10/08/2025
šÆ Treating Gravesā Disease Doesnāt Have to Feel Hopeless
In fact⦠it can even be kind of exciting. š
Gravesā disease often responds surprisingly well to treatment, especially when using Low Dose Immunotherapy (LDI).
Many patients are able to stabilize without staying permanently on medications like methimazole or PTU. š§¬
Hereās what weāve seen:
š¦ In about 75ā80% of cases, the trigger appears to be Yersinia ā a bacterial antigen that may drive the autoimmune process behind Gravesā.
š” These days, we donāt just use Yersinia alone. We use a broader GI bacterial mix that includes Yersinia plus dozens of other gut microbes. Itās hard to say which specific bug is the key ā but the immune system often calms down once that mix is introduced via LDI.
š« Forget stool testing.
You probably wonāt find Yersinia or the right culprit on a lab culture, and it doesnāt matter.
With autoimmunity, the immune systemās memory is the problem, not the current presence of the organism.
š Tracking Progress:
Whether you have Hashimotoās or Gravesā, hereās the gold standard for monitoring your response to LDI:
ā
10ā14 days after your dose, check your antibody levels:
* Hashimotoās: Track TPO antibodies
* Gravesā: Track Thyroid Stimulating Immunoglobulin (TSI) or TRAb
Skip the thyroglobulin antibody unless you're monitoring general inflammation. It's often non-specific and influenced by viral illness or pregnancy.
š Curious about how LDI can help regulate your thyroid autoimmunity?
Drop a š¬ below or DM me to learn more.