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Interesting info related to laminitis…
09/10/2024

Interesting info related to laminitis…

13/09/2024
10/09/2024
02/08/2024

A laminitis turnaround at near blinding speed--- only 8 weeks between these two photos as the damage grows out. (5 months post-onset, with medication). However, look at Magical Before & After pictures like this with a grain of salt before asking "Well why can't MY horse/pony/donkey do this?"

No laminitis cases are ever really "identical" (despite close similiarities) and such speed can be affected by many factors: extent of damage, previous episodes/chronic damage, ongoing management/ response to medication, horse's ability to grow good hoof genetically, any further minor episodes, farrier intervention, and pure luck.

A younger horse with no prior episodes and naturally good-quality hooves will usually recover much faster than an aged horse who's had many episodes over their lifetime. Each time damage happens, the recovery rate becomes slower and returning to "normal" becomes more difficult, usually due to lower numbers of healthy laminae attachments, any acquired flexural limb contracture, and permanent coffin bone damage.

Very severe damage, or poor physical responses to medications, or lack of effort in management (diet control, weight control, farrier visits, feeding the prescribed medications), all impact the timeline and overall quality of recovery. Horses with PPID, metabolic syndromes or insulin dysregulation typically need lifelong management practices or medications--- whereas others may have a one-off episode from eating a w**d or something and be fine the rest of their lives.

Sometimes the management is not simple and often it can be expensive. Some horses require much more effort than others and amount of effort may progress year to year. Sometimes they hit a point where there's no fixing it, or the sheer amount of effort becomes too much, or they have many other health and soundness issues. "Don't Be A Hero" unfortunately happens.

Nowadays many horses do make good recoveries with a solid Team Effort. If you feel you or your horse is struggling or has hit a wall, regressed, check in with your team (Vet, Farrier) and re-assess.

General Laminitis Management ideas:

1) A) Find the trigger! History of obesity, fresh grass, cold nights, drought hay, Cushings/PPID, Potomac Horse Fever, reaction to steroids or innoculations, high starch grain?

1) B) Metabolic Testing. This is handy for diagnosis, baselines and getting the correct medications if necessary.

2) Assess Horse's Weight. If you're unsure, ask the internet, they ain't holding back on low-glucose opinions.

3) Daily Diet Corrections. Paddock Paradise or track system building can be quite simple; slow-feed haynets, changing hay location/spread out further around the paddock, or feeding style, and reducing grass intake. Low-starch, high-fibre diets.

4) Check Magnesium intake. Some low-grade laminitis and chubby animals respond very well to magnesium, especially if deficient or in work.

5) Increase Exercise if Chubby (if possible! Do not forcibly exercise a lame horse!). If your horse is parked in a small paddock eating 24/7 off free-choice hay, their hooves just will not stop flaring and they have ripples in their skin from hard fat deposits on the neck and ribcage, are constantly itching their tail, or they are having acute laminitis flares frequently, then they are likely eating more calories than they are burning and you will have to either decrease their caloric intake per day OR start aerobic exercise program daily to increase fat metabolism and balance the calories.

Even 15-30 minutes of handwalking actively (no breaks, at a decent pace) per day will make a difference in a few weeks.

If you cannot exercise the horse by yourself or because they are too sore or have arthritis, etc, then you will have to bite that bullet and *stop* overfeeding them. Period. There are many ways to do this that will not cause ulcers! Hay stations, slowfeeders with smaller hole sizes, multiple feedings of a specific amount per day.

If he's too thin, feed more. If he's too fat, feed less.

If he won't gain weight or he won't lose weight, call your vet!

https://www.facebook.com/share/p/EFtTWEUChRpw5Rv6/?mibextid=WC7FNe
23/07/2024

https://www.facebook.com/share/p/EFtTWEUChRpw5Rv6/?mibextid=WC7FNe

I went for a little walk around the local show park yesterday during the season opener hunter/jumper show. I try not to look down at horse feet unless someone asks for my opinion, but despite my best efforts, I couldn’t stop noticing that most of the horses there, from the low level hunters to the 1.20m jumper class I watched for a while are very obviously NPA and/or showed obvious signs of caudal failure. NPA means “negative palmar angle” on front feet or “negative plantar angle” on hinds. It means that the back of the coffin bone is lower than the front. It is supposed to be the other way around! A normal palmar/plantar angle is 2°-10° yet soooo many horses work on feet with palmar/plantar angles of less than zero. It is so common that by most people it is seen as normal. Caudal failure means structural collapse of the caudal (back) part of the foot.

Horses may not be obviously lame with this condition, however there are often subtle signs. Reluctance to go forward, forging (stepping on or hitting the backs of the front shoes with the hind feet), overreaching, not tracking up, refusing jumps, bucking after jumps (because landing hurts), lack of hindquarter engagement, decreased gait quality all around, behavioural issues under saddle, etc. These symptoms can be easily mistaken for other things or riders and trainers can tend to use punishment to try to change some behaviours that have their root in hoof pain. It is also very hard on the legs and most specifically the DDFT (deep digital flexor tendon) and navicular area of the foot because of the biomechanics of a foot with an improper angle cause increased friction where the DDFT runs under the navicular bone to attach to the back of the coffin bone. This is why low heeled horses are at increased risk of developing navicular syndrome.

We need to retrain our eyes to know what is normal. We also need to realize that asking horses to work hard when their feet are a mess is not fair and causes sometimes irreparable damage, both to their feet and to their opinions about working.

How do we fix it? First we need to acknowledge that NPA is a systemic issue in farriery and we need to change the way we trim and shoe. Owners need to recognize NPA and find a farrier who recognizes NPA and knows how to fix it. Farriers need to stop trimming off the back of the foot and add frog support to our shoeing packages. Frog support needs to be normalized. More than one client has remarked to me that if we put frog pads on the horse, potential buyers will think there is something wrong with the horse. This is because what is normalized currently in farriery are open heeled regular metal shoes, which are a huge part of what causes NPA. When we lift up the foot off the ground and provide no structural support to the frog, the center of the foot collapses. This is a very simple concept, yet we are still doing things the same way we have always done and expecting a different result. This is the definition of insanity and it is killing our horses slowly. If I help to change only one thing in my time as a farrier, I hope for it to be this.

For reference, normal/ideal angles are generally as follows:
Hairline: about 20°
Dorsal wall (toe): about 50-55°, steeper on some breeds
Heel: equal to toe angle or perhaps 5° less than dorsal wall

This horse’s toe is close to 50° however the heel is 23° lower than the toe and I could not even measure the heel at the back because the bulb is sitting on the shoe! I had to measure it where I could see the angle of the tubules on the wall. The hairline is far too low. This foot is not helping this horse at all and this horse is one of many. I might get some angry messages for this, but we need to acknowledge that most of our performance horses are NPA and/or have some degree of caudal failure and then then we need to do something about it.

——-

Addendum: This post has pi**ed off some people. Fair enough. It’s also been shared all over the world, which has been great and also quite the experience. Wayne over at Progressive Equine has been writing about caudal failure and NPA for a few years now, as have others. This topic is not new. For whatever reason, this post in particular has caught peoples’ attention.

What I ask of those who are angry about it, or indignant that I dared to challenge the status quo is this: if you think I’m wrong, ok. Go prove it. If I’m wrong, why are you angry? If I am wrong, then this post is irrelevant to you and irrelevant to your work and there is nothing to be angry about. I did not (and will not) name any particular farrier. Who did the job is not even relevant, because this problem is not about one person. I am not trash talking anyone. I am saying that there is a systemic issue in how farriers are taught to trim and shoe and that it is on us, as a group, to correct it by continuing to learn and grow. That is my perspective. If you think I’m wrong then disregard it. And yes, some horses manage in regular shoes and to the owners and farriers of those horses I am glad for you. For everyone else, maybe what I wrote here will help you. That is all I am trying to do.

14/07/2024

The problem with softening the horse too much in the bridle before stabilizing the hind end is not only a biomechanics problem but a safety problem.

I just got done riding a new horse for the first time. Based on the weak hind end and bulging in the neck in the wrong spots, I had a good feeling he would break behind the vertical quite frequently.

When the horse breaks behind the vertical, it drops the back and negates correct use of the hind end. Over time, the horse becomes weak and wobbly while simultaneously developing painful arthritis and fusions.

So, that's the basics of the biomechanics problem.

The safety aspect is that a horse that is too light in the bridle and breaks behind upon first contact does not have the correct relationship with the contact. I believe the reins should connect to the feet and aid in correct flexion and bend.

When the horse evades the bit, you've got nothing. Sorry pal- this ain't a safe place to be.

This is why often horses that are taught this require a lot of holding together by riders that are used to riding in such a way.

And if you want to improve the horses relationship with contact, then be prepared for a long haul of conditioning and riding with little rein.

It can be done, but it should be done with knowledgeable riding with no time constraints. It's a whole thang.

Edit to add:
This photo is NOT mine. I'm grateful it was created and my favorite part is the "ah thank you!" Because of how freely forward the horse is 🙂
https://www.instagram.com/anjakyart?igsh=MXNnN21uZm13cDRlYQ==

26/06/2024
23/05/2024
01/04/2024
01/04/2024

All 3 of these bones are the same vertabrae…….

These are all C6 cervical vertabrae from the equine neck, the first is from a standardbred(bi-lateral), the second along is from a Swedish warmblood(uni-lateral) and the third is from an Appaloosa(normal). The first two are displaying ECVM, if you don’t know what this I have attached an article below from thirza Hendriks that is well balanced and concise. If that peaks your interest, which it should because this affects a sizeable number of our equines then there are full studies online.

https://www.thirzahendriks.com/post/the-un-balanced-horse

This is something all equestrians should know about. The C6 malformation is only a small part of what the problems can be. This can affect cervical vertabrae C6-7, ribs, sternum, muscles, nerves and soft tissue.

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