, 10 of which pertained to Aβ*56 (along with a co-researcher attesting to earlier scientific misconduct by the author).

The Amyloid Industry.

One of the remarkable things about this monumental fraud was how little was done about it. For example, the NIH was notified in January 2022, yet in May 2022, beyond nothing being done, the NIH gave the suspect researcher a coveted $764,792 research grant (signed off by another one of the authors of the 2006 paper10).

In July 2022, Science published an article exposing the incident and the clear fraud that had occurred.11 Despite this, the researcher was allowed to remain in his position as a tenured medical school professor.12 It was not until June 2024 that the 2006 article was retracted at the request of the authors13 — all of whom denied being at fault and insisted the doctored images had not affected the article’s conclusions.

Eventually, on January 29, 2025, during his confirmation hearing, RFK cited the paper as an example of the institutional fraud and wasted tax dollars within the NIH, and a few days later, the suspect researcher announced his resignation from the medical school professorship (while still maintaining his innocence).14

This odd behavior (e.g., the medical field continues to insist the proven fraud has not disproven the Amyloid hypothesis) likely results from how much money is at stake — beyond the research dollars, roughly 7 million adults have Alzheimer’s — equating to hundreds of billions in potential (Medicare funded) sales each year.15

The Failed Amyloid Drugs.

Recently, a monoclonal antibody that made immune cells target amyloid demonstrated limited success in treating Alzheimer’s — which was embraced as revolutionary by the medical community, the pharmaceutical industry, and drug regulators. In turn, the first new drug received accelerated approval (which the FDA proudly announced).16 The second then received a quiet backdoor approval (due to the immense controversy surrounding the first),17 and the third was partially approved a year and a half later.18

alzheimers new treatment options.

Each year, JP Morgan (Chase Bank) hosts a private conference for pharmaceutical investors that sets the tone for the entire industry. In 2023, its focus (covered in detail here) was on the incredible profitability of the new Alzheimer’s drugs and the GLP-1s like Ozempic (which the FDA has also relentlessly promoted). Most remarkably, the (widely viewed as corrupt) FDA commissioner was a keynote speaker, and a few days before the conference, had enacted the second backdoor approval.

However, despite the rosy pictures painted around the drugs (which each attacked different aspects of amyloids), they were highly controversial as:

•The FDA’s independent advisory panel, in a very unusual move, voted 10-0 (with one abstaining) against approving Aduhelm, the first amyloid drug (which targeted amyloid plaques), but the FDA approved it anyways. In a highly unprecedented move, three of the advisors then resigned, calling it “probably the worst drug approval decision in recent U.S. history.”19

•That drug was priced at $56,000 a year — making it sufficient to bankrupt Medicare, (which attracted a Congressional investigation).20

•Brain swelling or brain bleeding was found in 41% of patients enrolled in its studies.21 Additionally, headaches (including migraines and occipital neuralgia), falls, diarrhea, confusion, and delirium were also notably elevated compared to placebo.

•No improvement in Alzheimer’s was noted; rather one analysis found it slowed the progression of Alzheimer’s by 20% (although this could have been a protocol artifact rather than a real effect).

The second monoclonal antibody (which targeted amyloid precursors) had a somewhat better risk benefit profile22 (only 21% experienced brain bleeding and swelling due to reduced targeting of stable amyloid plaques), and 26.4% reduction in the progression of Alzheimer’s was detected in the trial (which for context, translated to a 0.45 reduction on a scale where a reduction of at least 1 to 2 points is needed to create an impact which is in any way meaningful for a patient).

The third monoclonal (which targeted amyloid plaques thought to be more pathologic)23 was also contested as it caused 36.8% of recipients to develop brain bleeding or swelling, like the other amyloid medications, frequently caused headaches and infusion reactions (e.g., nausea, vomiting, changes in blood pressure, hypersensitive reactions or anaphylaxis) and there were reasons to suspect the trial had greatly overstated its minimal benefits.

Remarkably, despite widespread protest against the third drug, the FDA’s new advisory panel voted unanimously in favor of it, even though it had a very similar mechanism, efficacy, and toxicity to the previously unanimously rejected amyloid drug.

It should therefore come as no surprise that, when the British Medical Journal conducted an independent investigation, it found that, within publicly available databases, 9 out of 9 (assessable) members of the advisory committee had significant financial conflicts of interest.24

Fortunately, despite the aggressive promotion of amyloid drugs and the industry’s best attempts to promote the sector, the market somewhat recognized how bad they were. The first drug had its price halved (then was withdrawn as no one wanted it — making around 5 million dollars total),25 while the other two have had very modest sales (e.g., 290 million for the most popular one26).

What Amyloids Drugs Show Us
From this, four things stand out:

•These drugs consistently damage brain tissue, indicating that their mechanism of action was inherently dangerous (e.g., it creates brain swelling by causing immune cells attacking amyloid also to attack brain tissue, or it creates brain bleeding by removing amyloid plaque that patches vessel walls and stabilizes brain tissue). Remarkably, despite this issue being recognized, it has not deterred the usage of these class drugs.

•Removing amyloid offers minimal benefit and may be counterproductive. In fact, one of the only protocols that has had proven success in treating Alzheimer’s instead views amyloid as a protective mechanism the brain uses to prevent further damage.

•An absolutely absurd amount of money and time has been wasted on this endeavor due to the medical field’s need to find a patentable drug.

•The focus on these lucrative drugs has diverted attention from other (off-patent) treatments that are more likely to help Alzheimer’s patients.

For example, a randomized controlled trial which gave MCTs derived from coconut found that over 6 months,27 80% remained stable or improved — which for context, is better than what any of the amyloid drug trials showed, and more importantly, does not cause brain bleeds (and costs a lot less than the annual rough $30,000 cost for those drugs).

Note: Numerous readers have shared that coconut oil improved their relative’s dementia.

Likewise, very few are aware of a 2022 study that should have revolutionized the entire Alzheimer’s field:28

change in cognitive performance.

Note: The RECODE protocol was based around identifying the underlying cause of a patient’s cognitive impairment (as five different things can cause dementia), and then providing appropriate natural therapies to address the applicable cause. Since then, many others have replicated its success in their patients.

DMSO and Dementia.

Dimethyl Sulfoxide (DMSO) is a naturally occurring compound that has a variety of unique healing properties that allow it to rescue tissues from dying and revive those damaged from previous injuries — best demonstrated by decades of evidence showing DMSO can heal strokes, brain bleeds, severe concussions, and spinal cord injuries and save patients from a lifetime of paralysis.

lance grindle dmso
As many of DMSO’s mechanisms directly counteract the processes that trigger dementia, I have received many accounts like these from readers:

“My uncle’s wife has dementia and has been unable to speak for over a year. My mom recently visited them and told them about DMSO. He began to give his wife DMSO orally. After two weeks she began to talk again.29

I read the article and began giving it to my 93 year old mother in her juice every morning at the end of November. She has had some form of dementia for over 15 years. Since taking the DMSO, she no longer suffers with severe sundowners. She is more ‘with it’ and can communicate and laugh with us. Her personality is back. She is crossing her legs again and lifting her pinky finger when drinking her coffee. It’s a lot of little things that make a difference.

She is able to understand when I am asking her to use the bathroom. She is more cognitive and has started coloring in her coloring books again.30

I deeply appreciate your posts on DMSO. You helped bring spontaneous interaction back into the life of my father with Alzheimer’s.”31

Numerous studies support these experiences:

•When rats had their carotid arteries surgically modified to reduce the blood going to the brain, DMSO prevented both the neuronal damage and the significant loss of spatial memory and learning that otherwise occurred.32

•In a similar study, rats who developed persistent and severe memory impairment from reduced brain blood flow received DMSO and FDP for 7 days, which improved their memory by 54%, nearly reaching the cognitive function rats whose blood flow was never cut off.33,34

•In rats, daily DMSO counteracted memory impairment induced by intracerebroventricular STZ infusions,35 while in a similar study,36 DMSO and Ginkgo biloba improved learning and memory in rats given Alzheimer’s disease.

•Drinking minute amounts of DMSO prevented the visual degeneration otherwise seen in rats engineered to have early Alzheimer’s disease.37 In another study of those rats, it protected key brain cells from disappearing and enhanced both their spatial memory and smell (while decreasing their anxiety).38 Likewise, in rats bred to develop cerebellar disorders, DMSO prevented age-related deterioration of certain cognitive functions (e.g., memory and spatial learning).

These results have also been replicated in humans:

•In 18 patients with probable Alzheimer’s after three months, DMSO greatly improved memory, concentration, and communication, alongside a significant decrease in disorientation in time and space.39

•In 104 elderly adults with dementia due to cerebrovascular diseases, concussions, or Parkinson’s, DMSO combined with amino acids significantly improved their cognition and motor function.40

•In 100 patients with cerebrovascular diseases (many of whom had dementia),41 DMSO caused almost all to have their cardiovascular parameters improve and:

“Recovery from the general symptoms was positive; there were favorable changes which were reflected in a feeling of well being, the recovery of agility, changes of mood from depressed to gay, improvement of sleeping, and clearer speech. As regards the ‘focal’ results, accelerated recovery from hemiplegia and hemiparesia was registered. A speedier recovery of speech in cases of defined or indicated aphasia took place.”

Conclusion

The Alzheimer’s story illustrates how medical science’s relentless focus on commercializable products has failed the country. This must be replaced with prioritizing understanding the root causes of the chronic illnesses we face.

Fortunately, now that MAHA can set national health policy and independent media has broken the media’s monopoly over the truth due to the lies we saw throughout COVID-19, more and more are stepping outside the medical orthodoxy to pursue therapies that can actually heal them. An opportunity like this has never existed before, and it is critical each of us brings attention to the need for real medicine before the window to fundamentally change the practice of medicine closes.

Author’s Note: This is an abridged version of a longer article which discusses the actual causes and treatments for Alzheimer’s disease and the cognitive decline which precedes it. That article, along with additional links and references, can be read here. Additionally, a companion article on how DMSO treats neurological injuries (e.g., strokes, brain hemorrhages, traumatic brain injuries, spinal paralysis and developmental delay) can be read here.

A Note from Dr. Mercola About the Author

A Midwestern Doctor (AMD) is a board-certified physician from the Midwest and a longtime reader of Mercola.com. I appreciate AMD’s exceptional insight on a wide range of topics and am grateful to share it. I also respect AMD’s desire to remain anonymous since AMD is still on the front lines treating patients. To find more of AMD’s work, be sure to check out The Forgotten Side of Medicine on Substack.

Billions wasted on failed Alzheimer's drugs while proven affordable treatments like DMSO and natural therapies remain buried by the medical industry.

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11/01/2025

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