TANGS CLINICAL TCM

10/10/2025

https://www.facebook.com/share/1CbHGKf7ur/?mibextid=wwXIfr

16/09/2025

My heart is with a few cases who are in a very difficult condition, I am earnestly praying for their speedy recovery and for strength to surround them and their family. Jeremiah 30:17, 'For I will restore health to you, and your wounds I will heal, declares the Lord.' May God open a door for their healing.

06/09/2025

https://tangsclinical.com/internal-imbalances-vs-external-invaders-a-tcm-view-of-acne-and-folliculitis/

Explore the differences between acne and folliculitis from a Traditional Chinese Medicine (TCM) perspective. Learn how TCM diagnoses and treats these conditions based on internal imbalances vs. external factors.

28/08/2025

23/08/2025

指甲银屑病影响高达50%的皮肤银屑病患者和80%的银屑病关节炎患者[1]，但也可能单独发生，且不累及皮肤或关节。单独的指甲银屑病诊断困难，常与甲癣、创伤性营养不良或甲状腺疾病等全身性疾病混淆。指甲银屑病的治疗通常很困难，因为指甲生长缓慢会延缓可见的恢复。此外，对于哺乳期患者，治疗选择有限，因为许多全身性或局部用药存在转入母乳或对婴儿造成潜在危害的风险。

Case report of isolated nail psoriasis in a postpartum woman with hyperthyroidism, achieving remission with oral herbal medicine and 10-month photo follow-up.

21/08/2025

这个孩子成了一个诊断的密码体——一个活生生的临床谜团，游走在炎症与肿瘤之间的灰色地带。

在传统治疗路径失败、病灶持续扩散的情况下，家长选择了一种更符合孩子生理脆弱性的方案，同时也出于对再次活检和免疫抑制治疗的顾虑。

一名10岁男童因进行性红斑样皮疹接受常规治疗无效，皮肤活检提示界面性皮炎，临床疑似早期蕈样肉芽肿或糠疹样苔藓。患儿在未接受系统性免疫抑制治疗的情况下，转向中医治疗，并于16个月内皮损完全消退，无复发或瘢痕。本文展现了中医免疫调节疗法在儿童免疫相关皮肤病诊断不明时的潜力与意义。

这个结局不应被解读为对循证医学的否定，而是对一种更包容的认识论的召唤——在这种框架中，不确定不再是阻碍，而是一种激发多元思考的契机。

A 10-year-old boy with progressive erythematous macules defied conventional diagnosis, presenting with ambiguous histopathology suggestive of early mycosis fungoides versus pityriasis lichenoides. After failing topical immunosuppressants, the patient underwent a 16-month Traditional Chinese Medicine...

12/08/2025

https://tangsclinical.com/a-58-year-old-woman-with-psoriasis-vulgaris-received-full-recovery-in-6-months/

30/07/2025

https://tangsclinical.com/what-is-a-possible-diagnosis-of-this-case/

30/07/2025

https://tangsclinical.com/angular-cheilitis-and-eczema-like-hand-lesions-in-a-35-year-old-woman/

19/07/2025

1. Microbial ‘de-wilding’. Caesarean birth, antibiotics, antiseptic urban life erode commensal diversity that normally trains immunity regulation.
2. Ultra-processed food & low fibre. Intestinal barrier impairment heightens systemic sensitisation.
3. Climate & pollution synergy. Elevated CO₂ extends pollen seasons; diesel particles make pollen grains more allergenic.
4. Dietary Westernisation in Asia. Rapid shift from fresh to packaged foods compresses a century of Western change into one generation.
5. Diagnostic visibility. Wider access to IgE panels and oral-food-challenge units partly inflates apparent growth, but cannot explain 2–3 fold jumps alone (signal > surveillance).

Doctors said adults can develop allergies to foods and other substances that they have tolerated for years.