Lexi Yoo, FNP, IFMCP, 9535 E 151th Street, Noblesville, IN (2026)

02/21/2026

𝗛𝗶𝘀𝘁𝗮𝗺𝗶𝗻𝗲 𝗶𝘀 𝗿𝗮𝗿𝗲𝗹𝘆 𝗽𝗮𝗿𝘁 𝗼𝗳 𝘁𝗵𝗲 𝗵𝗼𝗿𝗺𝗼𝗻𝗲 𝗰𝗼𝗻𝘃𝗲𝗿𝘀𝗮𝘁𝗶𝗼𝗻 𝗯𝘂𝘁 𝗰𝗹𝗶𝗻𝗶𝗰𝗮𝗹𝗹𝘆, 𝗶𝘁 𝘀𝗵𝗼𝘂𝗹𝗱 𝗯𝗲. 🧠

👉🏼Histamine is an excitatory neurotransmitter. It increases alertness, drives sympathetic activation, and can worsen anxiety and insomnia. In women with histamine intolerance or mast cell activation, baseline histamine levels are already high.

👑When progesterone is introduced into that environment, it doesn’t always act as a calming hormone. Instead, it can trigger histamine release or fail to activate its usual GABA-mediated calming effects.

That’s why some women feel:
🔥 More anxious
🌙 More wired at night
💓 Palpitations
😣 Mood instability

after starting progesterone even at low doses.

This isn’t a willpower issue. And it’s not something you should “push through.”

𝗛𝗼𝗿𝗺𝗼𝗻𝗲𝘀 𝗱𝗼𝗻’𝘁 𝘄𝗼𝗿𝗸 𝗶𝗻 𝗶𝘀𝗼𝗹𝗮𝘁𝗶𝗼𝗻. 𝗧𝗵𝗲𝘆 𝘄𝗼𝗿𝗸 𝗶𝗻𝘀𝗶𝗱𝗲 𝗻𝗲𝗿𝘃𝗼𝘂𝘀 𝘀𝘆𝘀𝘁𝗲𝗺𝘀, 𝗶𝗺𝗺𝘂𝗻𝗲 𝘀𝘆𝘀𝘁𝗲𝗺𝘀, 𝗮𝗻𝗱 𝗺𝗲𝘁𝗮𝗯𝗼𝗹𝗶𝗰 𝘀𝘆𝘀𝘁𝗲𝗺𝘀. 𝗜𝗳 𝗵𝗶𝘀𝘁𝗮𝗺𝗶𝗻𝗲 𝗶𝘀 𝗱𝗿𝗶𝘃𝗶𝗻𝗴 𝘁𝗵𝗲 𝗿𝗲𝘀𝗽𝗼𝗻𝘀𝗲, 𝘁𝗵𝗲 𝘀𝘁𝗿𝗮𝘁𝗲𝗴𝘆 𝗵𝗮𝘀 𝘁𝗼 𝗰𝗵𝗮𝗻𝗴𝗲.

This is why symptom patterns matter just as much as labs and why one-size hormone advice fails so many women.

➡️ Follow for continued clinical hormone education
💾 Save or share if this helped connect the dots
🧘🏻‍♀️ Have you ever noticed progesterone worsened anxiety or sleep?

02/21/2026

👉🏼If you’ve been struggling with menopause belly that just won’t budge this isn’t about effort. 🧠

Midlife changes fat distribution. And it’s driven by physiology.

Here’s what shifts:

🧬 Estradiol declines → fat redistributes toward the abdomen
📉 Muscle mass decreases → metabolic rate slows
🫀 Insulin sensitivity drops → more fat stored centrally
🔥 Visceral fat increases → more inflammatory signaling
😴 Sleep disruption + cortisol changes → worsened abdominal storage

❤️Visceral adipose tissue is metabolically active. It’s not just cosmetic it’s linked to cardiometabolic risk, insulin resistance, and long-term inflammation.

More cardio won’t fix hormonal signaling.
Undereating makes it worse.

👉🏼The strategy becomes:
• Progressive resistance training
• Adequate protein
• Sleep protection
• Stress regulation
• In some cases, hormone optimization or targeted metabolic support

You are not failing your body.
Your body is transitioning.

📌Follow for evidence-based menopause education.
📌Save this so we can discuss at your next appointment

02/21/2026

𝗙𝗮𝘀𝘁𝗶𝗻𝗴 𝗱𝗼𝗲𝘀𝗻’𝘁 𝗮𝘂𝘁𝗼𝗺𝗮𝘁𝗶𝗰𝗮𝗹𝗹𝘆 𝘁𝘂𝗿𝗻 𝗼𝗻 𝗮𝘂𝘁𝗼𝗽𝗵𝗮𝗴𝘆.

There’s a missing piece most people don’t talk about: spermidine.

In pre-clinical models, when spermidine was absent, even calorie restriction didn’t trigger autophagy. The cells simply didn’t activate that internal cleanup process.

That’s a big deal.

Spermidine isn’t just an enhancer of autophagy. It appears to be required.

📌Here’s why: spermidine inhibits something called EP300, which normally blocks autophagy. When EP300 is active, cellular cleanup stays off. Spermidine helps remove that brake.

𝗪𝗲 𝘂𝘀𝘂𝗮𝗹𝗹𝘆 𝗴𝗲𝘁 𝘀𝗽𝗲𝗿𝗺𝗶𝗱𝗶𝗻𝗲 𝗳𝗿𝗼𝗺 𝗳𝗼𝗼𝗱. 𝗕𝘂𝘁 𝗶𝗳 𝘆𝗼𝘂𝗿 𝗱𝗶𝗲𝘁 𝗶𝘀 𝗹𝗼𝘄 𝗶𝗻 𝗶𝘁, 𝘆𝗼𝘂’𝗿𝗲 𝗰𝗼𝗻𝘀𝘁𝗮𝗻𝘁𝗹𝘆 𝗲𝗮𝘁𝗶𝗻𝗴, 𝗼𝗿 𝘆𝗼𝘂’𝗿𝗲 𝘂𝗻𝗱𝗲𝗿 𝗰𝗵𝗿𝗼𝗻𝗶𝗰 𝘀𝘁𝗿𝗲𝘀𝘀, 𝗹𝗲𝘃𝗲𝗹𝘀 𝗺𝗮𝘆 𝗻𝗼𝘁 𝗯𝗲 𝗼𝗽𝘁𝗶𝗺𝗮𝗹.

🗝️The key insight: fasting alone may not be enough.

➡️Autophagy depends on more than just cutting calories. Your body needs the right internal signals and spermidine plays a central role in that process.

Longevity isn’t just about eating less. It’s about supporting the biology that makes fasting work.

Follow @‌lexiyoonp for more education.

02/20/2026

💉GLP-1 medications are powerful tools for cardiometabolic health. They reduce inflammation, improve insulin sensitivity, and support meaningful weight loss.

But rapid fat loss changes facial structure.

When volume decreases quickly especially in perimenopause you’re not just losing fat. You’re losing collagen!

You’re seeing:

🧬 Declining estrogen
💪 Reduced muscle mass
🧵 Lower collagen production
📉 Thinner dermal support

➡️The result? Skin that looks less supported. Jawlines that soften. Cheeks that flatten.

This isn’t “vanity.” It’s physiology.

🪄Sculptra works differently than traditional fillers. It doesn’t just replace volume it stimulates your own collagen production over time. That matters in midlife women whose collagen synthesis is already declining.

➡️By triggering fibroblast activity, Sculptra helps rebuild structural support gradually and naturally. It addresses the underlying collagen deficit rather than masking it.

Used strategically, especially alongside resistance training, adequate protein intake, and hormone optimization, it becomes part of a longevity-based aesthetic plan not a quick fix.

🫶🏻Weight loss should improve metabolic health without accelerating facial aging.

If you’re using a GLP-1, this conversation matters.

📌Save this so you understand your options.
❤️Follow for evidence-based perimenopause and longevity education

Have you tried Sculptra? Did it help?

02/20/2026

𝗣𝗿𝗼𝗴𝗲𝘀𝘁𝗲𝗿𝗼𝗻𝗲 𝗶𝘀 𝗼𝗳𝘁𝗲𝗻 𝗳𝗿𝗮𝗺𝗲𝗱 𝗮𝘀 𝘁𝗵𝗲 𝗰𝗮𝗹𝗺𝗶𝗻𝗴 𝗵𝗼𝗿𝗺𝗼𝗻𝗲 𝗮𝗻𝗱 𝗳𝗼𝗿 𝗺𝗮𝗻𝘆 𝘄𝗼𝗺𝗲𝗻, 𝗶𝘁 𝗶𝘀.

But when it causes anxiety, agitation, low mood, or insomnia, that experience deserves to be taken seriously.

As a functional medicine NP, I don’t see this as rare and I don’t see it as a failure. 🧠

Progesterone is a neuroactive hormone. Its effects depend on how it’s metabolized, how receptors respond, and how regulated the nervous system is, not just the milligrams prescribed.

Clinically, progesterone intolerance is often linked to:
🧬 Rapid conversion to stimulating metabolites
🧠 Nervous system hypersensitivity
🔥 Histamine or mast cell activation
🌀 Cortisol or thyroid dysregulation
⚖️ Estrogen dominance without receptor balance

This is why simply “lowering the dose” doesn’t always solve the problem. Sometimes the issue isn’t progesterone at all it’s the context it’s being introduced into.

Hormone care isn’t about chasing normal labs.
It’s about matching therapy to physiology, protecting sleep, mood, and long-term brain health.

𝗜𝗳 𝘁𝗵𝗶𝘀 𝗿𝗲𝘀𝗼𝗻𝗮𝘁𝗲𝗱, 𝘆𝗼𝘂’𝗿𝗲 𝗻𝗼𝘁 𝗶𝗺𝗮𝗴𝗶𝗻𝗶𝗻𝗴 𝗶𝘁 𝗮𝗻𝗱 𝘆𝗼𝘂’𝗿𝗲 𝗻𝗼𝘁 𝗮𝗹𝗼𝗻𝗲.

➡️ Follow for continued clinical hormone education
💾 Save or share if this helped connect the dots
🧘🏻‍♀️ Did progesterone ever make you feel worse instead of better?

02/20/2026

𝗜𝗻 𝘁𝗵𝗲 𝗹𝗼𝗻𝗴𝗲𝘃𝗶𝘁𝘆 𝘄𝗼𝗿𝗹𝗱, 𝘄𝗲 𝗰𝗮𝗹𝗹 𝗺𝘂𝘀𝗰𝗹𝗲 𝘁𝗵𝗲 “𝗼𝗿𝗴𝗮𝗻 𝗼𝗳 𝗹𝗼𝗻𝗴𝗲𝘃𝗶𝘁𝘆” 𝗳𝗼𝗿 𝗮 𝗿𝗲𝗮𝘀𝗼𝗻.

💪🏻More muscle means better blood sugar control and improved insulin use. That supports steady energy, healthier metabolism, and better long-term health.

🦴Muscle also protects your bones. As we age especially women bones become more fragile. Higher muscle mass lowers the risk of falls, fractures, and osteoporosis. Strong muscles help keep you stable and resilient.

🛑 It doesn’t stop there.

𝗠𝘂𝘀𝗰𝗹𝗲 𝘀𝘂𝗽𝗽𝗼𝗿𝘁𝘀 𝗵𝗲𝗮𝗹𝘁𝗵𝘆 𝗯𝗹𝗼𝗼𝗱 𝗳𝗹𝗼𝘄 𝗮𝗻𝗱 𝗯𝗹𝗼𝗼𝗱 𝗽𝗿𝗲𝘀𝘀𝘂𝗿𝗲. 𝗜𝘁’𝘀 𝗮𝗹𝘀𝗼 𝗹𝗶𝗻𝗸𝗲𝗱 𝘁𝗼 𝗯𝗿𝗮𝗶𝗻 𝗵𝗲𝗮𝗹𝘁𝗵. 𝗪𝗮𝗻𝘁 𝘁𝗼 𝗸𝗲𝗲𝗽 𝘆𝗼𝘂𝗿 𝗺𝗶𝗻𝗱 𝘀𝗵𝗮𝗿𝗽? 𝗟𝗶𝗳𝘁 𝘄𝗲𝗶𝗴𝗵𝘁𝘀. 𝗖𝗮𝗿𝗿𝘆 𝗸𝗲𝘁𝘁𝗹𝗲𝗯𝗲𝗹𝗹𝘀. 𝗣𝗶𝗰𝗸 𝘂𝗽 𝗵𝗲𝗮𝘃𝘆 𝘁𝗵𝗶𝗻𝗴𝘀, 𝗵𝗼𝗹𝗱 𝘁𝗵𝗲𝗺, 𝗮𝗻𝗱 𝗽𝘂𝘁 𝘁𝗵𝗲𝗺 𝗱𝗼𝘄𝗻.

Even grip strength is a strong predictor of longevity.

👉🏻If you could only do one thing for long-term health, strength training would be at the top of the list. It improves blood work, body composition, mood, and mindset often more than almost anything else.

Muscle isn’t just about how you look.

𝘐𝘵’𝘴 𝘢𝘣𝘰𝘶𝘵 𝘩𝘰𝘸 𝘭𝘰𝘯𝘨 𝘢𝘯𝘥 𝘩𝘰𝘸 𝘸𝘦𝘭𝘭 𝘺𝘰𝘶 𝘭𝘪𝘷𝘦.

What’s your favorite strength exercise?
Mine 👉🏻 deadlift!

02/19/2026

𝗖𝗔𝗟𝗠: 𝘁𝗵𝗲 𝗖𝗹𝗶𝗻𝗶𝗰𝗮𝗹 𝗔𝗽𝗽𝗹𝗶𝗰𝗮𝘁𝗶𝗼𝗻 𝗼𝗳 𝗟𝗼𝗻𝗴𝗲𝘃𝗶𝘁𝘆 𝗠𝗲𝗱𝗶𝗰𝗶𝗻𝗲 𝗖𝗼𝗻𝗳𝗲𝗿𝗲𝗻𝗰𝗲 — is happening February 27–28 at the Omni in Fort Lauderdale 🌴

𝘓𝘰𝘯𝘨𝘦𝘷𝘪𝘵𝘺 𝘮𝘦𝘥𝘪𝘤𝘪𝘯𝘦 𝘥𝘦𝘮𝘢𝘯𝘥𝘴 𝘮𝘰𝘳𝘦 𝘵𝘩𝘢𝘯 𝘪𝘴𝘰𝘭𝘢𝘵𝘦𝘥 𝘦𝘹𝘱𝘦𝘳𝘵𝘪𝘴𝘦. 𝘐𝘵 𝘳𝘦𝘲𝘶𝘪𝘳𝘦𝘴 𝘢 𝘴𝘵𝘳𝘶𝘤𝘵𝘶𝘳𝘦𝘥 𝘧𝘳𝘢𝘮𝘦𝘸𝘰𝘳𝘬 𝘵𝘩𝘢𝘵 𝘪𝘯𝘵𝘦𝘨𝘳𝘢𝘵𝘦𝘴:
✨ Advanced hormone strategy
✨ Cardiometabolic optimization
✨ GLP-1 innovation
✨ GI diagnostics
✨ Stress & sleep physiology
✨ Body composition + performance metrics

But here’s the difference 💡
CALM isn’t just about what to prescribe.

📚It’s about how to deliver longevity medicine at the highest level through aligned teams, precise communication, strategic branding, and systems that extend care beyond the consult room.

🎙️I’m honored to be speaking at CALM ’26, presenting a clinically grounded approach to mastering the hormone metabolome, moving beyond standardized HRT into truly personalized longevity strategies.

If you’re a provider ready to elevate outcomes and refine your clinical delivery model…

📍 Omni, Fort Lauderdale
📅 February 27–28

🔗 There is still time to sign up, comment 𝗖𝗔𝗟𝗠 to get a direct link to register!

02/19/2026

𝗣𝗿𝗼𝗴𝗲𝘀𝘁𝗲𝗿𝗼𝗻𝗲 𝗶𝘀 𝘀𝘁𝗶𝗹𝗹 𝗳𝗿𝗮𝗺𝗲𝗱 𝗮𝘀 𝗮 “𝘂𝘁𝗲𝗿𝘂𝘀-𝗽𝗿𝗼𝘁𝗲𝗰𝘁𝗶𝗻𝗴 𝗵𝗼𝗿𝗺𝗼𝗻𝗲” 𝗮𝗻𝗱 𝘁𝗵𝗮𝘁 𝗳𝗿𝗮𝗺𝗶𝗻𝗴 𝘂𝗻𝗱𝗲𝗿𝘀𝗲𝗿𝘃𝗲𝘀 𝘄𝗼𝗺𝗲𝗻. 🧠

Progesterone is a neuroactive, systemic hormone that influences brain signaling, sleep regulation, stress response, and tissue balance throughout the body. For many women, the earliest signs of perimenopause aren’t hot flashes, they’re insomnia, anxiety, mood shifts, and worsening PMS, driven by early progesterone decline.

This is why I often see women with “normal estrogen” but profound symptoms. Progesterone is usually the first hormone to fall and its loss destabilizes the nervous system long before cycles stop.

Clinically, progesterone’s benefits extend far beyond endometrial protection:
🌙 Improved sleep quality
🧠 Nervous system calming
⚖️ Estrogen balance at the tissue level
🔥 Better stress tolerance

Of course, progesterone isn’t universally tolerated and context matters. But dismissing it as a uterus-only hormone is outdated and incomplete.

𝗛𝗼𝗿𝗺𝗼𝗻𝗲 𝗰𝗮𝗿𝗲 𝗶𝘀𝗻’𝘁 𝗮𝗯𝗼𝘂𝘁 👙 bikini medicine!

➡️ Follow for continued clinical hormone education
💾 Save or share if this reframed progesterone for you
🧘🏻‍♀️ Did progesterone impact your sleep or mood more than your cycle?

02/17/2026

𝗛𝗶𝘀𝘁𝗮𝗺𝗶𝗻𝗲 𝗶𝘀𝘀𝘂𝗲𝘀 𝗶𝗻 𝗽𝗲𝗿𝗶𝗺𝗲𝗻𝗼𝗽𝗮𝘂𝘀𝗲 𝗮𝗿𝗲 𝗻𝗼𝘁 𝗮 𝗰𝗼𝗶𝗻𝗰𝗶𝗱𝗲𝗻𝗰𝗲 𝗮𝗻𝗱 𝘁𝗵𝗲𝘆’𝗿𝗲 𝗻𝗼𝘁 𝗮 𝗰𝗵𝗮𝗿𝗮𝗰𝘁𝗲𝗿 𝗳𝗹𝗮𝘄.

👉🏼I see this pattern constantly in clinic: women in their 40s with flushing, anxiety, insomnia, headaches, and “reactivity” that never existed before. They’re told it’s food, stress, or aging. None of that is the root cause.

Here’s what’s actually happening 👇
🧠 Estrogen activates mast cells
🌙 Progesterone normally stabilizes them
📉 Perimenopause removes that brake system

The result is exaggerated histamine release even without classic allergies.

❌𝗧𝗵𝗶𝘀 𝗶𝘀 𝘄𝗵𝘆 𝗮𝗻𝘁𝗶𝗵𝗶𝘀𝘁𝗮𝗺𝗶𝗻𝗲𝘀 𝗼𝗳𝘁𝗲𝗻 𝗱𝗶𝘀𝗮𝗽𝗽𝗼𝗶𝗻𝘁. 𝗧𝗵𝗲𝘆 𝗯𝗹𝗼𝗰𝗸 𝗿𝗲𝗰𝗲𝗽𝘁𝗼𝗿𝘀 𝗯𝘂𝘁 𝗶𝗴𝗻𝗼𝗿𝗲 𝘁𝗵𝗲 𝘂𝗽𝘀𝘁𝗿𝗲𝗮𝗺 𝗶𝗻𝗳𝗹𝗮𝗺𝗺𝗮𝘁𝗼𝗿𝘆 𝘀𝗶𝗴𝗻𝗮𝗹𝗶𝗻𝗴 𝘁𝗵𝗮𝘁’𝘀 𝗱𝗿𝗶𝘃𝗶𝗻𝗴 𝘁𝗵𝗲 𝗿𝗲𝗮𝗰𝘁𝗶𝗼𝗻 𝗶𝗻 𝘁𝗵𝗲 𝗳𝗶𝗿𝘀𝘁 𝗽𝗹𝗮𝗰𝗲.

📌In carefully selected patients, I sometimes use Amlexanox, a small-molecule immune modulator that helps quiet mast-cell activation and inflammatory pathways tied to metabolic and hormonal stress. It’s not a hormone. It’s not a supplement trend. It’s a targeted small molecule.

⚠️ This is not a DIY solution.
⚠️ It’s not for everyone.
⚠️ Context and labs matter.

𝘉𝘶𝘵 𝘥𝘪𝘴𝘮𝘪𝘴𝘴𝘪𝘯𝘨 𝘩𝘪𝘴𝘵𝘢𝘮𝘪𝘯𝘦 𝘴𝘺𝘮𝘱𝘵𝘰𝘮𝘴 𝘪𝘯 𝘱𝘦𝘳𝘪𝘮𝘦𝘯𝘰𝘱𝘢𝘶𝘴𝘦 𝘢𝘴 “𝘫𝘶𝘴𝘵 𝘢𝘯𝘹𝘪𝘦𝘵𝘺” 𝘪𝘴 𝘰𝘶𝘵𝘥𝘢𝘵𝘦𝘥 𝘮𝘦𝘥𝘪𝘤𝘪𝘯𝘦.

📌If this helped you understand your body better, save it.
👉🏼If you know a woman stuck in this loop, share it.
👌🏻And follow for continued, clinically grounded hormone education.

🗝️Have you noticed your symptoms track with your cycle or sleep? Drop 👇🏻

02/17/2026

🎤First time speaking at a legislative committee hearing and giving formal testimony. Grateful for the opportunity to advocate for patient safety at the state level.

My hope is that those of us who voiced our concerns helped clarify that we’re discussing two separate issues: regulation of compounding pharmacies and guidelines for medical spas. They are not the same, and they require different solutions.

If we truly want to protect patients, we have to define scope clearly, enforce existing standards consistently, and work collaboratively across professions.

Patient safety must remain the priority. Always.

🗣️Proud to use my voice in the room where policy shapes care.

Huge thank you to Alex R. Thiersch, JD founder of AmSpa and Dr.Jamie Harper for also providing your testimony.

02/17/2026

🎤Practicing my testimony in response to Senate Bill 2️⃣8️⃣2️⃣

🗣️Tomorrow I’ll be headed downtown to speak with legislators about patient safety and, more importantly, what that actually means. As a nurse practitioner and founder of a medical practice, I believe safety starts with clear standards, strong enforcement of existing laws, and accountability for noncompliant individuals.

📌More paperwork doesn’t automatically equal safer care. Thoughtful policy does. I’m looking forward to productive conversations about protecting patients while supporting responsible, licensed providers who are already committed to doing this work the right way.

02/17/2026

Most people won’t tell you this but… feeling something doesn’t always mean it’s working. ⚡️

When it comes to NAD IVs or injections, many patients report a flushing sensation, chest tightness, or that “mitochondrial rush.” For some, that intense feedback feels reassuring, like their cells just flipped a switch. For others, it’s 𝘂𝗻𝗰𝗼𝗺𝗳𝗼𝗿𝘁𝗮𝗯𝗹𝗲 𝗮𝗻𝗱 𝗼𝘃𝗲𝗿𝘀𝘁𝗶𝗺𝘂𝗹𝗮𝘁𝗶𝗻𝗴.

Here’s the clinical perspective 👇

🔬 𝗡𝗔𝗗 (𝗡𝗶𝗰𝗼𝘁𝗶𝗻𝗮𝗺𝗶𝗱𝗲 𝗔𝗱𝗲𝗻𝗶𝗻𝗲 𝗗𝗶𝗻𝘂𝗰𝗹𝗲𝗼𝘁𝗶𝗱𝗲)
• May cause flushing or a pounding sensation
• Can feel stimulating or intense
• Physical sensation ≠ therapeutic effectiveness

🧬 𝟭-𝗠𝗡𝗔 (𝟭-𝗠𝗲𝘁𝗵𝘆𝗹𝗻𝗶𝗰𝗼𝘁𝗶𝗻𝗮𝗺𝗶𝗱𝗲)
• Supports mitochondrial efficiency and vascular health
• Typically does not cause flushing
• Feels smoother and more subtle
• Focused on metabolic and endothelial support

In functional and longevity medicine, we don’t base success on whether you “feel” something happening. We look at improved cellular energy, metabolic resilience, and long-term mitochondrial health. ⚖️

𝗠𝗼𝗿𝗲 𝘀𝘁𝗶𝗺𝘂𝗹𝗮𝘁𝗶𝗼𝗻 𝗶𝘀𝗻’𝘁 𝗮𝗹𝘄𝗮𝘆𝘀 𝗯𝗲𝘁𝘁𝗲𝗿 𝗲𝘀𝗽𝗲𝗰𝗶𝗮𝗹𝗹𝘆 𝗶𝗳 𝘆𝗼𝘂𝗿 𝗻𝗲𝗿𝘃𝗼𝘂𝘀 𝘀𝘆𝘀𝘁𝗲𝗺 𝗶𝘀 𝗮𝗹𝗿𝗲𝗮𝗱𝘆 𝘂𝗻𝗱𝗲𝗿 𝘀𝘁𝗿𝗲𝘀𝘀.

If you’ve been curious about NAD or alternatives like 1-MNA, this conversation is worth your time. 🧠✨

🔗 Comment 𝗘𝗡𝗘𝗥𝗚𝗬 and I’ll send you the full link to my most recent podcast episode with Dr. Leonard Pastrana on The Better Yoo Project

Lexi Yoo, FNP, IFMCP

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