09/25/2025
Instead of fighting about autism and turning every new study into a political debate, let’s focus on what really matters — doing what’s best for our kids.
Autism is a complex condition. Genetics play only a small role, while most cases are shaped by a “perfect storm” of environmental triggers that lead to neuroinflammation and altered brain development.
The recent headlines about acetaminophen (Tylenol) and autism aren’t as shocking as they sound. Research linking prenatal acetaminophen use to higher autism risk was published over six years ago. This does not mean that Tylenol is the sole cause of autism, or that taking it during pregnancy guarantees an autism diagnosis. It is simply one of many contributors that can add up, especially when combined with:
- Genetic variants like MTHFR or in other detox pathways
- Maternal toxic burden (chemicals, heavy metals)
- Overuse of antibiotics → candida overgrowth
- High vaccine exposure with aluminum/mercury adjuvants
One proposed mechanism is key: acetaminophen depletes glutathione, the body’s “master antioxidant” and primary detoxifier. When glutathione is low — and children are already born with high toxic loads — fat-soluble toxins can build up in the brain, interfering with healthy neurodevelopment. See future post about all the other proposed mechanisms.
This is not about fear or blame. It’s about being informed and proactive. We owe it to our kids to reduce risk factors where we can, create healthier environments, and work together — not fight — to give them the best chance to thrive.
