Full Potential-Equine Sports Therapy & Barefoot Trimming

Full Potential-Equine Sports Therapy & Barefoot Trimming Full Potential, Equine Sports Therapy and Barefoot Trimming, working on your whole horse to achieve balance and health.

I have spent nearly my entire life with horses and it was never a complacent or unscholarly one. My first exposure to them began by working as a young girl with a highly respected and internationally recognized dressage rider, trainer, and judge. I stayed in her tutelage for several years as a professional groom and many foundations of responsibility and attention to detail regarding the healthcare of our equine athletes began during this time. In 2011, I was certified as an Equine Massage Therapist by Mary Schreiber of Equissage, a fully accredited program with the American Veterinary Medical Association. I was originally introduced to barefoot trimming in the early 2000s by James and Yvonne Welz of The Horse’s Hoof, after already spending quite a few years riding along with established farriers in the area and assisting. In more recent years, I have attended several equine chiropractic seminars with Dr. Bill Hampton of Equine Spine, Pete Ramey of Hoof Rehab, and Paige Poss of Anatomy of the Equine. I continue my education in the form of clinics and wet labs across the country, each year.

The first thing everyone considers for a horse that is sensitive across their flank is to blame gastric ulcers and right...
03/21/2026

The first thing everyone considers for a horse that is sensitive across their flank is to blame gastric ulcers and rightfully so, as it is estimated that 50-90% of adult horses suffer this—90-100% of performance horses!

Another common area for reactivity can be found along the lumbar spine.

Can flank sensitivity be attributed to L1 nerve compression?

The nerves that originated from the L1 space wrap around the flank and into the belly. They also run across the flank, over the stifle and end in the gaskin.

This could attribute to pain that mimics colic or ulcers, it can attribute to stifle pain which changes how the horse moves through the gaits or influences how they position while in rest.

Gastric ulcers should still be our primary concern, but we should also not rule out L1 compression!

02/28/2026

⭐⭐⭐⭐⭐ Rated 4.8/5 by loved customers

Excellent post.
02/26/2026

Excellent post.

𝐀𝐫𝐞 𝐖𝐞 𝐒𝐭𝐚𝐫𝐭𝐢𝐧𝐠 𝐇𝐨𝐫𝐬𝐞𝐬 𝐓𝐨𝐨 𝐘𝐨𝐮𝐧𝐠? 𝐖𝐡𝐚𝐭 𝐭𝐡𝐞 𝐒𝐜𝐢𝐞𝐧𝐜𝐞 𝐀𝐜𝐭𝐮𝐚𝐥𝐥𝐲 𝐒𝐚𝐲𝐬 🏇 🦴

If you’ve followed my page for any length of time, you know I don’t sidestep controversy. Whether it’s blanketing, NSC in grass, or even salt (yes, that one surprised me too), the most debated topics are often the most worth examining. Because where uncertainty exists, I prefer to replace opinion with evidence. And there is nothing I love more than digging into peer-reviewed research to determine if what’s commonly accepted truly holds up to scientific scrutiny.

And few topics ignite more debate in the horse world than the question of when a young horse should begin work.

On one side, there’s concern that starting too early risks long-term soundness issues.

On the other, some argue that thoughtful early training may actually support bone development.

So instead of arguing from a point of instinct or tradition, I think it’s time to take a look at what the research actually says.

𝐆𝐫𝐨𝐰𝐭𝐡 𝐏𝐥𝐚𝐭𝐞 𝐂𝐥𝐨𝐬𝐮𝐫𝐞 𝐢𝐧 𝐇𝐨𝐫𝐬𝐞𝐬

Let’s begin by addressing the color-coded diagram of an equine skeleton that frequently circulates social media. This diagram illustrates when growth plates close, which begins in the lowest parts of each limb and moves up the skeleton sequentially, ending at the spine. This diagram is popular as many use it to justify recommendations on when to start horses.

I decided to do some digging to track down the origin of this information, and my investigation led me to a table in a book that was published in 1975. This table cites literature that evaluated the closure of the epiphyseal growth plate in the appendicular skeleton (forelimbs and hindlimbs) through radiographs (Getty, 1975).

Since then, a review by Rogers et al. (2021) was published and concluded that the majority of growth for horses is completed by the time they are 2 years old. Additional research evaluating the vertebrae suggest that longitudinal growth of the spine ceases when wither height growth is complete (Butler et al., 1993). Based on these findings, the reviewers suggested that starting horses at the age of 2 is an acceptable practice that aligns with their developmental potential.

But that begs the question whether we should base recommendations on growth plate activity and active bone growth or on growth plate fusion and closure – as these are two very different metrics. This was detailed in a presentation by Collar et al. (2020) in which growth plate activity of lumbosacral vertebrae in Quarter Horses stopped when horses were 2 years old but growth plate closure or fusion was not complete until horses were between 2 and 8 years old.

𝐒𝐨 𝐰𝐡𝐢𝐜𝐡 𝐦𝐞𝐚𝐬𝐮𝐫𝐞𝐦𝐞𝐧𝐭𝐬 𝐬𝐡𝐨𝐮𝐥𝐝 𝐰𝐞 𝐮𝐬𝐞 𝐚𝐧𝐝 𝐰𝐡𝐚𝐭 𝐝𝐨𝐞𝐬 𝐭𝐡𝐞 𝐫𝐞𝐬𝐞𝐚𝐫𝐜𝐡 𝐬𝐚𝐲?

When evaluating race horses, Santschi et al. (2017) found that horses who began training at 2 years of age did not have a higher risk of injury during their racing careers. In fact, they tended to have more successful careers including more lifetime starts, wins, earnings, and years raced.

At first glance, it may seem counterintuitive. But young, growing bodies are built to adapt and specifically, bone development is supported by high cellular activity, an active periosteum, abundant blood supply, and open growth plates. As the body matures, it gradually shifts from a state of building to maintaining. Hormonal changes occur, bones become less adaptable, and osteoblasts (bone-building cells) struggle to keep pace with osteoclasts (cells that break bone down).

In other words - the window for skeletal adaptation is early and we accept this reality in humans all the time.

Young athletes routinely begin training long before their growth plates close. Elite gymnasts, swimmers, and figure skaters often compete internationally as teenagers. Many children enter organized sports as early as five or six years old despite the fact that human growth plates typically remain open until they are 14 to 17.

𝐒𝐨 𝐰𝐡𝐲 𝐝𝐨𝐞𝐬 𝐞𝐚𝐫𝐥𝐲 𝐰𝐨𝐫𝐤 𝐢𝐧 𝐡𝐨𝐫𝐬𝐞𝐬 𝐩𝐫𝐨𝐯𝐨𝐤𝐞 𝐬𝐮𝐜𝐡 𝐬𝐭𝐫𝐨𝐧𝐠 𝐫𝐞𝐬𝐢𝐬𝐭𝐚𝐧𝐜𝐞 𝐰𝐡𝐞𝐧 𝐢𝐭 𝐢𝐬 𝐚𝐜𝐜𝐞𝐩𝐭𝐚𝐛𝐥𝐞 𝐟𝐨𝐫 𝐡𝐮𝐦𝐚𝐧𝐬?

I believe the controversy is not tied to the work itself, but rather the conditions surrounding the work.

Youth athletes are typically offered diversity in the exercise they are allowed to complete, do not have to carry an external load, and can refuse participation or voice concerns. Youth sports are also framed as a crucial part of both physical development and confidence building.

In comparison, young horses are often subjected to repetitive, discipline-specific movement, asked to carry a rider, tend to be confined outside of training, and have no autonomy regarding their participation. Equine sports, specifically those centered around young horses, tend to be tied to economic benefits, tradition, and human timelines that do not always put the horse first.

I believe this is where we have significant room for improvement in the equine industry.

Another consideration is the amount of research we have to provide recommendations. There are a wide variety of breeds and disciplines in the equine industry and the current data is not representative of all demographics. Additionally, for many, performance outcomes aren’t the whole picture. And at the moment, equine research does not extend past a horse’s athletic career, so we may not currently grasp long-term implications of early work.

𝐁𝐮𝐭 𝐭𝐡𝐢𝐬 𝐢𝐬 𝐰𝐡𝐞𝐫𝐞 𝐰𝐞 𝐜𝐚𝐧 𝐮𝐬𝐞 𝐡𝐮𝐦𝐚𝐧 𝐫𝐞𝐬𝐞𝐚𝐫𝐜𝐡 𝐭𝐨 𝐨𝐟𝐟𝐞𝐫 𝐮𝐬 𝐚 𝐮𝐬𝐞𝐟𝐮𝐥 𝐩𝐞𝐫𝐬𝐩𝐞𝐜𝐭𝐢𝐯𝐞.

One of the clearest risks in youth athletics isn’t early movement, it’s repetition without variation.

While sports offer health benefits, single-sport specialization in children has been strongly linked to higher injury rates (Jayanthi et al., 2019). These risks are associated with children performing the same movements repetitively, which puts stress on the same joints and strains the same muscles.

Overuse injuries are especially likely during rapid growth phases, when muscle imbalances and coordination shifts are common (Arnold et al., 2017). This is because active growth is often tied to bone growth that outpaces muscles and tendon development. This imbalance can result in tight muscles, reduced flexibility, and structural instability, which temporarily declines coordination and balance and increases the risk of injury.

Youth athletes also face an increased risk of early-onset osteoarthritis which is linked to high-impact activities, repetitive movements, and severe joint injuries, all of which can accelerate cartilage degeneration (Saxon et al., 1999). However, osteoarthritis wasn’t identified until later in life due to a higher pain tolerance in youth and the time it takes for the condition to develop. I believe a long-term study evaluating this relationship in horses would be extremely insightful.

𝐒𝐨 𝐭𝐡𝐞 𝐢𝐬𝐬𝐮𝐞 𝐢𝐬𝐧’𝐭 𝐬𝐢𝐦𝐩𝐥𝐲 𝐰𝐡𝐞𝐭𝐡𝐞𝐫 𝐲𝐨𝐮𝐧𝐠 𝐛𝐨𝐝𝐢𝐞𝐬 𝐬𝐡𝐨𝐮𝐥𝐝 𝐰𝐨𝐫𝐤 𝐛𝐮𝐭 𝐡𝐨𝐰 𝐭𝐡𝐞𝐲 𝐰𝐨𝐫𝐤.

The key takeaway is that early training is not inherently harmful, rather the structure and approach to that training are what make the difference.

Variety is critical. Cross-training helps distribute stress across tissues and reduces the risk created by repetitive movement patterns. Youth athletes who were highly specialized in a single sport were almost twice as likely to sustain an overuse injury compared to someone competing in multiple sports (Bell et al., 2018). Trail rides, cavaletti work, or practicing a new discipline are all opportunities to not only improve musculoskeletal health but also support a horse’s mental wellbeing.

Short, intentional bouts of higher-intensity loading may stimulate bone adaptation more effectively than long periods of low-intensity exercise – as bone requires a dynamic strain above threshold to elicit bone formation. This was demonstrated by a study evaluating endurance horses completing ‘long, slow’ work, which found that horses in endurance training did not increase bone strength compared to horses allowed to freely exercise on pasture (Spooner et al., 2008).

Meanwhile, sprint exercises have been shown to result in greater bone strength (Logan et al., 2019), increased endosteal circumference (Firth et al., 2012), and greater bone mineral content (Hiney et al., 2004). However, balance is critical. When young horses were sprinted excessively, it had harmful impacts on joint health as the horse was responding to an unnatural amount of work (Van de Lest et al., 2002). While we still need to determine the appropriate level of high-impact work for horses, one study found that just one sprint a week could increase bone strength (Logan et al., 2019).

Load matters, too, and some weight-bearing can be beneficial. Research found that horses carrying 100 lbs while trotting had greater bone mineral deposition of the cannon bone compared to those who did not carry weight (Nielsen et al., 2002). However, it is important to note that the load these horses carried does not reflect most riding situations. In comparison, excessive loads could be detrimental to the horse and rider size is a real consideration when starting young horses.

Movement also builds coordination, balance, and proprioception. Expecting a horse to enter athletic work at maturity without foundational motor skills would be like asking a 22-year-old to learn and compete in a sport like soccer or gymnastics against someone who has trained since childhood. Early exposure to low-intensity technical challenges such as balance, body awareness, and varied terrain, can be incredibly valuable.

𝐀𝐧𝐝 𝐩𝐞𝐫𝐡𝐚𝐩𝐬 𝐦𝐨𝐬𝐭 𝐢𝐦𝐩𝐨𝐫𝐭𝐚𝐧𝐭𝐥𝐲: 𝐥𝐢𝐟𝐞𝐬𝐭𝐲𝐥𝐞 𝐦𝐚𝐭𝐭𝐞𝐫𝐬.

Work is only a small part of a horse’s day.

A two-year-old that is lightly trained but lives in turnout and is allowed to move freely, navigate space, and engage in natural behaviors, is experiencing something very different from one that lives in a stall for the majority of the day.

This is backed by research in which young horses pastured for at least 12 hours a day had greater bone mineralization and cannon bone circumference in comparison to their counterparts who lived in a stall (Bell et al., 2001). Since young horses often live in stalls during sale prep or once they enter training, they may be more likely to have bone loss or an increased risk of injuries. While that stall may be convenient for us, movement outside of structured exercise is critical for musculoskeletal development as well as mental wellbeing.

𝐀𝐫𝐞 𝐰𝐞 𝐚𝐬𝐤𝐢𝐧𝐠 𝐭𝐡𝐞 𝐫𝐢𝐠𝐡𝐭 𝐪𝐮𝐞𝐬𝐭𝐢𝐨𝐧?

Perhaps the real issue isn’t if young horses should work or even what age to start them, but whether the work we ask of them is age-appropriate.

Most horses are still in an active growth phase until around 2 years of age, and during this time, structured work should be limited while free movement through pasture turnout may be the most appropriate and beneficial form of loading.

Once rapid growth begins to slow, workload can be introduced thoughtfully and tailored to the individual, taking into account breed, maturity, and current developmental stage. At this point, how we develop the horse matters far more than simply when we begin.

𝐂𝐨𝐧𝐜𝐥𝐮𝐬𝐢𝐨𝐧

If I had to summarize some recommendations, they would include:

🌱 House your horse in a pasture or paddock over a stall.

🏋️ Cross train to reduce the risk of overuse injuries.

⚖️ Focus on low intensity, technical work at a young age to improve coordination and proprioception.

🏇 Utilize high-intensity work strategically to increase bone strength.

📈 Minimize work during any growth spurts.

🐴 Make decisions for your specific horse based on individual growth and characteristics.

The bottom line is that early work itself isn’t the issue - what really matters is how young horses are trained, managed, and allowed to live.

If you want to read more on this topic, I encourage you to read an open access review (which means it is accessible to everyone!) by Logan and Nielsen (2021) which highlighted a lot of the research I covered in this post. I will include a link in the comments!

There’s always more to unpack, but hopefully this reframes the conversation in a way that allows us to use science to mold our decisions instead of tradition.

Cheers,
Dr. DeBoer

Table 15-2; Getty R(ed): Sisson and Grossman's The Anatomy of the Domestic Animals , ed 5. Philadelphia , WB Saunders Co , 1975, p 272.

Rogers CW, Gee EK, Dittmer KE. Growth and bone development in the horse: when is a horse skeletally mature?. Animals. 2021 Nov 29;11(12):3402.

Butler, J.A., Colles, C.M., Dyson, S., Kold, S., Poulos, P. Clinical Radiology of the Horse. 1993.

Collar, E. M., Russell, D. S., Huber, M. J., Duesterdieck-Zellmer, K. F., & Stover, S. M. (2020). Investigation into lumbosacral vertebral anatomy and growth plate closure in Quarter Horses [Video]. AAEP Proceedings. American Association of Equine Practitioners.

Santschi, E.M.; White, B.J.; Peterson, E.S.; Gotchey, M.H.; Morgan, J.M.; Leibsle, S.R. Forelimb Conformation, Sales Results, and Lifetime Racing Performance of 2-Year-Old Thoroughbred Racing Prospects Sold at Auction. J. Equine Vet. Sci. 2017, 53, 74–80.

Jayanthi NA, Post EG, Laury TC, Fabricant PD. Health consequences of youth sport specialization. Journal of athletic training. 2019 Oct 1;54(10):1040-9.

Arnold A, Thigpen CA, Beattie PF, Kissenberth MJ, Shanley E. Overuse physeal injuries in youth athletes: risk factors, prevention, and treatment strategies. Sports health. 2017 Mar;9(2):139-47.

Saxon L, Finch C, Bass S. Sports participation, sports injuries and osteoarthritis: implications for prevention. Sports medicine. 1999 Aug;28(2):123-35.

Bell DR, Post EG, Biese K, Bay C, Valovich McLeod T. Sport specialization and risk of overuse injuries: a systematic review with meta-analysis. Pediatrics. 2018 Sep 1;142(3):e20180657.

Spooner HS, Nielsen BD, Woodward AD, Rosenstein DS, Harris PA. Endurance training has little impact on mineral content of the third metacarpus in two-year-old Arabian horses. Journal of Equine Veterinary Science. 2008 Jun 1;28(6):359-62.

Logan, A., Nielsen, B., Robison, C., Manfredi, J., Schott, H.; Buskirk, D., Hiney, K. Calves, as a model for juvenile horses, need only one sprint per week to experience increased bone strength. J. Anim. Sci. 2019, 97, 3300–3312.

Firth, E.C., Rogers, C.W., Rene van Weeren, P., Barneveld, A., Wayne McIlwraith, C., Kawcak, C.E., Goodship, A.E., Smith, R.K.W. The Effect of Previous Conditioning Exercise on Diaphyseal and Metaphyseal Bone to Imposition and Withdrawal of Training in Young Thoroughbred Horses. Vet. J. 2012, 192, 34–40.

Hiney, K.M., Nielsen, B.D., Rosenstein, D. Short-Duration Exercise and Confinement Alters Bone Mineral Content and Shape in Weanling Horses. J. Anim. Sci. 2004, 82, 2313–2320.

Van de Lest, C., Brama, P.A.J., René Van Weeren, P. The Influence of Exercise on the Composition of Developing Equine Joints. Biorheology 2002, 39, 183–191.

Bell RA, Nielsen BD, Waite K, Rosenstein D, Orth M. Daily access to pasture turnout prevents loss of mineral in the third metacarpus of Arabian weanlings. Journal of animal science. 2001 May 1;79(5):1142-50.

Nielsen BD, O'Connor CI, Rosenstein DS, Schott HC, Clayton HM. Influence of trotting and supplemental weight on metacarpal bone development. Equine Veterinary Journal. 2002 Sep;34(S34):236-40.

02/24/2026

🧠💪🏽 Laterality isn’t just a rider’s “feel” 👉 it’s measurable, it’s biomechanical, and it runs deep.

📊 Research by Krüger et al., 2022 (Laterality in Horse Training: Psychological and Physical Asymmetry in Horses, Animals, 12(8):1017) and Kuhnke et al., 2022 (A Comparison of Different Established and Novel Methods to Assess Laterality in Horses, Front. Vet. Sci., 9:789260) found that:

🔎 Rein tension is rarely equal. One rein almost always shows higher tension - and that isn’t a rider error. It reflects a fundamental left–right asymmetry in the horse’s neuromuscular system. What you feel in your hands is a direct representation of how the horse organises movement through the spine and limbs.

🔎 Hindquarter displacement is common. Even in apparently “straight” horses, the pelvis and hindquarters often drift a few centimetres off the midline. That subtle shift alters how force travels through the body, changes back muscle activation, and can lead to uneven loading in the forelimbs.

🔎 Rider asymmetry compounds the problem. The research also found that when a horse’s laterality matches the rider’s dominant side, rein tension and movement patterns become more balanced. But when they don’t, asymmetries often worsen - amplifying drift, poll bend, and uneven contact.

📏 What does this mean for us as vetrehabbers?
Laterality is not something to “fix”. It’s a pattern rooted in the horse’s brain, musculoskeletal system, and history. Recognising it allows us to:
✅ Design targeted groundwork to balance hindquarter control.
✅ Support more symmetrical development in the trunk and thoracolumbar fascia.
✅ Coach riders on how their seat and rein habits influence what happens beneath them.

🐴 Straightness isn’t about straight lines - it’s about balanced neuromuscular control. And the evidence shows that paying attention to these details can make the difference between subtle compensations and sustainable, symmetrical performance.

💡 How do we start to differentiate between asymmetry and laterality 👇

👨‍🏫 This months webinar with João Paulo Marques: DVM, MRCVS, IVCA, IVAS, CERP starts to unravel this question.
🟢 Equine Asymmetry and Laterality: Part 1
During this webinar, we will explore morphological and functional asymmetry and laterality in horses, reviewing key findings from the literature. The webinar will provide insights into how these factors influence equine performance and movement, laying the foundation for clinical applications in rehabilitation and training.

📢 Comment EQU if you would like the link to register for this webinar

02/24/2026

🚨 Steroids, Insulin, and Lamellar Structural Failure: What New Research Means for Our Horses 🚨
As many of you know, at our clinic we frequently discuss the risks associated with corticosteroid use—especially in horses with underlying metabolic risk. A large number of our lamellar structural failure cases each year are steroid-associated.
In many of these horses, the metabolic dysfunction was likely subclinical… until the steroid pushed them over the edge.
📚 Recent Research Adds Important Insight
A recent prospective, controlled crossover study evaluated the effect of the SGLT2 inhibitor ertugliflozin on insulin response after intra-articular corticosteroid administration.
Study Summary
8 metabolically normal geldings
Received intra-articular triamcinolone (18 mg)
Compared no treatment vs. 7 days of ertugliflozin before and after injection
Measured resting glucose, resting insulin, and oral sugar test (OST) responses
Key Findings:
✔ Insulin significantly lower 2 days after steroid injection with SGLT2 inhibitor
✔ Resting glucose significantly lower at 8–48 hours
✔ Resting insulin significantly lower at 12–72 hours
✔ Suggests reduced hyperinsulinemic response after steroid administration
Even in metabolically normal horses, insulin and glucose responses were blunted with SGLT2 inhibition.
The authors concluded that further investigation in insulin dysregulated horses is warranted — particularly regarding laminitis risk reduction.
🧬 Why This Matters Clinically
⚠ Hyperinsulinemia = Lamellar Risk
We now understand that hyperinsulinemia alone can induce lamellar pathology, even in the absence of systemic inflammation. Steroids can:
Increase insulin concentrations
Worsen underlying insulin dysregulation
Trigger lamellar structural failure
In horses with:
Equine metabolic syndrome
Regional adiposity (cresty neck, fat pads)
Obesity
Previous laminitis
Subclinical insulin dysregulation
…the addition of corticosteroids may be enough to initiate failure at the lamellar interface.
🐴 Equine Metabolic Syndrome (EMS)
Equine Metabolic Syndrome is characterized by:
Insulin dysregulation
Regional adiposity
Increased laminitis risk
Many performance horses today are:
Easy keepers
Over-conditioned
Fed high NSC diets
Exercised inconsistently
And importantly — some appear outwardly normal but have abnormal insulin dynamics.
💉 Steroids and the “Metabolic Push”
We use corticosteroids for:
Joint inflammation
Soft tissue injury
Respiratory disease
Allergic conditions
But steroids:
Increase insulin concentrations
Reduce peripheral glucose utilization
Can unmask latent metabolic dysfunction
In our practice, many steroid-associated lamellar structural failure cases are not classic “Cushing’s horses.”
They are subclinical metabolic horses that decompensate after steroid exposure.
💊 Where SGLT2 Inhibitors Fit
SGLT2 inhibitors:
Promote urinary glucose excretion
Reduce circulating glucose
Lower insulin concentrations
Improve insulin dynamics
Over the past few years, we have recommended SGLT2 inhibitors in suspect metabolic cases, particularly:
Prior to or surrounding steroid use
In known insulin dysregulated horses
In high-risk laminitis patients
This new research supports that strategy — showing measurable reduction in insulin and glucose changes even in metabolically normal horses.
🔎 Practical Take-Home Points
✔ Not all metabolic horses look obviously metabolic
✔ Steroids can induce hyperinsulinemia
✔ Hyperinsulinemia drives lamellar structural failure
✔ SGLT2 inhibitors may reduce steroid-associated insulin spikes
✔ Screening (resting insulin + oral sugar testing) matters
🧠 Our Philosophy
We are not anti-steroid.
We are anti-uninformed risk.
Every decision should be:
Mechanically informed
Metabolically informed
Individualized
If your horse requires corticosteroids, especially if:
Overweight
Previously laminitic
Cresty
Has regional adiposity
…let’s discuss metabolic screening and risk mitigation strategies.
Because when it comes to lamellar structural failure, prevention is always easier than rehabilitation.
— Dr. Sammy L. Pittman
Innovative Equine Podiatry & Veterinary Services
Helping Your Help Horses — From the Ground Up 🐴

12/24/2025

* Winter Laminitis *
As winter weather sets in, closely monitor your horses with metabolic issues or a past history of laminitis or founder. They could develop winter laminitis, which can appear suddenly, without typical warning signs.

While spring and fall laminitis are typically linked to insulin dysregulation, winter laminitis has a different trigger: cold stress when temperatures drop, which leads to limited perfusion throughout the limb.

Arteriovenous (AV) shunts are special blood vessels in the horse’s hoof that act like shortcuts for blood flow. Instead of moving through tiny capillaries that feed the sensitive laminae, blood can be redirected straight from arteries to veins. In a healthy hoof, these shunts help control temperature by opening in cold weather to conserve heat, and closing in warm weather so blood flows through the capillaries to deliver oxygen and nutrients to the laminae.

During a laminitis or founder episode, inflammation within the laminar tissues weakens the cellular bonds between the epidermal and dermal laminae, causing the white line to stretch. Their blood flow is restricted, which can lead to permanent damage of the hoof lamellar connection - the laminae rarely returns to its original strength. In founder cases, the laminae failure allows the coffin bone to rotate, and/or distally descend in the hoof capsule, also known as sinking, and can sometimes lead to the coffin bone penetrating the sole.

Even well-managed horses with a history of laminitis may have thin soles, reduced shock absorption, weaker hoof-lamellar connections, and ongoing low-grade inflammation, making them less tolerant of trimming errors, long cycles, diet or environmental changes, and metabolic stress. A horse can appear sound yet still have permanently compromised laminae, which increases the risk of relapse and often requires lifelong management, especially when a metabolic disease is involved.

If your horse is actively experiencing winter laminitis:

* Contact and consult with your veterinarian immediately.

* Ensure proper pergolide dosing with your vet’s help (if PPID/Cushings).

*Prioritize warmth: use blankets, layering if necessary. Wool socks, shipping boots, or leg wraps can all help provide extra warmth and protection - just make sure they’re not fitted too tight! Make sure your horse has adequate shelter to support temperature regulation.

* Reduce mechanical leverage and stress on the hoof capsule by staying on a short, consistent cycle with your hoof care provider. Providing hoof support with boots and pads can help keep your horse comfortable.

* Monitor weight and watch for fat deposits (commonly seen around the neck, ribs, tailhead, above the eyes).

* Re-evaluate diet and aim for consistent tested hay year-round, feeding a truly low-NSC (non-structural carbohydrate) diet. Don’t rely solely on “low sugar” or “low starch” labels - use evidence-based resources like the ECIR Group or Mad Barn for guidance. Consider metabolic support supplements if appropriate.

Wishing everyone - and their equines - warmth and good health this winter!

12/01/2025
11/25/2025

Two recent papers in the EVE Journal were punlished on the effects of corticosteroid injection sin joints. They were meta-analysis, meaning they took information from over 600 papers about this topic into account. This means the outcome of a meta-analysis is more reliable as an "overall opinion of scientists" than a single paper. Both papers show clearly that 1 injection in a joint (when needed, because acute joint inflammation is present) might be beneficial, but that more than 1 injection in the same joint will cause cartilage damage and degeneration of the joint. Think about that when you do "regular joint maintainance" or inject multiple joints without a clear indication.

Your vet should know this information!

There has been confirmed exposure to EHV-1/EHM to horses that attended the WPRA Finals in Waco, Texas. Also, case confir...
11/19/2025

There has been confirmed exposure to EHV-1/EHM to horses that attended the WPRA Finals in Waco, Texas. Also, case confirmed in Oklahoma. If you have attended an event where there have been exposed horses, it is of extreme importance to quarantine your horse for a minimum of 14 days. This is a good time to keep your horses home entirely! If you have recently travelled, your farrier or trimmer would very much appreciate you sharing this information prior to appointment, so appropriate biosecurity measures can be taken or appointment can be rescheduled if considered high-risk.

EHV spreads through nose to nose contact, shared buckets or tack, contaminated trailers, airborne droplets from coughing or snorting, and from people who handle exposed horses.

Symptoms
- Fever
- Nasal discharge
- Lethargy
- Hind end weakness or wobbliness
- Loss of tail tone or urine dribbling

Many horses will not show symptoms!

Some cases progress to the neurological form, EHM, which is a medical emergency. Take temperatures twice daily and monitor closely. Practice strict biosecurity protocols; do not share tack/equipment/water sources, disinfect high contact areas with bleach and most importantly do not move horses on or off the property!

AAEP Resources
https://aaep.org/resource/aaep-infectious-disease-guidelines-equine-herpesvirus-1-4/e y

Equine herpesviruses are very common DNA viruses in horse populations worldwide. The two most significant are EHV-1, which causes respiratory disease, abortion, and neurologic disease; and EHV-4, which primarily causes respiratory disease and only occasionally causes abortion or neurologic disease.....

10/23/2025

There is a very dangerous and misleading statement being made on the internet.

Quote
“Laminitis is not caused by diet”

I urge you to use caution if you choose to believe this.

I am a student of the hoof for the past 10 years and I am mentored by Prof Chris Pollitt, the pioneer who discovered the insulin relationship to laminitis. I beg you to be very careful with what you hear out there being touted by lay people.

Horses will die unnecessary painful excruciating deaths if you follow this misinformation.

By ignoring the well understood relationship between high insulin and laminitis you may be inclined to turn out your ponies onto the rich grass. Apparently it’s said that a balanced trim is the key- which I do not totally agree with.

Once the genie is out of the bottle and the laminae are failing due to high insulin stretching and snapping the laminar attachments of bone to inner hoof wall then good luck getting it back.

Is it worth the risk?

Have you seen laminitis appear in the spring when the grass starts to grow. Or after a long hot summer, when the rains start, and boom, laminitis rears its head. Why is that?

Why do many horses suffer laminitis after getting into the grain shed and gourged themselves? Is it the grain or the trim?

Trimming is important, but you cannot trim your way into preventing or treating laminitis without looking far deeper into the cause. Diet and insulin go hand in hand.

Do you think that these lay people that come up with such crazy and dangerous statements that are said as if they are fact have actually been in the lab and done any research?

These are frightening times my friends.

Anyone can say anything and mislead us.

Maybe it is intentional, it just feeds the algorithm and everyone comments and argues and shouts and the ones dropping bombshell dangerous statements just rub their hands in glee at the carnage.

It’s sick.
It’s dangerous
I follow the science.

This is my position statement.

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