The Researcher OG

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Mike Robinson, Researcher OG
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Creator of Researcher®️ Genevieve's Dream™️ Nano Terps®️ Thermonoids®️ GENEVEX®️ Plant Chat®️ PREVENT®️ Get Balanced®️ NANO®️ Jane®️ & more

CEO Nanobles/Global Cannabinoid Research Center GCRC

Q. Does Diet cause our own carbon footprint, as a human, to be larger? A: Yes. The food you eat, especially a diet heavy...
02/04/2026

Q. Does Diet cause our own carbon footprint, as a human, to be larger? A: Yes. The food you eat, especially a diet heavy in red meat, can influence the amount of carbon dioxide (CO₂) you emit when you breathe due to how your body metabolizes different macronutrients. Here’s how this process works and how it may affect circulating endocannabinoids:

When you consume food, your body breaks it down into energy. This metabolism process involves using oxygen to burn the nutrients, and one of the byproducts of this process is carbon dioxide. The more energy your body needs to extract from food, the more CO₂ you produce and release through breathing.

Red meat and carbon emission: Red meat is rich in proteins and fats, which require more energy for digestion and metabolism than carbohydrates. Fat, in particular, is calorie-dense, so when you consume red meat, your body generates more CO₂ as it breaks down these fats and proteins.

The excess carbon dioxide produced from metabolizing red meat can create physiological imbalances that affect the Endocannabinoid System (ECS). Here’s how excessive carbon interrupts circulating endocannabinoids:

When your body generates too much carbon dioxide, especially from a high-protein, high-fat diet, it can contribute to oxidative stress. This is a condition where there’s an imbalance between free radicals (reactive oxygen species) and antioxidants in the body. Oxidative stress can disrupt the signaling pathways of endocannabinoids like Anandamide (AEA) and 2-AG, which are crucial for maintaining ECS balance.

Endocannabinoid degradation: Oxidative stress can also increase the activity of enzymes such as FAAH (fatty acid amide hydrolase) and MAGL (monoacylglycerol lipase), which break down endocannabinoids like AEA and 2-AG. When these enzymes are overly active, they can reduce the circulating levels of these crucial endocannabinoids, thereby disrupting the balance of the ECS. -Mike Robinson, The Researcher OG

Many patients today face challenges when discussing cannabis with their doctors. Unfortunately, many healthcare professi...
02/04/2026

Many patients today face challenges when discussing cannabis with their doctors. Unfortunately, many healthcare professionals still have limited knowledge of the Endocannabinoid System (ECS) or the therapeutic potential of cannabinoids like THC, CBG, and CBD. While CBD has gained some recognition due to its approval in pharmaceutical drugs like Epidiolex, the broader medical benefits of cannabis are often underrepresented or misunderstood by the medical community.

As a result, patients often find themselves researching cannabis on their own or relying on support from other cannabis users. These communities share experiences about strain selection and dosage and now discuss ways to balance their ECS for better overall health. This significant gap highlights the need for more comprehensive education in the medical field about ECS and cannabinoids to better support patients - and shows the inability of the US to legalize cannabis as a scheduled drug.

Scheduled drugs require extensive knowledge from doctors who can advise them; with that knowledge base in place, a move to Schedule 3 for the plant would benefit citizens. Let’s forget about all those in suits who want a move just for a tax write-off and think about the people who’d be subject to the same doctor they have now, suddenly being the expert, and the people they know giving out information shut down by regulations.

Until doctors are more knowledgeable, patients will continue to rely on each other for guidance. Knowledge is empowerment— so stay informed and connected with others who understand the journey. We’re not at a place in time where medical professionals can pick up where the stoner, the grower, the researcher or formulator left off.

They’re nowhere near ready.

-Mike Robinson, Researcher OG

”If people let the government decide what foods they eat and what medicines they take, their bodies will soon be in as s...
02/03/2026

”If people let the government decide what foods they eat and what medicines they take, their bodies will soon be in as sorry a state as are the souls who live under tyranny.”- Thomas Jefferson ~~ I love plants, extracts, and the fun of using them.

Cannabis affects neural functions through the binding of its active component (Delta(9)-THC) to cannabinoid receptors in...
02/03/2026

Cannabis affects neural functions through the binding of its active component (Delta(9)-THC) to cannabinoid receptors in the CNS.

Recent studies have shown that endogenous ligands for cannabinoid receptors, endocannabinoids, serve as retrograde messengers at central synapses. Endocannabinoids are produced on demand in activity-dependent manners and released from postsynaptic neurons.

The released endocannabinoids travel backward across the synapse, activate presynaptic CB1 cannabinoid receptors, and modulate presynaptic functions.

Retrograde endocannabinoid signaling is crucial for certain forms of short-term and long-term synaptic plasticity at excitatory or inhibitory synapses in many brain regions, and thereby contributes to various aspects of brain function including learning and memory.

Molecular identities of the CB1 receptor and enzymes involved in production and degradation of endocannabinoids have been elucidated.

Anatomical studies have demonstrated unique distributions of these molecules around synapses, which provide morphological bases for the roles of endocannabinoids as retrograde messengers.” -Mike Robinson, The Researcher OG

Cancer taught me how to actually listen to my body instead of fighting it. When everything is on the line, you stop gues...
02/03/2026

Cancer taught me how to actually listen to my body instead of fighting it. When everything is on the line, you stop guessing and start paying attention. That’s when I learned how to examine my ECS, our Master Regulator, in real time and make adjustments instead of forcing outcomes. Pain, sleep, appetite, inflammation, mood - none of those exist in isolation. They are signals, and cancer makes those signals loud.
I quickly learned that relying on a single cannabinoid creation such as FECO or RSO, is like trying to tune an orchestra with a single instrument. THC-rich plants helped me manage pain and with sleep, but too much of it dulls the signal and eventually creates tolerance.
CBD helped calm inflammation, anxiety, and nervous system overdrive, but on its own it wasn’t enough. CBG changed the conversation. It supported gut function, inflammation control, and mental clarity in a way that felt foundational, not overpowering.
Then I honed in on CBGa, the precursor molecule. That’s where balance really started to show up. CBGa doesn’t push;; it organizes. It supports endocannabinoid signaling, helps other cannabinoids work better, and fills in gaps that THC-heavy routines often create. For me, that meant using less THC while getting more benefit overall.
Staying balanced is the whole game. If your THC tolerance is high, that’s not failure, it’s feedback. It’s your ECS telling you it needs support, not more pressure. That’s where adjusting protocols, adding CBG and CBGa, and actually listening to your system matters.
Balance isn’t about quitting anything. It’s about learning how to work with your biology instead of overpowering it. If you get Cancer, being imbalanced makes it so much harder to find effective therapy of any type, as our ECS is in charge.
-Mike Robinson, GCRC Founder - The Researcher OG

Address

Santa Barbara, CA

Opening Hours

Monday 10am - 6pm
Tuesday 7am - 5pm
Wednesday 7am - 5pm
Thursday 2pm - 10pm
Friday 2pm - 10pm
Saturday 10am - 5pm
Sunday 6pm - 10pm

Website

https://GlobalCannabinoidRC.com/, https://GenevievesDream.com/

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