Dermatology Clinic of Weslaco - Oasis Dermatology Group, PLLC

09/13/2025

We’re proud to serve our community with top-quality dermatology care! 🌟

📍 Come see us at Dermatology Clinic of Weslaco, 217 S Oklahoma Ave Ste , Weslaco TX 78596
Your neighborhood clinic for healthy, radiant skin.

✨ Book your appointment!
📞 (956) 971-0404
🌐 www.oasisderm.com

09/01/2025

Happy Labor Day from Oasis Dermatology! 💼✨

Today, we celebrate the hard work, dedication, and commitment that build the foundation of our community. Whether you’re spending the day relaxing or reflecting on your achievements, we hope you enjoy a well-deserved break. Thank you for contributing your passion and effort every day together, we make a difference.

Wishing everyone a safe and joyful Labor Day! 🌿✨
✨ Book your appointment!
📞 (956) 971-0404
🌐 www.oasisderm.com

08/15/2025

Thank you to everyone who entered our Back to School Giveaway. 📚✏️🍎 We loved seeing all your enthusiasm as we get ready for another amazing school year!

And now, the moment you've all been waiting for...
🥁 Drumroll, please…

🎉 Congratulations to Esmer Chavez and her shining daughter! 🎉

📚

08/07/2025

07/16/2025

🎒 BACK-TO-SCHOOL GIVEAWAY! 🧴✏️
We're giving one lucky student a fun + fresh start to the school year!

✨ TO ENTER:
1️⃣ Like this post
2️⃣ Follow our page
3️⃣ Share to your story & tag 2 friends

👯‍♀️ Bonus entry: Tag your friends in the comments (each tag = 1 extra entry!)

📍 Rules:
-Must be under 18
-Must live in Weslaco, Donna, Alamo, Edcouch, Elsa, La Feria, and Progreso
-Must pick up prize at our clinic on August 14th between 9AM–4PM.

🗓 Giveaway closes August 6th!! Winner will be announced in our stories—stay tuned!

07/04/2025

As we celebrate Independence Day, we’re grateful for the freedom to care for your skin and help you shine with confidence — all year long!

This July, protect your skin like you protect your freedom:
☀️ Sunscreen is your skin's best defense!
Wishing you a safe, glowing, and beautiful 4th of July!

✨ Book your appointment today!
📞 (956) 971-0404
🌐 www.oasisderm.com

07/02/2025

Derm Index: Let’s learn about Angiosarcoma

Angiosarcoma is a rare and highly aggressive malignancy that originates from endothelial cells, the cells that line the blood vessels. Representing approximately 1% of all soft tissue sarcomas, it is known for its poor prognosis, particularly when diagnosed at advanced stages. The five-year survival rate for angiosarcoma patients ranges between 12% and 25%, with a high incidence of local recurrence and distant metastasis.

The tumor is known to involve the vascular system; this neoplasm can present as solid masses, nodules, or irregular vascular formations, which complicate early diagnosis and management.

Pathophysiology and Etiology

Angiosarcomas arise from endothelial cells and proliferate in an uncontrolled manner, leading to tumor formation. These tumors may grow along pre-existing vascular channels or create new, disorganized vascular spaces within the tumor. The exact cause of angiosarcoma remains unclear; however, several risk factors have been identified. Notably, a history of chronic lymphedema and prior radiation therapy are well-established predisposing factors. Radiation-induced angiosarcomas often develop after breast cancer treatment and are associated with the tissue changes resulting from radiation-induced fibrosis.

Angiosarcoma can be categorized into cutaneous and visceral types, depending on the location of the primary tumor. Cutaneous angiosarcomas are the most common, typically present in elderly individuals, especially white men, with a median age between 60 and 71 years. The head and neck region is the most frequent site for cutaneous angiosarcoma, although the tumor is also frequently observed on the chest wall in patients who have undergone radiation therapy for breast cancer.

To Read More >>> https://oasisderm.com/angiosarcoma-2/

Download the full PDF:https://oasisderm.com/wp-content/uploads/2025/02/Angiosarcoma.pdf

Image Source: dermnetnz.org

06/26/2025

Derm Index: Understanding Nevus Sebaceous

Nevus sebaceous is a birthmark that is present at birth in less than 1% of babies, often found on the face or scalp. Initially, it looks flat, smooth, and pink, with no hair around its edges. As a child grows, especially during puberty, the lesion may change—it can become darker, larger, and develop raised bumps, taking on an orange, warty appearance. While nevus sebaceous is usually harmless, there is a small risk that it can develop into a skin cancer later in life, so it’s important for the lesion to be monitored by a healthcare provider.

Clinical Features

At birth, nevus sebaceous presents as a flat, pink, and velvety patch, typically localized to the scalp or face. Over time, especially during puberty, the lesion undergoes transformation, becoming more raised, yellow-orange, and papular. The surface may develop warty ridges, and hair around the edges of the lesion may be absent. Sebaceous glands, epidermal cells, hair follicles, and connective tissue are all components of the lesion, which is why it is classified as a hamartoma—a benign overgrowth of normal tissue types.

Nevus sebaceous is typically observed in both males and females across all ethnic groups, without any marked predilection for gender or race. The lesion usually appears as a single plaque, although multiple lesions can occur in rare instances. Most often, these lesions are asymptomatic, though they may be cosmetically concerning or present with complications in some cases.

To Read More >>> https://oasisderm.com/nevus-sebaceous/

Download the full PDF:
https://oasisderm.com/wp-content/uploads/2025/01/Nevus-Sebaceus.pdf

Image Source: dermnetnz.org

06/20/2025

Derm Index: Everything You Need to Know About Leprosy

Leprosy (Hansen’s disease) is a chronic infectious disease caused by Mycobacterium leprae, a slow-growing bacterium that primarily affects the skin, peripheral nerves, and upper respiratory tract. Although leprosy is a global health concern, it is more prevalent in tropical and subtropical regions, with significant cases reported in countries such as India, Brazil, and Indonesia. In the United States, cases are most commonly found in California and Hawaii. Leprosy remains a public health challenge due to its prolonged incubation period, low transmission rate, and potential for severe complications if left untreated.

Pathophysiology and Transmission

Mycobacterium leprae is an acid-fast bacterium with a slow replication rate, which leads to a long incubation period for leprosy. Symptoms of the disease may not appear until several years after infection, with a latency period of up to 20 years in some individuals. The bacteria have a marked affinity for cooler regions of the body, including the skin, peripheral nerves, and mucous membranes, particularly in the upper respiratory tract. Transmission primarily occurs through prolonged close contact with an infected person via respiratory droplets. Additionally, zoonotic transmission has been reported, particularly from armadillos, which can serve as a reservoir for M. leprae. Despite these transmission routes, leprosy is not highly contagious, and the risk of person-to-person transmission is low, particularly when the affected individual is undergoing appropriate medical treatment.

Genetics are thought to play a significant role in susceptibility to leprosy. Certain genetic variations, especially in immune system-related genes, have been associated with an increased risk of developing the disease. This suggests that a person’s immune response is crucial in determining whether M. leprae infection progresses to clinical leprosy.

To Read More >>> https://oasisderm.com/leprosy-2/

Download the full PDF:
https://oasisderm.com/wp-content/uploads/2025/01/Leprosy.pdf

Image Source: dermnetnz.org

06/15/2025

👔 Dads are tough—but their skin doesn’t have to be.
This Father’s Day, treat Dad to expert skincare tailored just for him.💪✨

✨Book a session for Dad today!
📞 (956) 971-0404
🌐 www.oasisderm.com

06/10/2025

Derm Index: Your Guide to Fox-Fordyce Disease

Fox-Fordyce disease (FFD) is a rare, chronic dermatological disorder characterized by intense pruritus (itching) and inflammation, predominantly affecting regions of the body with apocrine sweat glands, such as the axillae (armpits), anogenital area, and around the ni***es. The disease presents with the formation of small, raised, pruritic papules, often associated with hyperpigmentation (darkening) and dryness around hair follicles. Although FFD is most frequently observed in adolescent and premenopausal women, particularly those between the ages of 13 and 35, it can also affect men, children, and postmenopausal women. The condition is notably rare, with few cases documented in the medical literature.

Etiology

The etiology of FFD remains poorly understood, though several hypotheses have been proposed. One prominent theory suggests that the disease results from obstruction of the apocrine sweat glands, which are primarily responsible for the condition’s characteristic lesions. Inflammation that follows glandular blockage may contribute to the development of the disease. Additionally, hormonal fluctuations, particularly those associated with menstruation, pregnancy, and menopause, have been implicated in the exacerbation or onset of symptoms. A genetic predisposition may also play a role, though the specific genetic factors remain unclear. Research is ongoing to elucidate the precise mechanisms behind FFD.

Diagnosis

Diagnosis of FFD is primarily clinical, based on the characteristic appearance of the lesions and patient history. However, in atypical cases, skin biopsy may be necessary to confirm the diagnosis, as the condition can be mistaken for other dermatological disorders such as folliculitis, pseudofolliculitis, or miliaria rubra. The disease often presents abruptly following heat exposure, humidity, friction, or exercise in the affected areas, with stress and hormonal changes further aggravating symptoms. In some individuals, visual manifestations of FFD may occur without accompanying pruritus. Symptoms often worsen during menstruation, and in severe cases, the condition may result in permanent damage to the apocrine glands, leading to localized anhidrosis (lack of sweating) and hair follicle damage.

To Read More >>> https://oasisderm.com/fox-fordyce-disease-2/

Download the full PDF:
https://oasisderm.com/wp-content/uploads/2024/12/Fox-Fordyce-Disease.pdf

Image Source: dermnetnz.org

06/06/2025

Derm Index: Everything You Need to Know About Cowden Syndrome

Cowden syndrome (CS), also known as multiple hamartoma syndrome, is a rare autosomal dominant disorder characterized by the presence of multiple benign tumor-like growths called hamartomas. These growths affect various tissues, including the skin, mucosa, bones, gastrointestinal tract, genitourinary system, eyes, and central nervous system (CNS). Cowden syndrome is also associated with an increased risk of several malignancies, particularly breast, thyroid, and endometrial cancers, which makes early detection critical for effective management.

The disorder is inherited equally among males and females, although the incidence of malignancies tends to differ by gender. The estimated prevalence of Cowden syndrome is approximately 1 in 250,000 individuals, with the onset of clinical manifestations occurring between birth and approximately 46 years of age.

Genetic Basis and Pathophysiology

The majority of Cowden syndrome cases result from mutations in the PTEN (phosphatase and tensin homolog) gene, a tumor suppressor gene that plays a key role in regulating cell growth, survival, and division. Mutations in PTEN lead to uncontrolled cell proliferation, which causes the formation of hamartomas and predisposes individuals to the development of malignancies, particularly in tissues such as the breast, thyroid, and uterus. Approximately 20% of patients with Cowden syndrome have no identifiable genetic mutation, suggesting that additional genetic or environmental factors may contribute to the disease in some individuals.

To Read More >>> https://oasisderm.com/cowden-syndrome-2/

Download the full PDF:
https://oasisderm.com/wp-content/uploads/2024/12/Cowden-Syndrome.pdf

Image Source: dermnetnz.org