Sandi Krakowski

  • Home
  • Sandi Krakowski

Sandi Krakowski Certified Menopause & Peptide mentor. CEO 33 yrs. Investor. $100m+ Telehealth owner. AI creator. Change maker. Researcher.

When I moved to Texas in Jan 2023, my heart and soul truly found its home. In all of the states and places I’ve lived, n...
11/04/2026

When I moved to Texas in Jan 2023, my heart and soul truly found its home. In all of the states and places I’ve lived, nothing felt quite like this. The last 3 years I’ve dreamed of owning land, a nice big home all of my children and grandchildren would love to come visit…. And I found it.

Wednesday I move into my lil 5.5 acre ranch, with my protector here, Cargo and my 4 cartoon characters! God has prepared me for many years just for this.

I’m gonna call it REDEMPTION RANCH, because that’s exactly what He’s done in me! He’s redeemed all that the enemy tried to rob me of and destroy! He’s turned my mourning into dancing and laughter and He’s shown me how to multiply all that I’ve been given. Hallelujah!!! Never give up!

During the last two years, doctors have been popping up all over social media, posting about estrogen patches, as if it’...
10/04/2026

During the last two years, doctors have been popping up all over social media, posting about estrogen patches, as if it’s the greatest thing since store-bought bread! It’s supposed to make your life easier, every woman should be prescribed it, and it is the number one used menopause relief tool in the world. But is it really that good?

I have a large client base that gives me access to hundreds of thousands of women’s feedback. The majority of them cannot get their levels to stay optimized on a patch. Many go on a patch, but still can’t sleep, still have aching joints, and think this is just a part of menopause. No, it’s a part of being prescribed a dose that is so freaking low that it’s not doing anything!

So why is there an estrogen patch shortage? I’ve heard women say it’s because of the patriarchy. It always is lol 🤦🏼‍♀️ or because they don’t want women to be healthy. Or because they only care about men.

The reason there’s an estrogen shortage is because most of the clinicians who are prescribing any menopausal therapy are only prescribing an estrogen patch and progesterone. A very small minority are giving individualized care with specific modalities and dosing so that a woman can achieve high enough hormone levels to literally make all symptoms of menopause disappear. Even fewer even understand testosterone. But the shortage is because the patch has been pushed like it’s God‘s answer for women. And honestly, I think it’s the stupidest modality out there.

To achieve optimized levels, compounded creams, gel, oral, and injections accomplish that. But do you wanna know what is sadder than an estrogen shortage of patches? That the majority of clinicians don’t even know how to prescribe oral or injections! And have no clue how to adjust dosing of creams.

A shortage exists because proper dosing isn’t happening and it’s being prescribed like a Motrin.

08/04/2026

Oral Birth Control: Medically Forced Menopause.

How The Pill Works: Your Hormones on “Pause”
First, what happens naturally each month
Your body runs on a hormonal conversation between your brain and ovaries. Your brain sends out two messenger hormones — FSH and LH — that tell your ovaries to grow an egg and release it (ovulation). Your ovaries respond by making estrogen and progesterone, which prepare your uterus in case the egg gets fertilized.

What the pill does
Birth control pills contain synthetic (man-made) versions of estrogen and progesterone. When you take them every day, your brain detects those synthetic hormones in your bloodstream and essentially concludes: “Hormone levels are already elevated — no need to send signals to the ovaries.”
So it stops sending FSH and LH.
No FSH + no LH = no egg released = no pregnancy possible.

The “post-menopausal” part
Here’s the important piece: the pill suppresses your own ovarian hormone production — your ovaries go quiet because they’re getting no signal to work. The synthetic hormones in the pill replace them, but at carefully controlled, relatively low and flat levels — unlike the natural monthly peaks and valleys your body would otherwise create.
This flat, low hormonal environment is functionally similar to what happens after menopause, when the ovaries have naturally stopped producing hormones. The ovaries aren’t being stimulated — they’re essentially on standby.

Why this matters
∙ Your body’s natural cycle — with its rising and falling estrogen and progesterone — is suppressed the entire time you’re on the pill
∙ Some research suggests this can affect things like mood, bone density, muscle mass, metabolic issues, libido, energy, and even how your body responds to stress
∙ When you stop taking the pill, your brain-ovary conversation usually restarts — though it can take some time

The pill is effective, but it’s worth understanding that it works by temporarily replacing your natural hormone system with a synthetic substitute — not just “preventing pregnancy” in isolation.

08/04/2026

The risks of HRT for peri and postmenopausal women.

Oral Contraceptives: What the Research Actually Shows:Combined oral contraceptives (COCs) significantly upregulate s*x h...
07/04/2026

Oral Contraceptives: What the Research Actually Shows:

Combined oral contraceptives (COCs) significantly upregulate s*x hormone-binding globulin (SHBG) — in some studies by 3–4x — which reduces free testosterone and estradiol bioavailability and can persist for months to years after discontinuation, complicating hormone optimization protocols in menopause.

Ethinyl estradiol’s synthetic structure activates hepatic estrogen receptors differently than endogenous estradiol, altering serotonin transport, androgen receptor sensitivity, and HPA axis regulation — mechanisms linked in the literature to mood dysregulation and s*xual dysfunction.

COCs suppress the HPG axis, reducing circulating estradiol and testosterone to levels consistent with the early postmenopausal range. Sustained suppression of these hormones is independently associated with reduced bone mineral density, impaired collagen synthesis, and dysregulated immune modulation.

Prolonged estrogen and androgen insufficiency — beginning in a woman’s teens or twenties — carries well-documented risk for accelerated bone loss, increased susceptibility to autoimmune conditions, metabolic dysfunction, and s*xual health impairment.

COC use is associated with a 3–6x increased relative risk of venous thromboembolism compared to non-users, with risk profile varying by progestin generation and estrogen dose. This exceeds the VTE risk of most medications commonly prescribed to reproductive-age women.

Epidemiological data, including large Danish cohort studies, have demonstrated a statistically significant association between COC use and increased risk of arterial thrombotic events, including myocardial infarction, particularly in women with additional cardiovascular risk factors.
Informed consent around these risks remains inconsistent in clinical practice.

27/11/2025

Yes I’m a Texan but won my heart when I lived in KC! 🏈❤️

27/11/2025

Yes it’s AI. I don’t have a talking turkey named Gobble. Happy Thanksgiving! 😂😂😂😂😂

Address


Alerts

Be the first to know and let us send you an email when Sandi Krakowski posts news and promotions. Your email address will not be used for any other purpose, and you can unsubscribe at any time.

Contact The Practice

Send a message to Sandi Krakowski:

  • Want your practice to be the top-listed Clinic?

Share

Share on Facebook Share on Twitter Share on LinkedIn
Share on Pinterest Share on Reddit Share via Email
Share on WhatsApp Share on Instagram Share on Telegram